9.1 Shoulder Conditions Flashcards

1
Q

What is the definition of impingement syndrome?

A

Impingement syndrome is a general term describing shoulder pain, typically diagnosed following a positive mid range painful arc as well as various other impingement special tests. Typically referring to pinching on structures in the subacromial space during humeral elevation.

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2
Q

What structures in the subacromial space can get pinched with impingement syndrome?

A
  • Supraspinatus tendon
  • Subacromial bursa
  • Biceps long head tendon
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3
Q

At what range in the painful arc with their GH doing joint pain? Acromioclavicular joint pain?

A
  • GH joint: in 45° to 120°
  • AC joint: in 170° to 180°
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4
Q

What are some intrinsic factors that cause primary impingement at the subacromial/suprahumeral space?

A
  • Vascular changes in rotator cuff tendons (due to secondary disease process, age)
  • Structural variation in the acromion (flat vs hooked)
  • Trophic changes to the humeral head, AC joint, coracoacromial arch (fibrosis and thickening of coracoacromial ligament)
  • Post-traumatic/postsurgical scarring
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5
Q

What are some extrinsic factors resulting in decreased suprahumeral space and repetitive trauma to soft tissue during humeral elevation?

A
  • Posterior capsule tightness causes anterosuperior translation of humeral head
  • Weak external rotator/short internal rotators: restricts ability for humerus to externally rotate during abduction and restricts ability for greater tubercle to clear acromion (anterior cuff dominates posterior cuff)
  • Deltoid overpowers rotator cuff: net superior translation of humeral head during abduction
  • Poor scapular stability and muscular imbalances: if scapula is not stable, it doesn’t allow for optimal length-tension relationship of rotator cuff muscles
  • Hyperkyphosis: places scapula into position rotation, protraction and anterior tipping. Also places humerus into a position of internal rotation
  • Rotator cuff (and biceps LH) overuse and fatigue: if these muscles are fatigued from overuse, they no longer provide the dynamic stabilizing and compressive forces required for normal joint mechanics
  • Tendonopathy: inflammation (edema) decreases subacromial space with tendinitis, thickening/fibrosis decreases subacromial space with tendinosis
    • Supraspinatus: pain on palpation of tendon inferior to anterior of chromium, painful GH arc, positive impingement test
    • Infraspinatus: pain on palpation of tendon inferior to posterior corner externally of acromion when patient externally rotates and horizontally adducts, painful GH arc with abduction and flexion
    • Biceps long head: pain on palpation of tendon in bicipital groove, positive speeds test
  • Chronic inflammation and tendon overload can also stimulate the formation of osteophytes in the joint, which can further damage the surrounding musculature
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6
Q

What is difficult to differentiate from supraspinatus tendinitis because of the anatomical proximity?

A

Subacromial bursitis

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7
Q

Where might subacromial bursitis refer pain to?

A

To the deltoid insertion

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8
Q

How might subacromial bursitis present?

A
  • Acute burning or sharp pain
  • Painful “catch” when lowering arm from full abduction
  • Pain on release of speeds test (Isometric shoulder flexion)
  • May refer pain to deltoid insertion
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9
Q

In the cases of rotator cuff tears, either partial or full thickness, what should be emphasized during treatment?

A

Treatment needs to emphasize optimal joint/muscular mechanics. This includes normalizing posture, muscle imbalances (Length, strength, endurance) and tissue health/extensibility.

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10
Q

What is secondary impingement?

A

Due to instability (multi or unidirectional) impingement is the secondary result

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11
Q

What are some causes of secondary impingement?

A
  • Lax connective tissue, greater translation of humeral head in one or multiple directions
  • Labral or rotator cuff tears
  • Dislocation
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12
Q

What’s the aka for primary frozen shoulder?

A

Adhesive capsulitis

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13
Q

What is the capsular pattern for shoulder?

A

External rotation, abduction, internal rotation, flexion

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14
Q

Where are some causes of secondary frozen shoulder?

A
  • Another issue presents in which there is a period of pain and/or restricted motion that results in chronic inflammation and capsular adhesion
  • OA/RA
  • Immobilization (fracture, dislocation, surgery)
  • Trauma
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15
Q

What’s the onset, age of onset, and cause of primary frozen shoulder?

A
  • insidious onset
  • usually between ages 40-60
  • more prevalent in women
  • unknown cause
  • some studies have linked chronic inflammation in musculotendinous or synovial tissue (rotator cuff, biceps tendon, joint capsule) to the development of primary frozen shoulder
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16
Q

What happens in stage one primary frozen shoulder?

