Acute Abdomen Flashcards

1
Q

Peritonitis aka

Causes 4

Presentation 
5 Presentation features
1 Clinical FInding
5 signs of infections
1 crude test

Investigations
Bedside- 4
Bloods- 6
Imaging- 3

Management - cause dependant
intitial management -5
if unwell- 3

What presents similarly to this but doesn’t require a laparotomy?

A

‘Hot Belly’

Causes: 
Perf. peptic/duo ulcer
Diverticulum
Appendicitis 
Cholecystitis 

Presentation:
- rebound tenderness
- guarding (invol tensing of abdo)
- prostration (position knees and head on ground)
- no bowel sounds
- Abdo distention + rigid
- board-like abdo
- inc. HR, inc. BP, Fever, Dehydration, Anorexia
Test = +ve cough test (pain when ask to cough)

Investigations
bedside: Urine Dip, Preg, VBG/ ABG, ALWAYS ECG!
bloods: FBC, LFTs, CRP, Amylase, GROUP + SAVE, Blood Cult
Imaging: Erect CXR (gas under diaphragm), Ultrasound, CT abdo

Management
IV access + bloods, NBM, Analgesia, Urine dip, Consider imaging
unwell: IV fluids, Catheter, NG tube

extra- Pancreatitis

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2
Q
Acute Appendicitis 
Classical Presentation (2) 

Classic infection 6

3 signs and what they are

Differential Diag (M+F) 
G- 3
R- 3 
G- 4 
U- 2 

Investigations
Bedside- 2
Bloods- 2
Imaging- 2

Management 2

Complications 3

A

Classical Presentation:

  • Periumbilical pain which moves to RIF
  • To Mcburney’s Point (between umbilicus + Ant Sup il sp)
  • Tachy
  • Fever
  • Peritonism -> guarding + rebound tenderness or percussion tenderness
  • Anorexia is important!
  • Constipation (usual)
  • Vom is rare
  1. Rovsing’s Sign - Palp LIF causes RIF pain
  2. Psoas Sign- RIF pain with ext. of right hip
  3. Cope Sign- Pain on flexion and internal rotation in right, if appendix is close to obturator internus

DD

  • Gynae: ovarian cyst rupt, ectopic preg, pelvic inflammatory disease
  • Renal: UTI, ureteric stones, polynephritis
  • GI: IBD, Meckel’s Diverticulum/ Diverticular disease, Cholecytsitis, Perforated ulcer
  • Urological: Test torsion, epididymo-orchitis

Investigation
Bedside- preg test, urinanalysis
Bloods- FBC (neutrophil leukocytosis/ inc. CRP
Imaging- USS not always useful, CT good diag but not always needed

Management

  • Laparoscopic appendicectomy (gold standard)
  • Antibiotics- piptaz = pipercillin/ tazobactam

Complications

  • perforation
  • appendix mass: when the omentum covers so surgery/ antibiotics needed but rule out colonic tumour
  • Appendix abscess: if mass isn’t resolved this can occur, needs draining/ surgery or antibiotics
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3
Q

Acute Pancreatitis
What is it? 2

Causes: I GET SMASHED

Presentation 
-
- Eases with?
-
Exam 3 including 2 signs and why!

-
-

Investigations
- What to measure?

Modified GCS for severity- Called?

  • PANCREAS
  • …. indicates pancreatitis
  • mortality % (4)
  • When is the test done?

Management

  • initial 4
  • concepts of later 1

Complications

  • Local LATE 4/6
  • Systemic EARLY 4/6
A
  • Self- perpetuated pancreatic enzyme- mediated autodigestion, odema, fluid shift -> Hypovolaemia
  • Extra-cellular fluid trapped in gut, peritonium and retroperitonium

Causes
I-Idiopathic S- Steroid
M- Mumps (virus: HIV, Coxsachie)
G- Gallstones A- Autoimmune
E- Ethanol S- Scorpion venom
T- Trauma H- Hyper.. lipidamia, calcamia,
hypothermia
E- ERCP or Emboli
D- Drugs (thiazide diuretics,
azathioprine)
Could also be preg, anatomical defect

Presentation

  • Epigastric pain WHICH RADIATES TO BACK
  • Eases with sitting forward or fetal position
  • Nausea and vom assoc

Examination
- Epigastric tenderness +/- guarding
- Cullens Sign: Bruising around umbilicus
- Grey- Turners Sign: Flank bruising
from retroperitoneal haemorrhage or vessel autodigestion

Complications?

