A for laparoscopic surgery (linda's PPT) Flashcards

1
Q

advantages of minimally invasive surgery

A
  • smaller incision: less surgical stress
  • less postop pain: fewer opioids, earlier ambulation, shorter hospital stay, rapid return of ADLs
  • same surgical outcomes: good for obese population and older/sicker populations
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2
Q

complications

A
  • vascular injury
  • sub-Q emphysema
  • gas embolism
  • capnothorax
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3
Q

implications

A

pneumoperitoneum
- hemodynamics
- pulmonary
- neurohormonal

patient positioning
- upper abd → reverse trend
- lower abd → trend

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4
Q

surgical procedure

A
  • use insufflation of CO2 to create pneumoperitoneum that allows surgical exposure and manipulation
  • insufflator that stops gas flow at a determined inra-abd pressure: < 15 mm Hg
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5
Q

why CO2?

A

noncombustible
soluble
increased safety margin

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6
Q

closed technique

A

blunt needle
check for placement
trocar

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7
Q

open technique (Hassan)

A

mini-laparotomy
trocar
direct visualization

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8
Q

cardiovascular effects d/t pneumoperitoneum: hemodynamics

A
  • increase in MAP, SVR, and HR-increase
  • Cardiac filling volumes- increase or no ▲ (compression of the liver & spleen)
  • cardiac index- decrease or no ▲ (increase afterload, decrease filling pressures)
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9
Q

cardiovascular effects d/t pneumoperitoneum: cardiac arrhythmias

A

brady or tachyarrhythmias
vasovagal response
can increase QT interval

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10
Q

respiratory effects

A

reduction in lung volumes and pulmonary compliance

diaphragm has cephalad displacement and is even worse in trendelenburg position

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11
Q

cardiovascular effects d/t pneumoperitoneum: sick patients

A

exaggerated response - sick patients
elderly - decreased MAP
Obese? no difference

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12
Q

absorption of CO₂

A
  • hypercarbia: acidosis & increased SNS activity
  • most in the first parts of insufflation, will stabilize eventually
  • if acidosis is more severe you have more hydrogen ions which will switch for K in cells and cause hyperkalemia and the potential for serious arrhythmias
  • myocardial depressant effect but the increase in SNS activity overrides this
  • increase cerebral blood flow, ICP, IOP→ dilated pupils with sluggish response
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13
Q

mechanical effects pneumoperitoneum

A

cephalad diaphragm
position effects severity
endobronchial intubation
impair AA gradient
shunt can be a big deal in sicker patients

increase peak airway pressures & increase in CO₂, as you increase patietns MV to compensate for increase CO₂ will increase there peak pressures

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14
Q

Sub-Q CO₂

A

RAPID rise in CO₂
risk factors: BMI < 25, long surgical time, operative approach

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15
Q

renal effects

A

transient increase in creatinine clearance
decrease RBF, GFR, and UO

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16
Q

hepatic effects

A

inconclusive results
decreased hepatic blood flow

17
Q

cerebral

A

increased CBF & ICP
increased CO₂→ increased CBF

18
Q

A management: if done without general, need to have pneumo with low pressures

A

local: usually for minor GYN surgeries & sterilization procedures

regional: need high sensory level, shoulder pain

general: most common, new IA- quick on/off, LMA?

induction: intubation with mechanical ventilation

19
Q

ventilation

A
  • increase RR or TV (usually RR)
  • PCV > VCV
  • increase MV by 15-35%
  • ARMS, PEEP (improve compliance) PEEP @ 10 increase PIP to 20 slowly, improve arterial saturations
  • NO₂: shouldn’t necessarily avoid it
20
Q

anesthesia managment

A

opioids: beware of treating HTN with opioids, maybe Remi good choice
fluid management: controversial, watch fluids - facial, pharyngeal, and orbital edema. if they are exchanging air around the tube you should be good
PONV - greater risk, may be as high as 72%
Post-op pain: origin - visceral not parietal, shoulder pain, non-opioids if possible

21
Q

primary complicaitons

A

dysrhythmias
CAUSE: brady = peritoneal stretching → increased vagal
tachy: from hypercapnia
Tx: supportive

hypoxemia
CAUSE: low FiO₂, hypoventilation, endobronchial intubation, atelectasis
Tx: fix the problem, ARMS, listen for breath sounds

hypercarbia
increase CO2 absorption, capnomediastinum, CO2 embolism

22
Q

how does carbon dioxide absorption happen?

A

inadvertent placement of veres needle or passage of CO₂ into open vessels

signs: arrhythmias (widening of QRS), hypoxemia, hypotension

diagnosis: TEE is gold standard

treatment: deflate abdomen, hyperventilate, left lateral decub/trend → this allows the embolus to get to the top of the RV and not into the pulmonary vasculature

23
Q

other complications

A

hypothermia: no difference from open; heat loss due to convection

positioning problems: usually d/t steep trend and pneumo → POVL, facial edema, dont give too many fluids. brachial plexus injury

hemorrhage: trocar into major vessle, damage to cystic or hepatic artery, promt treatment is key!! can take patient off vent when trocar is going in. bleeding can be hard to see b/c of positioning

24
Q

subcutaneous emphysema

A

cause: inadvertent extraperitoneal insufflation

predictors: operative time > 200 mins, 6 or more surgical ports

signs: crepitus, increased ETCO₂ hypersomnolence, increased SNS, resp acidosis