A
  • Gradual onset of pain that is aggravated by movement present at night
  • Loss of external rotation motion with intact rotator cuff strength is common (capsular restriction)
  • Duration is usually less than three months
17
Q

What happens in stage two “freezing” primary frozen shoulder?

A
  • Persistent and intense pain even at rest
  • GH motion is limited in all directions and cannot be fully restored within an intra-articular injection (anti-inflammatory)
  • Duration is usually 3 to 9 months
18
Q

What happens in stage three “frozen” primary frozen shoulder?

A
  • Pain only with movement
  • Significant adhesions, limited GH motions with substitute motions in the scapula
  • Atrophy of the deltoid, rotator cuff, biceps, triceps may be noted
  • Duration is usually 9 to 15 months
19
Q

What happens in stage four “thawing” primary frozen shoulder?

A
  • Minimal pain
  • No synovitis
  • Significant capsular restrictions from adhesions
  • Motion may gradually improve during this stage, though some patients never regain normal ROM
  • Duration is usually 15 to 24 months
20
Q

Which stages of frozen shoulder are treated as acute, subacute and chronic?

A
  • Stages 1 and 2: tx as acute
  • Stage 3: subacute
  • Stage 4: chronic
21
Q

What’s a bankart lesion?

A

Detachment of the interior capsule and glenoid labrum. May also include the biceps long head tendon.

22
Q

What’s a SLAP lesion?

A

Tear of the superior labor, anterior to posterior. May also include the biceps long head tendon.

  • This may occur through traumatic mechanism (fall on an outstretched arm or bracing steering wheel during MVA)
  • Or through repetitive overhead motions (Baseball throw, volleyball spike). Biceps LH tendon attached to labrum superiorly. Studies suggest that loaded maximum external rotation and abduction creates a ‘peel back’ mechanism –> a twist through the biceps LH tendon that pulls on the superior labrum
23
Q

What’s a reverse bankart lesion?

A

tearing of the posteroinferior labrum and often the posterior band of the inferior GH ligament

  • Typically occurs with posterior dislocation
  • Can also have a combination of the three lesions, which is called a 270° lesion
24
Q

What’s a Hill-Sachs lesion?

A
  • Compression fracture of the posterior aspect of the humeral head
  • Occurs during anterior dislocation as the posterior humeral head contacts the anterior rim of the glenoid
  • Usually accompanies the Bankart labrum lesion
  • May or or may not be repaired surgically depending on the size
  • IndiCongratulate shoulder instability

* can also have a reverse Hill-Sachs lesion during a posterior dislocation that typically accompanies a Reverse Bankart labrum lesion

25
Q

90% of dislocations happen in this direction (any why)

A
  • anterior
  • This is because of scapular position (30° anterior to the frontal plane) and the relative anterior resting position of the humeral head
26
Q

What’s the rehabilitation schedule after a dislocation?

A
  • AAROM (3 wks post), limiting external rotation
  • pendulum exercise, scapular retractions (4-6 wks post)
  • AROM limiting external rotation, isometric rotator cuff and scapular retraining (7-8 wks post)
  • AROM with end range stretch, isotonics (9-12 wks post)
  • Non-contact sports with no overhead activity (3 mos post)
  • contact sports with overhead activity (4 mos post)
27
Q

What’s a shoulder separation?

A
  • injury involving the AC joint
  • ranges from a sprain of AC lig to a full separation of clavicle from acromion
28
Q

What are the grades of shoulder separation?

A
  • gr 1: sprain to AC lig, no separation
  • gr 2: rupture of AC lig, sprain to coracoclavicular lig, slight displacement of clavicle
  • gr 3: rupture of AC and CC ligs, full separation of clavicle from acromion
29
Q

What are degrees of SC joint injury?

A

(uncommon, SC joint is very stable with strong ligamentous support; typically clavicle will fracture prior to SC dislocation)

  • 1st degree: sprain of SC lig
  • 2nd degree: complete tear of SC lig, partial tear (at most) of CC lig, clavicle subluxes from manubrium
  • 3rd degree: complete rupture of SC, CC and dislocation of clavicle from manubrium
30
Q

What are treatment options for shoulder separation?

A
  • NSAIDs
  • ice
  • immobilization 1-3 wks
  • taping for stability
  • therapeutic exercise
  • possible surgery for gr 3 separation
31
Q

What are treatment options for SC joint injury?

A
  • NSAIDs
  • Ice
  • Immobilization
  • Possible stabilization surgery (uncommon). Typically a tendon graft to stabilize joint, usually with the palmaris longus tendon.