Abdominal imaging activities 7 -12 Flashcards
briefly describe the etiology of a hypertrophied column of Bertin explain how they are differentiated from pathology
echogenic line that extends from the renal sinus to perinephric fat. The defect is typically located at the junction of the upper and middle thirds of the kidney
During normal development, two parenchymal masses called ranunculi partially fuse. Parenchymal junctional defects occur at the site of fusion and must not be confused with pathologic processes (e.g., renal scar, angiomyolipoma). The junctional parenchymal defect is most often located anteriorly and superiorly, typically at the junction of the upper and middle thirds of the kidney, and can be traced medially and inferiorly into the renal sinus. Usually, it is oriented more horizontally than vertically and therefore it is best appreciated on sagittal scans. Junctional cortical defects are more often shown within the right kidney, although left junctional cortical defects may be detected with favorable acoustic windows.
briefly describe the etiology of junctional parenchymal defect and explain how they are differentiated from pathology
Normal variant of unresorbed polar parenchyma from one or both of the two subkidneys that fuse to form the normal kidney.
Sonographic features
indentation of the renal sinus laterally’
bordered by junctional parenchymal defect
located at junction of upper and middle thirds
Continuous with adjacent renal parenchyma
similar colour flow to surrounding parenchyma (demonstration of arcuate arteries)
contains renal pyramids
less than 3cm in size
List the most common anomalies of the kidneys
- Hypoplasia:
- Hyperplasia:
- Ectopia:
- Crossed renal ectopia:
- Horseshoe kidney:
- Renal agenesis:
- Supernumerary kidney:
- Duplex collecting system:
- Pelviureteric junction (PUJ) obstruction: Hydronephrosis of the renal pelvis and collecting system down to the level of the proximal ureter is noted.
- Congenital megaureter: The distal ureter does not peritalse effectively, resulting in a spectrum of findings from minor dilatation of the distal ureter to significant hydronephrosis.
- Aberrant vessels: Most commonly at the level of the PUJ a vessel may be seen causing mechanical obstruction to the ureter, resulting in hydronephrosis. This may also occur if the ureter passes posterior to the IVC.
Hypoplasia
A small but otherwise normal kidney results in some hypertrophy of the contralateral kidney to compensate. It is only a clinical concern if the variant is bilateral or if the dominant kidney is damaged.
Hyperplasia
A large but otherwise normal kidney results from increased workload following reduced function of the contralateral kidney. Focal forms of hypertrophy may occur as spared portions of a diseased kidney hypertrophy. Differentiation between focal hypertrophy and renal masses is difficult.
• Fetal lobulation: A lobular outline of the renal capsule is noted, especially in paediatrics. This embryological lobulation may persist and be noted in adults.
Ectopia
Failure to ascend or, in rarer cases, ascent into the thorax, results in an ectopic kidney. Once the renal fossa is found to be empty you should search diligently for an ectopic kidney before diagnosing renal agenesis. Pelvic kidneys are often beneath the bowel and malrotated, making them difficult to find and then image. A full urinary bladder may be of help as will a graded compression technique.
Crossed renal ectopia
If a kidney is absent and not seen in the pelvis, it may be located on the other side of the abdomen. It may be found in the contralateral pelvis or fused with the other kidney.
Horse shoe kidney
A horseshoe kidney appears as a kidney on each side of the abdomen, with the lower poles fused together across the midline. Clues to a horseshoe kidney are:
o slight inferior location of one or both kidneys;
o malrotation of the lower poles;
o no clearly defined lower pole to either kidney; and
o lower poles tend to ‘reach’ toward the midline.
Scanning in the midline reveals the isthmus of renal tissue superficial to the aorta. This is usually a bridge of renal tissue that is readily identifiable if you are looking for it. However, the bridge may consist of only a thin fibrous band and that is difficult to identify. Do not mistake the isthmus of a horseshoe kidney for para-aortic lymphadenopathy.
Renal agenisis
This may be diagnosed when all other attempts to locate the missing kidney have failed. However atrophic kidneys are sometimes difficult to identify so agenesis is a difficult diagnosis to make based on ultrasound examination.
