5.5.9: Controlling heart rate Flashcards

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1
Q

What is the role of blood circulation?

A
  • Transport of oxygen and nutrients,such as glucose, fatty acids and amino acids to the tissues.
  • Removal of waste products, such as carbon dioxide from the tissues to prevent accumulation that may become toxic.
  • Transport of urea from the liver to the kidneys.
  • Distribute heat around the body or deliver it to the skin to be radiated away.
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2
Q

How do the requirements of the cells and tissues vary according to their level of activity?

A

-When you are being physically active, your muscle cells need more oxygen and glucose so that they can respire more, releasing more energy for contraction.

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3
Q

How does the function of the heart muscles affect its requirements?

A
  • Your heart muscle cells also need more oxygen and fatty acids.
  • All the muscles will also need to remove more carbon dioxide and heat.
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4
Q

How can heart function be modified?

A
  • By raising or lowering the heart rate (beats per min.)
  • Altering the force of the contractions of the ventricular walls.
  • Altering the stroke volume (volume of blood pumped per beat).
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5
Q

What is the hearts pacemaker?

A
  • The sinoatrial node (SAN)

- The SAN initiates waves of excitation that usually override the myogenic action of the cardiac muscle.

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6
Q

Describe the action of the SAN.

A
  • A region of tissue that can initiate an action potential…
  • which travels as a wave of excitation over the atrial walls, through the AVN…
  • and down the Purkyne fibres to the walls of the ventricles…
  • causing them to contract.
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7
Q

Which hormone does the heart respond directly to?

A
  • The hormone adrenaline in the blood.

- This increases heart rate.

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8
Q

At rest, what is heart rate controlled by and what is the typical frequency of excitation?

A
  • The SAN.
  • This has a set frequency, varying from person to person, at which it initiates waves of excitation.
  • The frequency of excitation is typically 60-80 per minute.
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9
Q

What is the frequency of the excitation waves create by the SAN altered by and how?

A
  • Output from the cardiovascular centre in the medulla oblongata.
  • Nerves from here supply the SAN,
  • These nerves are a part of the autonomic nervous system.
  • These nerves don’t initiate a contraction but they can affect the frequency.
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10
Q

How can output from the cardiovascular centre in the medulla oblongata increase the frequency of contractions in the heart?

A

-Action potentials sent down a sympathetic nerve (the accelerans nerve) cause the release of noradrenaline at the SAN. This increases heart rate.

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11
Q

How can output from the cardiovascular centre in the medulla oblongata decrease the frequency of contractions in the heart?

A

-Action potentials sent down the vagus nerve release acetylcholine, which reduces heart rate.

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12
Q

How can environmental factors affect heart rate?

A
  • Input from sensory receptors is fed into the cardiovascular centre in the medulla oblongata.
  • Some inputs increase heart rate, others decrease it.
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13
Q

How can stretch receptors in the muscles lead to an increase in heart rate?

A
  • Stretch receptors in the muscles detect movement of the limbs.
  • These send impulses to the cardiovascular centre, informing it that extra oxygen may soon be needed.
  • This leads to an increase in heart rate.
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14
Q

Why do athletes stretch to ‘warm up’?

A

-This boosts heart activity in preparation for the race, ensuring that the muscles will have a good supply of oxygen.

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15
Q

How does sensory output from chemoreceptors lead to an increase in heart rate?

A
  • Chemoreceptors in the carotid arteries, aorta and brain monitor the pH of blood.
  • During exercise, muscles produce more carbon dioxide.
  • Some CO2 reacts with water in the blood plasma forming carbonic acid.
  • This reduces the pH of blood.
  • Change in pH is detected by chemoreceptors.
  • This sends action potentials to the cardiovascular centre.
  • Heart rate increases.
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16
Q

Why does our heart rate decline again when we stop exercising?

A

-The concentration of CO2 in the blood falls. This reduces the activity of the accelerator pathway. therefore the heart rate declines.

17
Q

How do stretch receptors in the walls of the carotid sinus affect heart rate?

A
  • Stretch receptors in the walls of the carotid sinus monitor blood pressure.
  • The carotid sinus is a small swelling in the carotid artery.
  • An increase in blood pressure, perhaps during vigorous exercise, is detected by these stretch receptors.
  • If the pressure is too high, the stretch receptors send action potentials to the cardiovascular centre, leading to a reduction in heart rate.
18
Q

If the mechanism of controlling heart rate fails, an artificial pacemaker is fitted. How does it work?

A
  • A pacemaker delivers an electrical impulse to the heart muscle.
  • It is implanted under the skin and fat on the chest (or sometimes within the chest cavity itself),
  • May be connected to the SAN or directly to the ventricle muscle.