9. ECG Common Abnormalilites Flashcards

1
Q

What 2 things can abnormal rhythms be due to?

A

Abnormal pulse formation

Abnormal conduction

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2
Q

Where can supraventricular rhythms arise from?

A

Sinus node
Atrium
AV node

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3
Q

Do you get normal or abnormal ventricular depolarisation with supraventricular rhythms? What can be seen on the QRS complexes?

A

Normal ventricular depolarisation

Normal QRS complexes

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4
Q

What happens to the length of depolarisation in ventricular rhythms (ventricular ectopic beats)? What happens to the QRS complexes on an ECG?

A

Depolarisation takes longer, as not via usual His-purkinje system
Wide and bizarre QRS complexes

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5
Q

Give 2 examples of ventricular rhythms

A

Ventricular premature beats
Ventricular fibrillation
(Ventricular tachycardia)

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6
Q

What changes on an ECG depending on where the origin of the impulse is, which allows the diagnosis of an arrhythmia?

A

P wave and QRS complex

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7
Q

What is the rhythm strip on an ECG?

A

Lead II (sometimes also V1 and V5) best for looking at P waves

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8
Q

What is atrial fibrillation?

A

Multiple atrial foci making impulses chaotic

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9
Q

What is seen on an ECG in atrial fibrillation?

A

No P waves - wavy baseline

Narrow QRS complexes, irregularly irregular

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10
Q

What is heart block (AV conduction blocks)?

A

Delay/failure of conduction of impulses from atrium to ventricles via AV node and bundle of His

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11
Q

What are 2 causes of heart block?

A

Acute myocardial infarction (most common)

Degenerative changes

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12
Q

What happens in first degree heart block?

A

Atrioventricular conduction lengthened due to slow conduction in AV node and bundle of His

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13
Q

What is seen on an ECG in first degree heart block?

A

Normal P wave
PR interval prolonged (>5 small squares)
Normal QRS

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14
Q

What is seen on an ECG in Mobitz type 1 (Wenkebach) 2nd degree heart block?

A

Progressive lengthening of PR interval until one P is nor conducted, then cycle begins again

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15
Q

What is seen on an ECG in Mobitz type 2 second degree heart block?

A

PR interval normal

Sudden dropped QRS

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16
Q

Which type of second degree heart block has a high risk of progression to complete heart block?

A

Mobitz type 2

17
Q

What happens in third degree heart block?

A
Atrial depolarisation normal
Impulses not conducted to ventricles
Ventricular pacemaker takes over
HR too slow to maintain BP and perfusion
Urgent pacemaker insertion required
18
Q

What is seen on an ECG in third degree heart block?

A

No relationship between P waves and QRS complexes
More P waves than QRS complexes
Wide QRS
HR slow 30-40bpm

19
Q

What is ventricular tachycardia?

A

Run of 3 or more consecutive ventricular ectopics. Is a broad complex tachycardia. If persists then is a dangerous rhythm, as high risk of VF need to shock to reset.

20
Q

What is ventricular fibrillation?

A

Abnormal ventricular depolarisation
Impulses from numerous ectopic sites in ventricular muscle
No coordinated contraction, ventricles quiver
No cardiac output - cardiac arrest

21
Q

What is seen on an ECG in VF?

A

Very rapid, irregular reading

22
Q

What changes are seen on an ECG in ischaemia and MI?

A

Need to look at PQRST in all 12 leads, changes seen in leads facing affected area
Leads facing affected area show ST segment depression, T wave inversion

23
Q

What are of the heart is most vulnerable in ischaemia and MI? Why?

A

Sub endocardial, as furthest from major coronary arteries

24
Q

When are ischaemic ECG usually seen?

A

During exercise (as flow through atherosclerosed coronary artery is usually during diastole, but diastole is shorter during exercise)

25
What is am ST segment elevation myocardial infarction (STEMI)?
MI due to complete occlusion of lumen of coronary artery by thrombus. Muscle injury extends full thickness from endocardium to epicardium.
26
What is seen on an ECG with an acute STEMI?
ST segment elevation in leads facing area affected
27
What develops on an ECG following a STEMI that indicated necrosis?
Deep Q waves (more than 1 small square wide and 2 deep), ST segment elevation may still be present, or returned to normal
28
What happens to the resting membrane potential in hyperkalaemia? What does this do to channel proteins and the heart?
``` Less negative (more depolarised) Inactivates dome voltage gated Na+ channels Heart becomes less excitable leading to conduction problems ```
29
What happens to the resting membrane potential in hypokalaemia? What affect does this have on the action potential?
``` More negative (hyper polarised) Longer ventricular action potential, increasing likelihood of early after depolarisations, leading to arrhythmia ```
30
What is seen on an ECG in hyperkalaemia?
High T wave | Progressing to VF
31
What is seen on an ECG in hypokalaemia?
Low T wave | Development of high U wave (wave that follows T wave)