Acute MSK / MSK emergencies Flashcards
Compartment syndrome.
a) Causes
b) Signs and symptoms
c) To diagnose
d) Management
e) How many compartments in upper limb?
a) Crush injury, tight bandaging/casting, broken bones (supracondylar and tibial shaft especially), burns
b) PAIN!! and increased on extension of affected muscle compartment (e.g. foot dorsiflexion if leg affected)
c) Intracompartmental pressure measurement:
- >20 abnormal
- >40 abnormal
d) Urgent surgical decompression via open fasciotomy
e) 4
CES.
a) Causes
b) Presentation
c) Management
a) Disc herniation, malignant compression, fracture, abscess, haematoma
b) Back pain, bilateral leg weakness / sciatica, bowel/ bladder/ sexual dysfunction, saddle anaesthesia
c) - Urgent MRI spine
- Call spinal surgeons
- Surgical decompression
- Dexamethasone?
Fat emboli.
a) Generally post…? (pathogenesis)
b) Prevention
a) Long bone fractures: the yellow marrow in the diaphysis is exposed and damaged
b) Early traction
Fracture description.
AABCS
Adequacy Alignment Bones - OLD ACID (Open/closed, location, degree of fracture, articular involvement/angulation, communion, intrinsic bone quality, displacement/rotation) Cartilage Soft tissue
NB. Angulation - varus or valgus
Open fracture management
a) Non-surgical
b) Surgical
a) - Clean and cover
- Analgesia
- Check neurovascular status (if impaired require urgent surgery)
- IV access
- Take bloods
- IV fluids/ transfusion
- IV ABx (co-amox)
- Tetanus booster
- XR imaging
b) - Reduce and splint
- Surgical debridement
- Later- fixation
Hot, swollen joint: differentials
a) Monoarthritis
b) Oligoarthritis
c) Polyarthrtitis
a) Septic, gout, pseudogout
b) RA, reactive arthritis
c) RA, psoriatic, polyseptic arthritis (gonococcal)
Septic arthritis: management
- IV Abx (fluclox)
- Bloods - sepsis 6 (FBC, CRP, cultures), urate
- Joint aspiration - gram stain, cell count, crystal analysis, MCS
- XR/ MRI
- Surgical washout if necessary
Fracture.
a) stages of healing
b) drugs to avoid during acute healing stages
a) - Inflammation: ruptured blood vessels results in a haematoma and release of inflammatory cytokines (days)
- Formation of soft callus: haematoma becomes organised and is infiltrated by fibrovascular tissue, which forms initial spongy bone, which is still tender and liable to re-fracture; needs immobilising (days - weeks)
- Formation of hard callus: soft callus is replaced by hard callus over period of weeks; should be non-tender and able to move/weight bear (~ 6 weeks post-injury)
- Remodelling: gradual replacement of hard callus by strong cortical bone (months - years)
b) Bisphosphonates, NSAIDs, steroids and other immunosuppressives, quinolones
Fracture management principles (3)
Reduce – Hold – Rehabilitate
Fracture reduction.
a) What is it?
b) Why is it done?
c) How is it done?
a) Restoring the anatomical alignment of a fracture or dislocation of the deformed limb
b) Allows for bleeding tamponade and relief of pressure on surrounding structures (eg. nerves, vessels)
c) - requires analgesia (ideally a local/regional nerve block)
- usually performed ‘closed’ in ED; however, may be performed ‘open’ in theatre
- may or may not involve traction
Fracture immobilisation.
a) Methods
b) Casting principles
c) Further management
a) Methods:
- Splinting (eg. Thomas splint for femoral shaft #)
- Plaster cast
- Surgical
b) - Initially only back-slab, not circumferential (allows swelling to occur)
- Not too tight - prevents compartment syndrome
- Should cover joint below the fracture (and also joint above if there is axial instability - prevents bone turning on its long axis)
c) - Decide whether suitable for weight-bearing or not
- VTE given
- Advice on compartment syndrome
Fracture rehabilitation.
Physiotherapy.
- Ensure unaffected and non-immobilised joints are kept moving to avoid joint stiffness
- Once there is fracture union (usually after ~ 6 weeks), the affected bones should be stressed within limits to allow for strengthening
- Strengthen surrounding muscles
Occupational therapy.
- May need aids/devices to assist with ADLs
Osteomyelitis.
a) Most common site and spread in children
b) Risk factors
c) Presentation
d) Investigations
e) Management - acute vs. chronic
a) - Haematogenous (systemic infection) or direct (trauma, surgery)
- Metaphysis of long bones (eg tibia, femur, humerus).
- Usually staph aureus
b) IVDU, trauma, fracture, prosthesis, SCD, diabetes, septic arthritis/cellulitis, sepsis, old age, debilitation, haemodialysis
c) Fever, painful limb, tender, non-weight bearing (if legs)
- (usually acute)
d) - Bloods: FBC, CRP, blood cultures
- Imaging: MRI (gold standard)*
*XR may not show changes for ~ 1 week (patchy osteopenia, periosteal reaction, necrosis, etc.)
e) - Surgical debridement and ABx in all cases
- ABx - IV for 2 weeks, then consider switching to oral
- Acute non-MRSA: Fluclox + fusidic acid (4 - 6 weeks)
- Acute MRSA: Vancomycin + fusidic acid (4 - 6 weeks)
- Chronic: 12 weeks ABx + removal (12 weeks)
- Treatment guided by cultures, clinical findings and inflammatory markers
Post-fracture complications
Short-term: Haemorrhage, Ischaemia, Infection
Long-term: Arthritis
Types of fracture.
- Stable fracture: The broken ends of the bone line up and are barely out of place.
- Open, compound fracture: Breaks the skin (need ABx)
- Transverse fracture: horizontal fracture line.
- Oblique fracture: an angled pattern.
- Spiral fracture
- Green-stick (in kids)
- Comminuted fracture: three or more pieces.