  • ARDS
  • Hypovolaemia leading to AKI
  • Pleural effusions

Investigations

  • Lipase is more sensitive than amylase but not as readily available
  • Amylase x3 upper limit of normal is very suggestive
Modified GCS- Glasgow Imrie (gallstones or alcohol)
P- PaO2 < 8kPa
A- Age  >55y/o
N- Neutrophilia WBC > 15 x10^9 /L
C- Calcium <2 mmol/L
R- Renal Funct    Urea> 16mmol/L
E- Enzymes   LDH> 600iu/L     AST> 200iu/L
A- Albumin < 32g/L
S- Sugar  Blood Glucose > 10mmol/L 
 - 3+ indicates pancreatitis 
- mortality 0-2 2%, 3-4 15%, 5-6 40%, 7-8 100%
- at 48 hours
Management 
Initial: 
Analgaesia (opioid)
Fluid resus (crystalloid),
Monitor fluid balance (catheter)
NBM -> NG tube 
  • concepts of later: treat underlying cause ERCP

Complications
Local: LATE > 1 week
- Pancreatic Pseudocyst- collection of enzymes, blood, necrotic tissue surrounded by fibrotic/ vascular wall, forms weeks after episode and can be incidental or mass found
- Pancreatic Necrosis- Inflamm causing inchaemia/ infarct of pancreas. suspect of persistent systemic inflam >7days
- Bleeding -> eroded major vessel eg splenic artery
- Thrombosis-> splenic or gastroduo artery, or colic branches SMA
- Abscess
- Fistulae
- Recurrent oedematous pancreatitis

Systemic:

  • Disseminated Intravascular Coag (DIC)
  • Acute Resp. Distress Synd (ARDS)
  • Hypocalcaemia
  • Hyperglycaemia
  • Sepsis
  • Renal Failure
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4
Q
Diverticular Disease 
Definitions 
- Diverticulum 
- Diverticulosis 
- Diverticulitis 
- Diverticular bleed 
- Diverticular disease

Presentation - Diverticular disease 4
Presentation- Diverticulitis 6

Complications 5/7

Surgical Management of diverticulitis? main procedure? and 4 stages
- two more managments to complications
Non-surgical Management of diverticulitis 5
mild attack 2
not tolerated 4/5

A

Definitions

  • Diverticulum- outpouching of bowel, usually sigmoid affected but can affect whole bowel.
  • Diverticulosis- symptoms arising from diverticula
  • Diverticulitis- inflam of diverticula
  • Diverticular bleed- erosion into vessel causing large vol painless bleed.
  • Diverticula disease- symptoms arising from diverticula

Presentation - Diverticular disease

  • Intermittent lower abdo pain
  • Colicky
  • Relieved by defecation
  • Altered bowel habit (nausea/ flatulence)

Presentation- Diverticulitis
- Acute abdo pain, sharp
- LIF
- Worse on movement
- Localised tenderness
- Systemically unwell (pyrexia, inc WBC, ESR, CRP)
perforation- localised or general guarding

Complications

  • Colovesical fistuala: tract between bowel + bladder
  • Colovaginal fistula: ‘’ bowel + vagina
  • Perforation
  • Haemorrhage: large rectal bleed painless
  • Post-infection from suture
  • Abscess: persistent with swinging fever, leukocytosis, local sign such as rectal mass but if not then pus can be under diaphragm

Surgical Management: Sigmoid colectomy with formation of end colostomy or Hartmanns procedure
Stage 1: pericolic/ mesenteric abcsess NOT
Stage 2: walled off/ pelvic abscess SOMETIMES
Stage 3: Generalised purulent peritonitis SURGERY
Stage 4: faecal peritonitis SURGERY
- If haemorrhaging embolization is needed
- If abscess then will need draining under CT

