Abdomen Flashcards
location of visceral pain and often accompanied with
vague and midline →nausea, pallor, diaphoresis
arises from the walls of hollow viscera and solid organs → due to abnormal stretching/distention, ischemia or inflammation
visceral pain
pain that is sharp in quality and is well localized
somatic pain
arises from the parietal peritoneum, mesneteric roots, and anterior abdominal wall due to chemical or bacterial inflammation
somatic pain
pain due to fibers from different organs returning to CNS overlapping with pathways from cutaneous sites which had similar embryologic origin
referred pain
Diaphragmatic irritation that refers pain to the shoulder via C4 due to splenic rupture/abscess, renal calculi, ruptured ectopic
Kehr’s Sign
pulsatile and well localized pain
burns, lacterations, fractures, infection, inflammations
somatic pain
aching and cramping, nonpulsatile, poorly localized pain or referred to distant locations
angina, hepatic distention, bowel distention, hypermobility, pancreatitis
visceral pain
spontaneous, burning, lancinating or shooting pain → may be distal or proximal to site of injury
complex regional pain syndrome, sciatica
neuropathic pain
stomach, 1st and 2nd parts of duodenum, liver, gallbladder, pancreas
epigastric area
3rd and 4th parts of duodenum, jejunum, ileumm, cecum, appendix, ascending colon, first two thirds of transverse colon
periumbilical area
last one third of transverse colon, descending colon, sigmoid, sectum, intraperitoneal GU organs
suprapubic area
two big history points to hit in patient with abdominal pain
social hx. (IVDA, Smoke, EtOH, GU, STD, pregnancy hx)
surgical hx.
respiratory arrest on inspiration during palpatino of RUQ [cholecystitis]
Murphy’s Sign
pain referred to RLQ on palpation of LLQ [appendicitis]
Rovsing Sign
Pain with internal rotation of flexed hip
Obturator Sign
Pain with hyperextension of hip
Psoas Sign
patients coming to ED for abdominal pain should be…
NPO
where are pediatrics most likely to have foreign body entrapment in their esophagus?
upper esophageal sphincter
where are adults most likely to have foreign body entrapment in their esophagus?
lower esophageal sphincter
sign that esophageal foreign body has > 80% chance of passing
if it reaches the stomach
MC cause of esophageal foreign body in adults
mechanical dysfunction
diameters of object that will not pass through the esophagus
> 2.5 cm wide and > 6 cm long
MC common GI foreign bodies in pediatrics
food boluses and other object (coins, toy, button battery, magnet)
how will esophageal foreign body present in an adult?
foreign body sensation → mild/severe pain
can’t swallow/handle secretions, drooling, spitting, vomiting
issue with foreign bodies in pediatrics patients
asymptomatic or can’t communicate
Worse cause scenario for foreign esophageal body in pediatric patient
In UES → tracheal impingement → stridor, respiratory compromise, death
sodium hydroxide is generated by current produced by the battery → 20 mm button batteries have higher capacitance and generate more current compared to other disc batteries
liquefaction necrosis
How can you differentiate button batteries from coins?
X-ray will show “double contour” of button battery
Time line for button battery:
liquefaction necrosis
perforation
LN → as early as 2 hours
Perforation → 6 hours
methods to remove button battery
endoscopy
Foley catheter removal
Magill forceps removal
Even though a button battery is removed, what can continue to cause injury in the body?
residual alkali
weakened tissues
You supect patient has foregin body in esophagus that you can’t see on X-ray, what should you order next?
CT
Giving this will supposedly help move esophageal foreign body
Glucagon
How do you usually remove esophageal foreign body? What if that foreign body is a magnet?
endoscopy
magnet → open exploratory laparotomy
which esophageal FB may pass alone?
distal smooth solitary FB
MC cause of esophageal perforation
iatrogenic (endoscopy and dilation)
Boerhaave Syndrome, Mallory-Weiss tear, trauma, FB, infection, ingestion
why does esophageal perforation have high mortality?
gastric contents leaking into mediastinum and pleural space, etc
acute, severe, diffuse , unrelenting chest/neck/abdominal pain that radiates to back/shoulder
Esophageal perforation
What is Mackler’s Triad? Indicates?
Vomiting, chest pain, SubQ emphysema
Esophageal rupture
Hallmark sign for esophageal rupture
Subcutaneous emphysema → will appear usually after 1 hour of onset
raspy, crunchy sound heard over precordium with each heartbeat
Hammon crunch
signs of esophageal rupture
tachycardia
tachypnea
NO HEMATEMESIS
What makes the pain worse with esophageal rupture?
swallowing or laying supine
Chest X-ray indicators for pneumomediastinum
SubQ emphysema mediastinal air fluid levels pleural effusion free air under the diaphragm pneumothorax
You see pneumomediastinum on CXR, what do you order next?
contrast esophagogram (+ water soluble agent) CT Chest with contrast
3 antibiotic options for esophageal rupture
piperacillin-tazobactam (Zosyn)
ceftriaxone + metronidazole (flagyl)
clindamycin
Esophageal rupture is a ____
emergency surgical consult
suspensatory muscle of duodenum, thin muscle connecting junction between duodenum, jejunum, and duodenojujenal flexure to connective tissue surrounding superior mesenteric artery and coeliac artery
Ligament of Treitz
MC location of GI bleed
Upper GI Bleed → proximal to ligament of treitz
PUD, erosive gastritis/esophagitis, esophageal/gastric varices, Mallory-Weiss tear, alcoholic, ASA/NSAID use
MC cause of lower GI bleed
diverticular disease, anal fissue, AVM, colitis (ischemic or infetious), polyps, malignancy, anticoagulant/NSAID, hemorrhoids
Signs of Upper GI Bleed
hematemesis, coffee ground emesis, melena
why is hematochezia worrisome in Upper GI bleed?
indicates rapid transit through intestines
subcutaneous emphysema with history of vomiting → presenting with retrosternal CP
Boerhaave’s Syndrome
where does Boerhaave’s most likely occur?
lower 1/3 esophagus → posterolateral distal esophagusepigastric
severe vomiting then painless hematemesis → 90% are self-limiting
Mallory Weis tear
Who are Mallory Weis tears MC in
patient with Cirrhosis
Describe the type of tear in in Mallory Weis
incomplete → only mucosa and submucosa tear
These are the result of portal HTN due to cirrhosis or banding
esophageal varices
Signs of shock
tachycardia/hypotension
poor perfusion
AMS