5th year Flashcards

1
Q

are supernumerary teeth more prevalent in primary or permanent?

A

permanent (1.5-3.5%)

primary (0.2-08%)

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2
Q

commonest region for supernumerary teeth?

A

premaxilla

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3
Q

4 associated syndromes/conditions with supernumerary teeth

A

cleidocranial dysplasia
palatals clefts
Gardners syndrome
oro-facial digital syndrome

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4
Q

how are supernumerary teeth classified?

A

shape - conical, tuberculate, supplemental, odontome-like

position - mediodens, distomolar, paramolar

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5
Q

if supernumerary tooth is unerupted and not causing issues, what must you monitor for?

A
follicular expansion
cyst formation
resorption of adjacent roots
worsening ectopic position
ectopic eruption
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6
Q

when to remove supernumerary teeth?

A

if causing pathology/resorption/cystic/ectopic

if causing crowding

if causing permanent tooth to fail to erupt (extract + maintain space)

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7
Q

when should permanent tooth erupt after extraction of supernumerary?

A

18months-2yrs

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8
Q

prevalence of hypotonia

A

permanent 3.6-6.5%

primary 0.1%

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9
Q

are hyper/hypodontia more prevalent in male of female

A

Female

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10
Q

order for hypotonia

A

8 - L5 - U2 - U5

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11
Q

levels of hypodontia

A

mild - 1-2
moderate - 3-5
severe - 6 or more

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12
Q

genes involved in hypodontia

A

PAX9 MSX1

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13
Q

syndromes associated with hypodontia +/- microdontia

A
downs
CLP
ectodermal dysplasia
Ehlers danlos
epidermolysis bullosa
chondro-ectodermal dysplasia
incontinetia pigmentii
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14
Q

symptoms of ectodermal dysplasia

A
sparse hair
dry skin
may be anhidrotic
xerstomia
dry lips
speech/learning difficulties
chronic rhinitis/pharyngitis/URTIs
HYPODONTIA
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15
Q

difference between fusion and germination

A

fusion = 1 crown 2 roots

germination - 1 root 2 crowns

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16
Q

prevalence of microdontia

A

permanent - 2.5%

primary - 0.2-0.5%

17
Q

complication of missing upper 2

A

can lead to ectopic 3 as not guided into place

18
Q

complication of dens in dente and treatment to prevent

A

early pulp death

fissure seal asap

19
Q

complication of taurodontism

A

none except difficulties with endo

20
Q

what is odontodysplasia

A

rare ‘ghost teeth’
structurally abnormal teeth - if erupt should be removed, if unerupted can be monitored

if affects area = regional odontodysplasia

21
Q

causes of enamel defects

A

genetic - Amelogenesis imperfecta
systemic - MIH, fluorosis, nutrition, metabolic upset, infection
local - trauma, infection

22
Q

how to test difference between fluorosis and hypomatured AI

A

fluorosis responds to micro-abrasion

23
Q

treatment of AI in young patient

A

PFMC posterily

comp restorations anteriorly

24
Q

aims for AI treatment

A

preserve remaining tooth tissue
maintain OVD
reduce sensitivity/function

25
Q

causes of dentine defects

A

genetic - dentinogensis imperfects, dentinal dysplasia, fibrous dysplasia of dentine

other conditions - Ehlers dans, rickets, hypophosphatasia

environmental - nutrition, drugs, trauma

26
Q

when restoring tooth with DI what may need to be placed first?

A

fissure sealant - more white to cover discolouration

27
Q

why may hypophosphatasia cause early loss of teeth?

A

hypoplastic cementum

28
Q

dental symptoms of cleidocranial dysplasia

A
cementum defect
supernumeraries
delayed exfoliation
delayed eruption
small sinuses
delayed closure of sutures
29
Q

causes of premature eruption

A

familial
precocious puberty
high birth weight baby
endocrine abnormalities

30
Q

prevalence of infraocclusion

A

1-9%

31
Q

grading of infraocclusion

A

mild - just below occlusal
mod - at contact area
severe - right below contact

32
Q

treatment options for infraocclusion

A

monitor
restore contact point
restore occlusion if static
extraction +/- ortho