Acquired & Developmental Disturbances Flashcards

1
Q
  • What is “Concrescense”
  • Which teeth are most affected?
A
  • joined only by the cementum
  • maxillary molars

p.42 A&M

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2
Q

What is the rate of occurence of cleft lip or palate

A

1 in 1000

p.39 A&M

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3
Q

why is tooth abscess from primary teeth is more concerning than permanent teeth?

A

drainage through the pulp chamber of a primary tooth is almost impossible if subperiosteal pus is present but permanent tooth is possible through pulp chamber with proper access

p.39 A&M

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4
Q
  • What are the bacteria invovled in dental infection?
  • What are the 2 broad categories?
A
  • 2 categories = gram + facultative & gram - anaerobic
  • Gram + Facul = streptococcus, staphylococcus, Actinomyces (SSA)
  • Gram - anae = Prevotella, Treponema, Aggregatibacter, Fusobacterium (PTAF = pulp therapy as fuck)

p.41 A&M, Slide 130 of core curriculum read

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5
Q

what is the most affected area with “fusion”?

A

maxillary central and lateral incisors (most frequently affected teeth)

fusion = fewer teeth
gemination = normal counts

p.42 A&M

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6
Q

What is another name for geminatino?

A

Schizodontism

p.43 A&M

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7
Q
  • What is another name for Dens Invaginatus?
  • Which teeth are common?
A
  • Dens in Dente
  • Maxillary lateral incisor –> Maxillary canine –> other Mx molars

p.43 A&M

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8
Q
  • What is hypophosphatasia?
  • What does it cause?
A
  • inherited metabolic disorder which is deficient in ALP (ALPL) enzyme (impaired mineralization of tooth and bone)
  • premature exfoliation of the primary teeth (primary premature loss of anterior primary teeth associated with deficient cementum)

p.46 A&M

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9
Q
  • What is hypophosphatemia? aka?
  • what are clinical and dental manifestation?
A
  • disorders of Vit D metaboilsm or action. X-linked
  • Vit D deficient rickets
  • Clinical = short stature + bowling of the lower legs in affected boys
  • Dental = periapical radiolucency, abscesses due to abration of thin hypomineralized enamel which exposes pulp horn
  • VDRR - 25% vit D resistant rickets were affected by abscess in primary teeth but prophylactic pulps aren’t recommended (only 44% success rate)

p.52 A&M

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10
Q
  • What is another name for Cherubism?
  • how is it inherited?
  • how does it present?
A
  • familial fibrous dysplasia
  • autosomal dominant
  • symmetric enlargement of the jaw that may be noted at early age. multilocular area of bone distruction (soap-bubble lesions)
  • permanent teeth are usually ectopic and being displaced by the growing lesion.
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11
Q
  • What is acrodynia?
A
  • acrodynia or pink disease results from the exposure of young children to minute amounts of mercury. Pain and redness as extremities
  • dental amalgam restoration do not cause acrodynia
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12
Q
  • What are the stages of amelogenesis?
  • enamel hypoplasia vs. hypocalcification
A
  • Enamel matrix secretion by ameloblasts (apposition) –> calcification –> maturation
  • enamel hypoplasia = issue with matrix secretion (stage 1)
  • hypocalcification = issue with stage 2 or 3

p.56 A&M

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13
Q

what are characteristics of enamel hypoplasia vs. hypocalcification

A
  • enamel hypoplasia = pitting of the enamel surface, developmental of horizontal lines

p.56 A&M

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14
Q

which vitamins deficiency causes enamel hypoplasia?

A

Vitamins A, C, D, Ca, Phosphorus

p.57 A&M

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15
Q

what is plumbism?

A

lead poisoning (it can cause enamel hypoplasia)

p.58 A&M

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16
Q

what is “turner tooth”

A

turner tooth is the name for the tooth that has enamel hypoplasia due to local infection (mostly premolars)

p.58 A&M

17
Q

what are some causes of MIH?

A
  • MIH = molar-incisor hypoplasia
  • asthma, pneumonia, upper respiratory tract infection, otitis media, Abx use

p.62 A&M

18
Q
  • What gene mutation causes dentinogenesis imperfecta (DI)?
  • what are type of DI?
A
  • DSPP gene mutation (autosomal dominant)
  • type 1 = associated with OI (syndomic)
  • type 2 = non-syndromic, most common type
  • type 3 = bradywine type (non-syndromic, rare)

p.64 A&M

19
Q

what are treatment options for DI?

A

the placement of SSC on primary posterior teeth may be considered as a means of preventing gross abrasion of the tooth structure

p.65 A&M

20
Q
  • what is anodontia?
  • what is oligodontia?
  • what is hypodontia?
A
  • anodontia = complete failure of the teeth to develop (super rare)
  • oligodontia = missing more than 6
  • hypodontia = missing less than 6

p.70 A&M

21
Q

which genes are responsible for non-syndromic hypodontia?

A

WNT10A

p.70 A&M

22
Q
  • what are the most common teeth missing in permanent dentition?
  • which teeth are commonly present in permanent dentition?
    (most likely to least likely)
A
  • 3rd molar –> Md 2nd premolar –> Mx lateral incisor –> Mx 2nd premolar
  • Central incisors –> 1st perm molar –> canine

p.70 A&M

23
Q

Regarding displaced canines:
- which gender is more common?
- which way commonly displaced in %?

A
  • Female > male by 2-3x
  • 85% = PDC - palatally dispalced canine
  • 15% = laterally

p.71 A&M

24
Q

Regarding ED (ectodermal dysplasia):
- what is hypotrichosis?
- what is hypohydrosis?
- what is asteatosis?

A
  • hypotrichosis = sparse, absent hair growth
  • hypohidrosis = difficulty cooling down, deficiency in sweat glands
  • asteatosis = dry, cracked, scaly skin (often itchy)

p.72 A&M

25
Q

what is consanguinity?

A

people has blood relation (1st cousins getting married)

p.73 A&M

26
Q

what is “wiktop syndrome”?

A

it is type of ectodermal dysplasia (ED):
hypoplastic nails + hypodontia

p.74 A&M