Abnormal EKG Flashcards

1
Q

Heart rate 100 beats/min that originates with the SA node. Short distance between end of t-wave and start of p-wave

A

Sinus tachycardia

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2
Q

What can cause Sinus tachycardia?

A

fever, any sympathetic stimulation including pain, hyper thyroids, hypovolemia, and anemia

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3
Q

What can cause Sinus Bradycardia?

A

hypothermia, hypothyroidism, increased vagal tone, and marked intracranial hypertension.

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4
Q

Sinus bradycardia is not normal in well conditioned athletes, true or false?

A

False

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5
Q

A premature beat caused by an ectopic focus in the atria that fires before the SA node has a chance to depolarize is a condition called

A

Premature atrial contraction (PAC)

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6
Q

Which wave does PAC change?

A

The P-wave

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7
Q

What can cause premature atrial contractions

A

Smoking, lack of sleep, too much caffeine and alcoholism

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8
Q

a regular, fast heart rate (120-220 beats/minute) that begins and ends suddenly that can originate from any area above the ventricles

A

Paroxysmal Superventricular tachycardia

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9
Q

What is the cause of paroxysmal superventricular tachycardia?

A

may be triggered by a premature beat from an ectopic focus that continues to fire.

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10
Q

An excessively fast heart rate with an atrial rate of up to 300 beats/mnute and a ventricular rate of up to 125 beats/minute because the atria are beating so fast that not every action potential will be conducted through to the ventricle

A

Atrial Flutter

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11
Q

What usually causes an atrial flutter?

A

and ectopic focus

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12
Q

What usually causes an atrial flutter?

A

and ectopic focus

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13
Q

What is it called when there are no P- waves, but the QRS complex is normal?

A

Atrial fibrillation

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14
Q

What can cause atrial fibrillation?

A

Ectopic action potentials in the atria

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15
Q

The QRS interval is prolonged and they have an exaggerated voltage, there is no P wave and the T wave is inverted.

A

Premature Ventricular contraction (PVC)

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16
Q

How can we cause PVC?

A

lack of sleep, too much caffeine, irritability of the myocardium and can occasionally be seen with coronary thrombosis

17
Q

condition that consists of many PVC’s one after another that occurs when there is considerable damage to the AV node or ventricles

A

Ventricular Tachycardia (v-tach)

18
Q

No coordinated contraction of the ventricles and if not corrected the subject will die within a few minutes. No recognizable waves and no rhythmic contractions. Typically caused by an ectopic focus in the ventricle

A

Ventricular fibrillation (V-fib)

19
Q

Prolonged PR interval (longer than 0.2 seconds) every action potential that arrives at the AV node is conducted to the ventricles, but there is a delay in the conduction

A

First degree block

20
Q

What is one cause of First degree block?

A

Inflammation of the AV bundle

21
Q

When of the P-waves trigger a QRS complex and others don’t with a constant ratio of P-waves to QRS complexes like 2:1 or 3:1

A

Second degree block

22
Q

None of the P-waves get through the AV node. The QRS complex becomes completely dissociated from the P-waves. the atria have a rate around 100 beats/minute and the ventricles will have a rate around 40 beats/minute. The ventricles are now paced by the auto rhythmic cells in the AV bundle.

A

Third degree block

23
Q

What are some causes of 3rd degree block?

A

ischemia of the AV fibers or compression of the AV bundle by scar tissue or by calcified portions of the heart

24
Q

which phase of blood pressure is a loud clear tapping sound is heard that increases with intensity as the cuff deflates. The phase begins just as the pressure cuff drops below the systolic pressure of the subject’s blood

25
Which phase is best described as a series of murmurs?
phase 2
26
which phase of blood pressure is there a loud thumping sound similar to phase 1 but less clear
phase 3
27
which phase of blood pressure is there a muffled thumping sound?
phase 4
28
which phase of blood pressure is there silence and the pressure cuff reaches diastolic pressure of the subjects blood?
Phase 5
29
stains orange/ red, general inflammation, bilobed
Eosinophil
30
Differentiates into B and T cells, round nucleus, thin ring of cytoplasm ,synthesizes antibodies
Lymphocyte
31
Most numerous, first line of defense in bacterial infection
neutrophil
32
Have similar properties to mast cells, release heparin and histamine, LEAST NUMEROUS
basophil
33
Can become a macrophage
Monocyte
34
sound of AV valves closing
S1
35
sound of Semilunar valves closing
S2
36
sound of rapid filling of rigid ventricles
S3
37
Atria contracting with excessive force
S4
38
When does S4 occur?
between S1 and S2
39
when does S3 occur?
following S2