Non-surgical Managment 
Mild attack: home with bowel rest (fluid only) and antibiotics PO
Not tolerated: admit with 
- IV Fluids
- Analgaesia 
- NBM
- IV antibiotics
- (Antispasmodic eg mebeverine)
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5
Q

Biliary Disease
-Draw anatomy of biliary system

  • Different types of gallstones
    1) Not as common and what causes it?
    2) Most common and what causes it?
    3)
  • Different conditions caused by gall stones. WHERE IS THE STONE? (3 name and def.)
  • Presentation and management for each
    1) Presentation 5 Management 3
    2) Presentation 4 + 1 Sign! Management 3
    3) Presentation TRIAD + 3 due to… Management 3
A

L/R hepatic duct, common hepatic duct, cystic duct joins, common bile duct, pancreatic duct joins, Ampulla of Varter +Sphincter of Oddi into 2nd portion of Duo

Types
- Pigment stones: small, friable, irreg cause- haemolysis
- Cholesterol Stones: large, yellow, usually solitary
cause = excess cholesterol, 3Fs: Fat, forty, female
- Mixed Stones: faceted (calcium salt pigment, cholesterol)

3 conditions caused by gall stones

  • Biliary Colic- Gallstones impacts neck of gallbladder
  • Acute Cholecystitis- Inflam of gallbladder
  • Acute Cholangitis- Stone blocks CBD

Presentation
- Biliary Colic: AFTER EATING, Dull, colicky pain, RUQ, assoc nausea + vom.
Manage: Analgesia, Lifestyle (low fat, weight loss, exercise), Elective laparoscopic cholecystectomy

-Acute Cholecystitis: systemic features (lethargy, fever), constant RUQ pain +/- Guarding, Murphy’s +ve! (large inhale and palpate RUQ +ve is pain)
Manage: Antibiotics, Analgesia, Laparoscopic cholecystectomy within 1 week

  • Acute Cholangitis: Charcot’s triad Fever, Jaundice, RUQ pain, Itching, pale stool, dark urine due to obstructive jaundice! ( Reynold’s Pentad + Hypotension + Delerium/ Confusion)
    Management: IV anti-biotics + treat sepsis, ERCP (endoscopic Retrograde Cholangiopancreatography- endoscopic removal of stone), Laparoscopic cholecystectomy
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6
Q

Know the difference in blood results for the 3 biliary conditions:
- WCC, CRP, Bilirubin, ALT, ALP what is happening with each?

Presentation table (Simple Revision tool)

  • Biliary Colic
  • Acute Cholecystitis
  • Acute Cholangitis

Imaging for Biliary Disease 2

A

Biliary Colic: WCC, CRP, Bilirubin, ALP, ALT NORMAL
Acute Cholecystitis: Inc WCC + Neut, Inc CRP but Bilirubin, ALP, ALT normal
Acute Cholangitis: Inc WCC + Neut, CRP, Bilirubin, Inc. Inc. ALP, ALT Inc./ normal

Presentation

  • Biliary Colic: RUQ pain but NO fever or jaundice
  • Acute Cholecystitis: RUQ and Fever but NO jaundice
  • Acute Cholangitis: ALL 3!

USS (cant see if passed to CBL) so…
MRCP (MRI of biliary)

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7
Q

Small Bowel Obstruction
5 Cardinal Presentations of Obstructions
3 Presentation specific to Small Bowel

Two main types:
Mechanical-
Functional-

Causes:

  • Intrinsic 5
  • Extrinsic 4 (The big two are within here!)
  • Intraluminal 3

Investigations:

  • Bedside 1
  • Bloods 4
  • Imagery 1

Early non-surgical management of small bowel obstruction 4

 Provide a broad overview of the surgical options for managing a patient with small bowel obstruction **

A
Small Bowel 
Presents: 
Cardinal Signs of Obstruction:
- Vom + Nausea
- Annorexia 
- Constripation
- Abdo distention 
Small Bowel
- vomiting occurs early
- less distention
- pain higher

Two main types:
Mechanical- A blockage within the lumen
Functional- No block in the lumen, no peristalsis

