ACLS Flashcards
best place to survive cardiac arrest
vegas
Cardiac arrest rhythms
4
- VF
- pVT
- PEA
- Asystole
immediate goal ACLS
return of spontaneous circulation (ROSC)
Cardiac arrest survival dependens on
BLS +/- ACLS
CPR cycle
Start CPR: give O2 + attach monitor (defib)
2 min cycle
End of cycle: pulse + rhythm check
Drug therapy:
* IV/IO EPI every 3-5 min
* Amio/Lido if refractor VF/pVT (try 2-3 shocks first)
Treat reversile causes
quality CPR: 100-120/min; 2cm deep
Medication role in ACLS
No proven benefit
Only specific therapy proven to increase surivial to discharge = defib of VF/pVT
Shockable rhythms
Ventricular Fibrilation
pulseless Ventricular Tachycardia
Non-shockable rhythms
Asystole
Pulseless electrical activity
VT/VF treatment
CPRx2min
Shock #1: CPR 2min + IV/IO access
Shock #2: CPR 2 min + EPI 3-5min
* Consider advanced airway capnogrpahy
Shock #3: CPR 2min + Amio/Lido+ treat reversible causes
if non-shockable rhythm - CPR+ EPI
Asystole/PEA treatment
EPI ASAP
CPRx2 min + EPI 3-5 min
* Consider advanced airway capnogrpahy
CPRx2 min, treat reversible causes
If still no signs of ROSC: continue CPR/EPI
Medication ROA
- IV
- IO
- ET*
* NAVEL drugs
NAVEL drugs
Naloxone
Atropine
Vasopressin
Epi
Lidocaine
Endotracheal ROA considerations
give 2-2.5x of IV/IO dose down ET tube (lung absorption)
Requires dilution 5-10ml SWFI or NS
Vasoactive agents role in ACLS
Epinephrine
vasoconstriction = organ perfusion (cerebral/coronary)
increases arterial/aortic diastolic pressures
Epinephrine dose
1mg IV/IO every 3-5 min
Give ASAP for non-shockable rhythms for ROSC
Antiarrythmics role in ACLS
Help cell re-establish electrical activity
normalize abnormally depolarizing/conducting heart cells
Poor evidence to suggest routine use = improve survival
Lido ≥ Amio
Magnesium: torsades (+) VF/pVT only
Amiodarone dose
VF/pVT, stable VT with pulse
VF/pVT: 300 mg IV bolus
* may repeat 150 mg IV bolus in 3-5 min
* flush with 20 ml NS for circulation
if stable VT (pulse)
* 150mg IV over 10 min
Amiodarone ADR
bradycardia, hypotension d/t diluent
* consider adding vasopressor to minimize hypotension
QTc prolongaiton - avoid use if hx, opt lidocaine instead
Lidocaine dose
weight based dosing
VF/pVT, stable VT with pulse, ROSC
VF/pVT: 1-1.5 mg/kg IV or IO
* may repeat 0.5-0.75 mg/kg every 5-10 min
stable VT with pulse
* 0.5-0.75 mg/kg IV
with ROSC: give as drip to prevent arryhtmia
* infuse 1-4 mg/min
Magnesium dose
dose not well established
generally: 2g IV bolus
flush with 10-20ml NS
if pulse: infuse
no pulse: bolus
wide complex QRS
Torsades (VF/pVT)
reversible causes of arrest acronym
H & T
H’s
reversible causes of arrest
hydrogen ion
hyperkalemia
hypothermia
hypoglycemia
hypovolemia
hypoxia
T’s
reversible causes of arrest
Tension pneumothorax
Tamponade,cardiac
Toxins
Thrombosis
* pulmonary
* coronary
Hypovolemia tx
loss of effective circulating volume
* trauma, blood loss
restore volume:
Lactated ringers
Normal saline
PRBC
other blood factors
Hypoxia tx
lack o2
give 100% O2 mask
Hydrogen ion Tx
H+ acidosis, acidemia
Severe metabolic acidosis
* No buffer/bicarb
Pre-existing metabolic acidosis (pH <7.1-7.2)
* consider bicarb
Hypothermia Tx
CPR + restore body temp simultaneously
Do not stop CPR until core temp reached
Hyperkalemia tx
- stabilize myocardial membrane
* Calcium Cl or gluconate - intracellular K+ shift
* Sodium bicarb
* Insulin + dextrose if hypo
* Albuterol neb not feasible - Excretion once ROSC
* diuresis, kayexalate, dialysis, potassium binding resins
renal, dialysis, drug induced
Toxin tx
Opioid: IV naloxone push
Local anesthetic tox: lipid emulsion
TCA: sodium bicarb
etc
What is the utility of bicarb in ACLS?
there are three
- Hyperkalemia
- TCA toxicity
- Severe pre-existing metabolic acidosis - maybe
Cardiac Tamponade tx
needle
relieve pericardium fluid
Tension pneumothroax tx
identified by ultrasound/CXR
Needle
release air
caused by lung injury, broke ribs, trauma, etc
Thrombosis tx
pulmonary, cardiac
PE: alteplase/tenecteplase
* ECLS, ECMO catheters
MI: tenecteplase, alteplase, PCI