Abdominal Emergencies Flashcards
Abdominal complaints are what percent of ED visits? How do we diagnose them?
6% of ED visits. H&P + directed studies = Dx. MUST rule out life-threatening emergencies. Most common diagnosis is NONSPECIFIC.
Why AP is Tricky
Imprecise pain generation and transmission to the CNS.
Comorbid disease.
Medications/Polypharmacy.
Extra-abdominal pain
In considering the age of the patient, what diagnoses should we think of in 0-2 yo, 2-12yo, teens/adults, elderly?
0-2- Colic, GE, viral illness, constipation
2-12- Functional, appendicitis, GE, toxins
Teens to adults- Addition of genitourinary problems
Elderly- Beware of everything!
What are the types of Pain?
Visceral, Somatic & Referred
Describe Visceral Pain.
Stretch fibers in capsules or walls of hollow viscus that enter both sides of spinal cord. Crampy, achy, diffuse, poorly localized.
VAGUE
Describe Somatic Pain.
Fibers dermatomally distributed and enter unilaterally in the spinal cord. Sharp, lancinating, well localized
SPECIFIC
Describe Referred Pain.
Overlap of fibers from other locations. Pain is distant from site of generation. Symptoms, but no signs
RADIATING
What part of the physical exam tends to be the most helpful for abdominal pain?
Palpation; Tenderness versus pain, Start away from painful area. Look for guarding, rebound, masses.
The Physical Exam: Signs
Iliopsoas Obturator Rovsing’s Murphy’s Hop Test
T/F: A rectal exam is one of our most useful tools in diagnosing abdominal pain.
False, A Rectal exam adds very little beyond gross blood or melena.
What is the first step in the management of Abdominal Pain
Always start with ABC’s
What is one Laboratory Test with abdominal pain we have to always use with women?
Pregnancy test in women of child bearing age
Is the CBC helpful in diagnosing abdominal pain?
NO, it lacks sensitivity, and has no specificity. The CBC should not dramatically alter approach (tender is still tender).
How are various imaging procedures used with abdominal pain in the ED?
In Plain films we look for free air, obstruction, air-fluid, FBs
Ultrasound: No radiation exposure, Limited exam. Used as a rapid “yes or no” ED evaluations. May add doppler.
CT: Much more specific (Contrast vs. non-contrast) does have radiation exposure
MRI: Limited use for abd pain, Pregnant patients
Acute Appendicitis Pathophysiology:
Thought to be due a luminal obstruction (food, adhesions, or lymphoid hyperplasia). The obstruction leads to increase in intraluminal pressure. and eventually, the increased pressure leads to arterial stasis and tissue infarction.
Clinical Features of Acute Appendicitis:
1 symptom is abdominal pain. Classically, patients develop periumbilical pain followed by anorexia, NV, and low grade fever. As the illness progresses, the pain becomes more localized to the the RLQ.
Diagnosing Acute Appendicitis:
*MANTRELS + CT Abdomen and Pelvis w/ oral & IV contrast
Labs: CBC, BMP, UA
Pelvic Exam and/or Rectal Exam
Treatment of Acute Appendicitis
Antibiotics, Surgery or conservative management
Characteristics of Small Bowel Obstruction on Plain Film:
Small intestine is characterized by transverse linear densities that extend completely across the bowel lumen (plicae circulares).
Characteristics of Large Bowel Obstruction on Plain Film
The colon is seen peripherally in the abdomen, is larger in diameter, and contains short, blunt, and thick projections (haustra) that arise from the bowel wall and extend only partially into the lumen.
Small Bowel Obstruction (SBO) Pathophysiology:
ADHESIONS!!!! or Strangulated and incarcerated inguinal hernias.
Clinical Features of SBO:
Diffuse abdominal pain that is often crampy, colicky, intermittent and severe.Followed by vomiting and inability to pass flatus.
PE may show distended abdomen that is diffusely tender with occasional high-pitched bowel sounds on auscultation.
Diagnosis of SBO
3-View Plain Film AXR showing dilated loops of small bowel and AF levels.
Typically followed by CT AP to assess degree and location of obstruction.
Labs: CBC, BMP
Large Bowel Obstruction Pathophysiology:
Neoplasms are by far the most common cause of LBO, followed by diverticular disease then simoid volvulus.