5.5 plant and animal responses Flashcards

1
Q

difference between abiotic and biotic

A

abiotic = non living
biotic = living

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2
Q

name the chemical defences of plants when under threat of herbivores

A

tannins
alkaloids
pheromones

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3
Q

what do tannins do

A

toxic to microorganisms and larger herbivores
make leaves taste bad
prevent infiltration by pathogenic microorganims in roots

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4
Q

what do alkaloids do

A

taste bitter
in tips and flowers and peripheral cells in stems, roots

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5
Q

what do pheromones do

A

chemicals released that can affect anothers behaviour/physiology

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6
Q

define tropism

A

a directional growth response determined by an external stimulus

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7
Q

name the different types of tropisms as a response to abiotic stress

A

photo - light
geo - gravity
chemo - chemicals
thigmo - physical contact eg. ivy growing around structures
hydro - water

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8
Q

define nastic response

A

a non directional response to external stimuli

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9
Q

example of nastic response

A

folding in when touched

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10
Q

name the plant hormones

A

cytokinins
abscisic acid
auxins
gibberellins
ethene

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11
Q

effects of cytokinins

A

promote cell division
delay leaf senescence
overcome apical dominance
promote cell expansion

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12
Q

effects of abscisic acid

A

inhibits seed germination/ growth
causes stomatal closure when low water availablity

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13
Q

effects of auxin

A

promote cell elongation
inhibit growth of side shoots
inhibit leaf abscission (leaf fall)

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14
Q

where is auxin made

A

apex of the shoot

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15
Q

effects of gibberellins

A

promote seed germination
stem growth

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16
Q

effects of ethene

A

promotes fruit ripening

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17
Q

general role of plant hormones

A

coordinate plant responses to environmental stimuli

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18
Q

how do hormones move in a plant

A

active transport
diffusion
mass flow

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19
Q

leaf senescence vs leaf absission

A

senescence = development of leaves
absission = leaf fall

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20
Q

define apical dominance

A

inhibition of lateral buds further down the shoot due to chemical produced by the apical bud at the tip of the shoot

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21
Q

explain the relationship between auxin concentration and growth

A

normal auxin conc. in lateral buds inhibits growth
where as low auxin conc. promotes growth

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22
Q

how are auxins, abscisic acid and cytokinins involved in plant growth

A

abscisic acid = inhibits bud growth - high auxin levels in shoot keeps the AA levels high = when the tip is removed AA levels drop and the bud starts to grow
cytokinins = promotes bud growth = high auxin levels = sink for cyto. = apical dominance = when tip removed cytokinin spreads evenly around plant = lateral growth

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23
Q

role of gibberellins in seed germination

A

seed absorbes water = releases gibberellins = stimulates amylase production = breaks starch into glucose = substrate for respiration/ protein synthesis = embryo can grow

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24
Q

how do gibberellins cause growth

A

causes growth in internodes by stimulating cell elongation (loosening of cell walls) and cell division (stimulates protein production that controls cell cycle)

