5.5.9 controlling heart rate Flashcards

1
Q

important roles of the circulatory system

A
  • transport oxygen & nutrients (eg. glucose, fatty acids, amino acids) to the tissues
  • removal of waste productions to prevent accumulation which may become toxic (eg. carbon dioxide)
  • transport urea from liver to kidneys
  • distribute heat around the body or deliver it to skin to be radiated away
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2
Q

how can the heart action be modified to allow the circulatory system to adapt to meet the tissues’ needs

A
  • raise or lower heart rate (number of beats per minute)
  • altering force of contractions of ventricular walls
  • altering stroke volume (volume of blood pumped per beat)
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3
Q

what is the cardiac muscle described as

A

myogenic

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4
Q

which muscle has a higher myogenic rate

A

atrial muscle has a higher myogenic rate than the ventricular muscle

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5
Q

pacemaker of the heart

A

sinoatrial node (SAN)

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6
Q

describe the sinoatrial node (SAN)

A
  • initiates waves of excitation which usually override myogenic action of cardiac muscle
  • region of tissue that can initiate an action potential
  • action potential travels as a wave of excitation over the atria walls, through the AVN (atrio-ventricular node) & down the Purkyne fibres to walls of ventricles = causes them to contract
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7
Q

what hormone does the heart muscle respond directly to

A

adrenaline in the blood = increases heart rate

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8
Q

what is heart rate controlled by at rest

A

= SAN

  • set frequency at which it initiates waves of excitation
  • frequency of excitation usually 60-80 per minute
  • however, frequency of excitation waves altered by output from cardiovascular centre in medulla oblongata
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9
Q

cardiovascular centre & SAN

A
  • nerves from cardiovascular centre in medulla oblongata of brain supply SAN
  • nerves are part of autonomic nervous system
  • nerves don’t initiate a contraction, but can affect frequency of contractions
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10
Q

how can the nerves from the cardiovascular centre affect frequency of contractions

A
  • action potentials sent down sympathetic nerve (accelerans nerve) cause release of noradrenaline at SAN = increases heart rate
  • action potentials sent down vagus nerve release acetylcholine = reduces heart rate
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11
Q

outline sensory input to cardiovascular centre

A
  • stretch receptors in muscles detect limb movement = send impulses to cardiovascular centre, informing it extra oxygen may be needed soon (increases heart rate)
  • chemoreceptors in carotid arteries, aorta & brain monitor pH of blood (when exercising, blood pH reduced as carbonic acid formed which will affect oxygen transport) = change in pH detected, sending action potentials to cardiovascular centre (increases heart rate)
  • concentration of carbon dioxide in blood (when stop exercising, concentration of carbon dioxide in blood falls) = reduces activity of accelerator pathway (heart rate decreases)
  • stretch receptors in walls of carotid sinus monitor blood pressure = if pressure rises too high, stretch receptors send action potentials to cardiovascular centre (heart rate decreases)
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12
Q

what happens if the mechanism controlling heart rate fails

A

= artificial pacemaker fitted

  • delivers electrical impulse to heart muscle
  • implanted under skin/fat on chest (or sometimes within chest cavity)
  • may be connected to SAN or directly to ventricle muscle
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