Causes:
Intrinsic:
- Inflammatory stricture (Crohns, Gasteroenteritis, TB)
- Neoplasia (Primary= adenocarcinoma, lymphoma, GIST or secondary)
- Vascular lesion
- Hematoma (coag, trauma, blood dyscrasias)
- Intussusception

Extrinsic:

  • ADHESION (75%) (fibrous scar tissue which joins two surfaces but are usually separate due to surgery)
  • Hernias (external or internal)
  • Endometriosis
  • Heamatoma

Intraluminal:

  • Gallstone ileus
  • Bezoars- collection of partially digested material that collects in the GI
  • Foreign Body

Investigations:

  • Bedside: AXR
  • Bloods: FBC, Amylase, U+E (monitor electrolytes), Group and save
  • Imagery: CT abdo with gastrografin (iodine contrast)

Non- surgical management early
- ‘Drip and Suck’ (NG tube and Fluid resus) Analgaesia, catheterise

Surgery
- laperotomy or laperscopic removal

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8
Q
Large Bowel Obstruction 
presentation 1
Identify the causes of large bowel obstructions
Mural 2 (big one)
Intramural/ within lumen 1 
Extramural 2

Provide a broad overview of the surgical options for managing a patient with large bowel obstruction **

Definitions
What is Ileus? and what examination finding?
Mechanical

Simple
Closed loop What is it and a complication?
Strangulated What is it and 3 presentations?

Paralytic ileus
What is it?
4/6 causes SHUT PAS

Psuedo- obstruction
What is it? And what else is it called?

Sigmoid Volvulus
What is it? Key clinical fact?

A

Presentation:

  • pain constant
  • Distension

Causes
Mural:
- Colorectal Cancer
- Diverticular Stricture

Intramural:
- Faecal impaction

Extramural:

  • Hernia
  • Volvulus - sigmoid volvulus looks like coffee bean as it twists on its mesentry

Surgical Options Large bowel
- Endoscopic stent (for malignancies)

Ileus- functional obstruction from dec. motility, bowel sounds abscent
eg paralytic ileus- caused by abdo surgery, pancreatitis, spinal injury, hypokalaemia, hyponatrraemia, ureamia, peritoneal sepsis, drugs (trycyclic anti-depressants)

Mechanical- Physical block within the lumen of bowel

Simple- one ob point and no vascular compromise
Closed loop- two ob points, therefore distended, could perforate
Strangulated- Blood supply comp, sharper + localised pain (peritonism), could also have fever, inc WCC, bowel ischaemia signs

Paralytic ileus- no peristalsis causes include: abdo surgery, spinal injury, tricyclic antidepressant, hypokalaemia, uraemia, peritoneal sepsis

Psuedo- obstruction- resembles mech. bowel ob but no lesion and called Ogilvie’s Syndrome
Sigmoid Volvulus- bowel twists on its own mesentry, severe rapid strangulation obstruction in elderly, constipated and looks like coffee bean on AXR

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9
Q

Glasgow Imrie used for Pancreatitis

Write it out

Interpret results

When is the test done?

Causes of drug-induced pancreatitis
FATSHEEP pneumonic

A
Modified GCS- Glasgow Imrie (gallstones or alcohol)
P- PaO2 < 8kPa
A- Age  >55y/o
N- Neutrophilia WBC > 15 x10^9 /L
C- Calcium <2 mmol/L
R- Renal Funct    Urea> 16mmol/L
E- Enzymes   LDH> 600iu/L     AST> 200iu/L
A- Albumin < 32g/L
S- Sugar  Blood Glucose > 10mmol/L 

Results

  • 3+ indicates pancreatitis
  • mortality 0-2 2%, 3-4 15%, 5-6 40%, 7-8 100%

Done at 48 hours

Causes of drug-induced pancreatitis
A good mnemonic for drug-induced pancreatitis is FATSHEEP:

Furosemide
Azathioprine/Asparaginase
Thiazides/Tetracycline
Statins/Sulfonamides/Sodium Valproate
Hydrochlorothiazide
Estrogens
Ethanol
Protease inhibitors and NRTI (Nucleotide Reverse Transcriptase Inhibitors) used in HIV
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