7B Electrical Activity in the Heart, Homeostasis and Sport Flashcards
respiratory minute ventilation / ventilation rate (per min)
tidal vol x breathing rate (breaths per min)
tidal volume
vol air in each breath (~0.4 dm³)
oxygen consumption
vol O₂ used by body (usually rate)
find change in vol of gas in spirometer (as O₂ used for respiration)
breathing rate
count no. peaks in spirometer trace per min
design an experiment to investigate ventilation in humans
use spirometer
-> gives reading of tidal volume
-> use to find breathing rate, O₂ consumption and resp min vol
- canister containing soda lime (absorbs CO₂ that subject exhales) inserted between mouthpiece and floating chamber
- after calibration, spirometer filled with O₂ with tube connected to O₂ chamber
- subject breathes into tube for 1 min -> as they breathe in = lid of chamber moves down and vice versa
- movements of lid recorded by pen attached to lid -> writes on rotating drum = makes SPIROMETER TRACE
- subject then exercises for 2 mins and spirometer chamber refilled with O₂ (used up in respiration -> vol gas in chamber ↓ over time)
- immediately after stopping exercise -> breathe into spirometer again for 1 min
- spirometer trace before and after exercise recorded
cardiac output
heart rate (bpm) x stroke vol (cm³)
cm³ min⁻¹
state diff in fast and slow twitch muscle fibres, including structural and physiological diff
- fast twitch contract quickly whereas slow contract slowly
- fast used for fast movement (mainly in eyes and legs) whereas slow used for slow movement (posture - in back)
- fast good for short bursts of speed/power (sprint) whereas slow good for endurance activities (long distance)
- fast get tired quickly whereas slow is opp
- fast energy released quickly through anaerobic resp using glycogen (↓ mitochondria) whereas slow energy released slowly through aerobic resp (↑ mitochondria) and blood vessels supply O₂
- fast are white (not much myoglobin -> so can’t store much O₂) whereas slow are pink (rich in myoglobin -> red coloured protein that supplies O₂)
examples of performance enhancing drugs and their diff effects on the body
- anabolic steroids -> ↑ strength, speed + stamina by ↑ muscle size + allowing athlete to train harder . ↑ aggression
- stimulants -> speed up reactions, ↓ fatigue + ↑ aggression
- narcotic analgesics -> ↓ pain so injuries don’t affect performance
discuss whether use of performance enhancing drugs by athletes is acceptable
absolutist:
- some drugs are illegal
- competition becomes unfair -> ppl get adv by taking drugs nit working hard
- serious health risks -> ↑ b.p, heart problems
- athletes not fully informed of health risks of drugs they take
rationalist:
- up to each individ. -> have right to make their own decision, whether its worth the risk etc
- drug-free sport isnt fair anyway -> diff athletes have diff equipment, training access -> use of drugs might overcome these inequalities
- athletes that want to compete at higher level may only be able to using drugs
adv of keyhole surgery
- doesnt involve opening patients up as much -> lose less blood + less scarring
- less pain caused after + recover faster as less damage done
- so easier for patient to return to normal activities + hospital stay shorter
describe how damaged cruciate ligaments can be fixed by keyhole surgery
(2 marks)
- crucviate ligament found in middle of knee, connects thigh bone to lower leg bone
- damaged cruciate ligament removed + replaced with graft
- graft usually from tendon in patients leg (or from donor)
prosthesis
artificial device to replace damaged or missing body parts
describe how prostheses enable ppl with damaged knee joints to participate in sport
- metal device inserted into knee to replace damaged cartilage + bone
- knee joint + ends of leg bones replaced to provide smooth knee joint
- cushioning in new joint helps ↓ impact on knee
- knee joint replacement allows ppl to participate in low impact sports
explain why it is important to have stable internal conditions in the human body
- intracellular enzymes: fragile
- pH affects molecular structure (H-bonds disrupted + charge distrib. on active site changes)
- temp affects proteins -> rate of reactions + stability of shape affected
- water potential -> could cause swelling / shrinking of cells
negative feedback
- mechanism that restores level of normal
- effectors respond to counteract change + bring level back to normal