9 - CHF Flashcards

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1
Q

Acute heart failure can manifest in 2 ways:

A

1) Acute pulmonary edema

2) Acute MI–>cardiogenic shock

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2
Q

Increased pressure inside heart chambers cause contractility to increase or decrease? What is this law called?

A

Increase, Frank-Starling

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3
Q

Causes of HF:

A

1) MI
2) htn (this is only rapid in pre-eclampsia)
3) dysrhythmia
4) “itis” (pericard, myocard, endocard)
5) dissection
6) cardiac tamp
7) valve disorder
8) PE
9) cardiomyopathy
10) high output heart failure

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4
Q

What are some examples of high output heart failure?

A

1) anemia
2) Beriberi
3) Paget’s
4) thyrotoxicosis
5) AV fistula

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5
Q

In systolic heart failure, EF will be < _____%

A

40

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6
Q

What will the body do to compensate for low CO?

A

Increase Renin and Angiotensin to increase fluid volume

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7
Q

The heart has an inability to relax. This is called ______heart failure

A

diastolic

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8
Q

What are some causes of diastolic HF?

A

LVH, htn, CAD

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9
Q

Would you want to diurese pts with diastolic HF?

A

NO! The problem is with back up in preload, you need to increase fluid volume and give vasodilators

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10
Q

Patient has SOB, cough, weakness, orthopnea, PND, fatigue, JVD. Do you suspect right or left sided failure?

A

left (mixed b/c it’s also causing right heart failure)

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11
Q

What are the right sided heart failure symptoms

A

peripheral edema
RUQ pain
JVD
hepatojugular reflex

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12
Q

Most common cause of right heart failure

A

left HF

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13
Q

Labs to dx HF in ED:

A

LFT (end organ damage)
BNP
cardiac enzymes

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14
Q

How do you treat hypotensive HR?

A

Inotropes, fluids. worst prognosis

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15
Q

heart sound specific to CHF

A

S3

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16
Q

why can you expect liver tenderness/pain/megaly?

A

backflow and engorgement into liver from backup in vena cava

17
Q

what’s are some fun findings on CXR is common with CHF?

A

Kerley B lines and cephalization (vertical lines), big heart, pleural effusion, big vena cava

18
Q

What rule of thumb are you using for ventilation of CHF patients?

A

Mild-Mod: Conscious and cooperative–non-invasive

Mod-Severe: AMS or unstable–intubation

19
Q

Reduce preload with

A

nitrates and diuretics

20
Q

Reduce afterload with

A

ntg

ACE inhibitors

21
Q

Inoptropes

A

dobutamine, dopamine

22
Q

pain control

A

morphine which will also vasodilate

23
Q

dilation of arterial wall

A

aneurysm

24
Q

AAA that is concerning will be > ___cm

A

3, monitor

25
Q

AAA that should be considered for repair is > ____cm

A

5 (also repair if pt is symptomatic)

26
Q

What is the red flag complaints for AAA?

A

back pain + abd pain

27
Q

What is the classic triad of AAA?

A

1) back pain + abd pain
2) pulsatile mass
3) hypotension

28
Q

Cullen’s sign is __________ and Grey-Turner sign is ________. Both are extremely alarming for what?

A

umbilical, flank, AAA rupture

29
Q

Dx AAA

A

1) CXR is 65% sensitive, but NOT specific (can’t r/o)
2) US is 90% sensitive but can’t r/o
3) CT w/ is gold standard***

30
Q

Tx for AAA?

A

1) rupture suspected? emergent repair
2) >5cm? elective repair
3) 3-5cm? close monitoring

31
Q

what lethal problem mimics many other “chest pain” complaints?

A

dissection

32
Q

what are the 3 layers of the aortic wall?

A

inside vessel out: intima, media, adventitia

33
Q

Dissection dissects through what layers?

A

intima and media

34
Q

most dissections are ascending, descending, or localized to the arch?

A

ascending 60%
descending 30%
arch 10%

35
Q

Patient c/o back pain, diaphoresis, n/v, syncope, what is leading DDx

A

dissection, must rule this out first!!

36
Q

Common exam findings in dissection

A

MI (dissects at coronary vessels), syncope, neuro deficit

37
Q

EKG findings in dissection

A

normal, or may show inferior MI

38
Q

Dx test of choice for dissection

A

CTA –will show aortic dilation, false lumen, etc

39
Q

Tx of dissection

A

1) pain control–Fentanyl (only enough to take edge off, you need to monitor)
2) decrease pressure to 100
3) minimize shear pressure (lower HR <60)***
So #1 is BB–Esmolol that primarily targets HR
4) sx repair