AANEM Monograph: NEE Flashcards

1
Q

Information provided by NEE

A

confirm dx. exclude mimics, identify unrecognized, localize, severity, pathophysiology, prognosticate

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2
Q

NEE technique

A

pull skin, insert needle (resting), withdraw to sub q then antigravity with insertion ID units morphology (duration/polyphasia/duraiton) recruitment (occ to interference pattern)

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3
Q

When should you quantitate

A

for mild/early processes

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4
Q

Coagulopathy avoid

A

diaphragm, TA, iliopsoas,fpl, pull skin avoid <30k plat

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5
Q

6 firing patterns

A

regular (<1% variation) - regular no change - regular with exponential change - irregular - semi rhythm (10% variation) - bursts

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6
Q

Origins: single fiber firing alone

A

endplate, fibs ( spike/psw), myotonic)

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7
Q

Origins: single fiber firing in groups

A

insertional - CRDS

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8
Q

Origin: Motor unit - individual spont

A

fasciculation - neuromyotonic - myoclonus - dystronia - stiffman

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9
Q

Origin: Motor unit - bursts spont

A

myokmic - synkinesis - termor - hemifacial spasm

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10
Q

Origin: voluntary

A

motor unit potentials

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11
Q

6 factors affecting Motor unit morphology morphology and synchrony

A

Reinnervation - collateral sprouting - loss of fibers - fiber splitting - atrophy - fiber regeneration

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12
Q

Recruitment frequency in limb and cranial

A

limb - 7-10 hz

cranial 16hz

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13
Q

Poor activation reasons

A

UMN, Pain, cooperation, strong muscle, two joint muscles (gastroc)

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14
Q

Goal rise time

A
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15
Q

When looking at 1-4 MUAPs note the

A

rise time < .6ms - duration (5-15ms) - polyphasia <5 - amplitude <20 - 200 microv - stability(MMAV)

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16
Q

increased insertional activity

A

> 200ms, seen in denervation and mytonic disorders

17
Q

fibrillation potentials reflect

A

muscle fiber diameter

18
Q

Main categories associated with fibrillation potentials

A

LMN - NMJ - Myopathies - muscle trauma

19
Q

disease associated with myotonic potentials

A

myopathy with or without clinical myotnia and neurogenic disorders

20
Q

CRDS usually indicated

A

chronic neurogenic, myopathic, normal in iliopsoas and biceps

21
Q

ALS: fascics without fibs =

A

not sufficient for dx

22
Q

situations associated with fascics

A

hyperthyroid, mestinon , normal in BFS, cramp fasciculation syndrome, neurogenic

23
Q

neurotonic discharge

A

interoperative nerve irritation

24
Q

2 situations for synkinesis

A

bell’s - arm-diaphragm

25
Q

First change in a neurogenic lesion

A

reduced recruitment

26
Q

4 situations that cause long duration MUAPs

A

increased fiber density, fiber number, loss of synchrony, collateral sprouting, reinnervation

27
Q

diseases causing long duration MUAPs

A

chronic neurogenic, myopathies

28
Q

diseases causing short duration

A

myopathy - nmj - early reinnervation or late stage atrophy - periodic paralysis

29
Q

diseases causing unstable MUAPs

A

NMJ, neurogenic (reinnervation/progressive) - inflammatory myopathies

30
Q

MUAPS in tremor fire in ___.

A

groups

31
Q

Patterns: Normal recruitment with short duration polyphasic MUAPs

A

with variation - severe NMJs occ primary myopathies -

without variation - primary myopathies

32
Q

Patterns: normal recruitment and normal muaps

A

normal and some metabolic myopathies

33
Q

Patterns: reduced mixed durations

A

chronic myopathies - no variation in acute neurogenic but subacute there can be

34
Q

Patterns: Reduced short duration polyphasic

A

without variation severe myopathy, end stage neurogenic disorder
with variation early/ongoing reinnervation

35
Q

Patterns: Reduced long duration polyphasic

A

chronic neurogenic or if variation then ongoing neurogenic lesion

36
Q

Patterns: Reduced mixed durations

A

rapid neurogenic lesion

37
Q

Patterns: Rapid recruitment

A

instability except for mild myopathies - long duration in chronic myopathies short duration in all other myopthies.