Supernumery kidney
An extra independent small kidney functions separately from the two kidneys.
Duplex collecting system
This is the most common anomaly resulting from paired collecting systems of variable degrees.
Does the absence of a urinary jet indicate obstruction? Under what circumstance might a urinary jet, although present, not be seen?
Renal failure, incorrect probe position, incorrect colour Doppler settings and dehydration may cause a urinary jet to be present and not seen.
Describe three areas of ureteric narrowing which are common sites for a calculus to become lodged.
If a stone passes into the ureter, the calculus may lodge in three areas of ureteric narrowing: just past the UPJ; where the ureter crosses the iliac vessels; and at the UVJ (ureterovesical junction – where the ureter enters the bladder) . The very small diameter of the UVJ (1-5 mm) accounts for the large percentage of calculi that lodge within the distal ureter. Approximately 80% of stones smaller than 5 mm will pass spontaneously.
List other entities which may mimic a renal calculus.
including intrarenal gas, renal artery calcification, calcified sloughed papilla, calcified transitional cell tumor, alkaline-encrusted pyelitis, and encrusted ureteral stents.
List the sonographic appearances that may be seen in acute pyelonephritis.
renal enlargement
compression of the renal sinus
decreased echogenicity (secondary to edema) or increased echogenicity (potentially from hemorrhage)
loss of corticomedullary differentiation
poorly marginated mass(es)
gas within the renal parenchyma
and focal or diffuse absence of color Doppler perfusion corresponding to the swollen inflamed areas.
Describe the sonographic appearances of chronic pyelonephritis
- renal scarring
- renal atrophy
- renal cortical thinning
- compensatory hypertrophy of residual normal tissue (which may mimic a mass lesion)
- calyceal clubbing: secondary to retraction of the papilla from overlying scar
- thickening and dilatation of the calyceal system
- overall renal asymmetry
describe the Robson criteria for staging renal cell carcinoma
- I: Tumor confined within renal capsule
- II: Tumor invasion of perinephric fat
- III: Tumor involvement of regional lymph nodes or venous structures
- IV: Invasion of adjacent organs or distant metastases
List predisposing factors to ATN, which may lead to increased renal cortex echotexture.
If drugs, metals or solvents have caused ATN the kidneys will appear large and echogenic.
List possible causes of acute cortical necrosis
The cause is likely related a severe systemic illness resulting in transient intrarenal vasospasm, intravascular thrombosis or glomerular capillary endothelial damage. This may include severe haemodynamic shock (traumatic blood loss, postpartum haemorrhage, septic shock, venom toxin, transfusion reaction, severe dehydration), haemolytic uraemic syndrome (HUS) (multisystem thrombotic microangiopathic disease and renal transplantation.
briefly describe ACN sonographic appearances
At initial presentation the renal cortex is hypoechoic. As time goes on kidneys atrophy and the cortex may calcify.
Describe the formation and appearance of parapelvic cysts
Parapelvic cysts may originate from lymphatics or embryologic rests and appear as well-defined, anechoic, renal sinus masses. If they have haemorrhaged, internal echoes will be seen in the cysts.
explain how parapelvic cysts may be differentiated from hydronephrosis
Parapelvic cysts originate from the adjacent parenchyma and protrude into the renal sinus. Careful scanning will show the renal pelvis is separate from the parapelvic cyst.
explain what other imaging modalities may be useful in differentiating parapelvic cysts from hydronephrosis.
CT IVP is a useful alternative imaging in differentiating parapelvic cysts from hydronephrosis. A combination of non-contrast, portal venous and excretory phase imaging allows complete assessment of the renal tract and will show definitively the parapelvic cyst(s) do not communicate with the renal pelvis. MRI may also be helpful.
List other anomalies associated with autosomal dominant polycystic kidney disease.
- liver cysts (30%-60%)
- pancreatic cysts (10%)
- splenic cysts (5%)
- cysts in thyroid, ovary, endometrium, seminal vesicles, lung, brain, pituitary gland, breast, and epididymis
- cerebral berry aneurysms (18%-40%)
- abdominal aortic aneurysm
- cardiac lesions
- colonic diverticula