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25
explain the experiment about gibberellins in dwarf and tall plants
tall plants = high concentrations of Ga1 (gibberellins) tall plants = have homo. dominant Le allele dwarf = have homo. recessive le allele Le is responsible for converting G20 to GA1 (gibberellins) plant with mutant (na-1) blocks gibberellin production in first step grafted this shoot onto homozygous le plant = grew tall as shows it has the enzyme toconvert G20 to GA1 and can use the G20 provided from the other plant therefore proves GA1 causes stem elongation/ growth
26
where does the growth occur in plants
meristem in roots/ shoots
27
explain Darwins results in investigating plant hormones
the shoot tip is responsible for phototropic responses
28
explain Boysen-Jensons results in investigating plant hormones
substance in the tip needs to be able to move backwards for phototropism to happen - permeable block = shoot still showed phototropism - impermeable block = no phototropic response
29
explain Went's results in investigating plant hormones
chemical messenger doesn't need tip to be present to allow phototropism
30
how was auxin proved to be the chemical messenger for growth in plants in investigating plant hormones
agar blocks with with different auxin concentrations on gave the same results using a series of blocks with diff. conc. of auxin (IAA) created by serial dilution gave shoot curvature in proportion to the amount of auxin equal light = equal growth all around unequal light = auxins move to shade = cells elongate quicker = bends to light extent of cell elongation = con. of auxin
31
mechanism of auxin
causes active tranpsort of H+ by ATPase enzymes on plasma membrane into cell wall results in low pH provides optimum pH for enzymes to break bonds within the cellulose wall becomes less rigid and can expand as cell takes in water
32
explain the effect of auxin in the roots and shoots
shoots - light on one side = auxin moves to shaded side = zone of elongation = cells elongate roots - auxin accumulates on lower side and inhibits cell elongation = upper side continues to grow = root bends down
33
explain the effect of auxin concentration on stimulation and inhibition of growth in the roots and shoots
at low concentrations = root growth is stimulated at high concentrations = shoot growth stimulated and root growth inhibited
34
the commercial use of auxin
prevent leaf and fruit drop promote flowering herbicides - promotes growth too much that cell cannot support= breaks = plant dies seedless fruit encouraging root growth/ taking cuttings
35
the commercial use of cytokinins
delay leaf senescence / prevent yellowing of leaves mass produce plants in tissue culture promote bud and shoot growth
36
the commercial use of gibberellins
fruit production -delay senescence - makes fruits available for longer -can elongate apples to improve shape -grape stalks elongate = less compact so bigger grapes brewing - speeds up germination of barely sugar production - stimulates growth in nodes = stems elongate = more sugar can be stored plant breeding - speed up process by inducing seed formation
37
commercial use of ethene
speeds up fruit ripening promotes fruit drop promotes lateral growth promotes female sex expression in cucumbers
38
central nervous system
central part of the nervous system composed of the brain, spinal cord and relay neurones
39
peripheral nervous system
the sensory and motor neurones connecting the sensory receptors and effectors to the cns
40
what does the PNS divide into
somatic and autonomic
41
describe the somatic nervous system
conduct action potentials from the cns to the effectors under voluntary control mostly myelinated = responses are rapid one single neurone connecting
42
describe the autonomic nervous system
conduct action potentials from the cns to the effectors under involuntary control mostly non myelinated = responses are not rapid two neurones are connected by a ganglia
43
what can the autonomic nervous system be divided into
sympathetic and parasympathetic
44
describe the sympathetic nervous system
prepares the body for activity fight or flight response noradrenaline as neurotransmitter increases activity during times of stress
45
describe the parasympathetic nervous system
conserves energy relaxing responses acetylcholine as neurotransmitter decreases activity during sleep/ relaxation
46
effects of the sympathetic and parasympathetic nervous systems (opposite)
sympathetic - increase heart rate - dilates pupils - increase ventilation rate - reduces digestive activity parasympathetic - decrease heart rate - constricts pupils - reduce ventilation rate - increase digestive activity
47
Name the structures of the brain
Cerebrum Cerebellum Medulla oblongata Hypothalamus Pituitary
48
Function of the cerebrum
Conscious thought Conscious actions Emotional responses Intelligence, decision making etc. Memory Divided into two cerebral hemispheres (left side controls right side of body)
49
Function of the cerebellum
Coordinates balance and muscle control/ coordination
50
Function of the medulla oblongata
Controls physiological processes Eg. Cardiac centre , Vasomotor centre, Respiratory centre
51
Function of the hypothalamus
Coordinates homeostasis responses Temperature regulation Osmroregulation
52
Functions of the pituitary
endocrine gland anterior lobe - secretes hormones posterior lobe - stores hormones
53
define reflex action
a response that doesn't involve any processing by the brain
54
what neurones make up the reflex pathway
sensory - relay - motor
55
explain the blinking reflex
temporary closure of the eye is a cranial reflex - passes through the brain
56
explain the knee jerk reflex
relex that straightens the leg is a spinal reflex - passes through spinal cord response is quick as only uses sensory and motor neurone
57
where is the fight or flight response coordinated
hypothalamus
58
how does the hypothalamus coordinate a response
increases activity in the sympathetic nervous system stimulates release of hormones from the anterior pituitary gland
59
what does the hypothalamus secrete and their role
releasing hormones into the blood passes into the pituitary which causes release of tropic hormones from anterior pituitary stimulates endocrine glands
60
what does CRH cause
from the hypothalamus causes the release of ACTH from the pituitary
61
what does TRH cause
from the hypothalamus causes release of TSH
62
what does ACTH cause
adrenal cortex to release cortisol hormones
63
what does TSH cause
thyroid stimulating hormone the thyroid to release more thyroxine
64
explain what happens in response coordination in the nervous pathway
sympathetic nervous system activates adrenal medulla - adrenaline released into the bloodstream impulses activate glands/ smooth muscles fight or flight response initiated
65
explain what happens in hormonal coordination in the nervous pathway
hypothalamus releases hormones to stimulate the pituitary gland pituitary secretes ACTH - adrenal cortex releases corticoid hormones - into the bloodstream pituitary secretes TSH - thyroid gland secretes thyroxine - into the bloodstream initiates fight or flight response
66
mechanism of adrenaline
protein hormone = can't enter cell adrenaline (first messanger) binds to receptor = associated with a G protein inside cell = activates enzyme adenyl cyclase converts ATP to cAMP (second messanger) cyclic AMP causes activation of enzymes
67
how can the heart generate its own heartbeat
myogenic cardiac muscle - initiate its own beat SAN - pacemaker - controls wave of excitation/ action potential by nerves (accelerater, vagus)
68
describe the role of the accelerator and vagus nerve
accelerator = increases heart rate vagus = reduces heart rate
69
what is the cardiovascular centre located
medulla oblongata
70
how does the cardiovascular centre recieve information
stretch receptors = in muscles = detect movement = send impulses to the cardiovascular centre = extra 02 needed= hr increases chemoreceptors = in carotid arteries, aorta = monitor pH = exercise produces co2/ carbonic acid baroreceptors in carotid sinus = detect blood pressure
71
3 types of muscle
smooth (involuntary) skeletal (voluntary) cardiac
72
explain the structure of cardiac muscle and its benefits
cells form long fibres branch to form cross bridges ensure electrical stimulation spreads evenly over the walls cells joined by intercalated discs - allow movement of ions doesn't fatigue easily, myogenic
73
explain the structure of smooth muscle
spindle shaped cells contracts slowly and regularly doesn't tire easily controlled by autonomic nervous system in longitudinal and circular layers
74
explain the structure of skeletal muscle (sarco)
striated sacorlemma - membrane that surrounds the fibres that contain many nuclei sarcoplasm - muscle cell cytoplasm that contains lots of mitochondria and sarcoplasmic reticulum - specialised endoplasmic reticulum myofibrils - contents of the fibres are arranged in sarcomeres - myofibrils are divided into chain of subunits that contain protein filaments, are the contractile units of muscle
75
what proteins are in muscles to allow contraction
actin myosin
76
neuromusclar junction
junction where a neurone and muscle meet
77
how is a muscle contraction stimulated at a NMJ
action potential arrives at the end of the axon opens calcium ions channels = move into axon causes vesicles of acetylcholine to move to the membrane exocytosis = acetylcholine molecules diffuse across the gap = fuse with receptors on the sarcolemma opens sodium ion gates = enters muscle fibres = causes depolarisation wave of depolarisation passes along sarcolemma = spreads down tubule (T system) causes calcium ions to be released from sarcoplasmic reticulum = causes muscle contraction
78
name the 4 proteins found in muscles
actin myosin tropomyosin troponin
79
explain what the H zone, A band, I band and Z lines are
H zone = area in the centre where only myosin is, dark band A band = the whole length of myosin I band = area where there is only actin, light band Z lines = hold together actin
80
what are the thin filaments and thick filaments of a muscle
thin = actin thick = myosin
81
structure and function of the proteins of thin filaments
actin = two strands that wind around each other, twisted double string of beads tropomyosin winds around the actin = blocks binding sites at rest troponin = complex of 3 polypeptides - one binds to actin, one to tropomyosin and one to calcium
82
structure and function of thick filaments
bundle of myosin molecules has two potruding heads heads are mobile and bind to actin when sites are exposed
83
briefly explain the mechanism of muscle contraction
caused by movement of myosin heads muscle stimulated = tropomyosin moves exposing binding sites = myosin binds to sites on actin = causes actin to slide past myosin
84
control of muscle contraction
action potential passes to sarcoplasmic reticulum = calcium ions released into sarcoplasm calcium ions bind to the troponin = alters the shape and tropomyosin is pulled aside = exposes binding sites on the actin myosin heads bind to the actin = forms cross bridges between the filaments myosin heads move = pulls actin past past the myosin myosin heads detach and bind again further up the actin
85
how is a muscle relaxed
calcium ions pumped back into sarcoplasmic reticulum
86
role of ATP in muscle contraction
myosin head attaches to the actin filament = cross bridge myosin head tilts backwards = causing actin to move past = power stroke = ADP +Pi are released after the power stroke a new ATP joins = breaks cross bridge myosin head returns to original position as ATP is hydrolysed to ADP +Pi = as it releases energy myosin can now form new cross bridge
87
maintaing the supply of ATP
aerobic respiration = high oxygen supply = lots of ATP released = limited to how much 02 is supplied aerobic respiration = release less ATP but lactic acid builds up = fatigue creatine phosphate = reserve store = phosphate transferred to ADP molecules creating ATP = supports contraction for another 2-4 seconds