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module 5 Communication, homeostasis and energy > 5.5 plant and animal responses > Flashcards

5.5 plant and animal responses Flashcards

1
Q

difference between abiotic and biotic

A

abiotic = non living
biotic = living

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2
Q

name the chemical defences of plants when under threat of herbivores

A

tannins
alkaloids
pheromones

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3
Q

what do tannins do

A

toxic to microorganisms and larger herbivores
make leaves taste bad
prevent infiltration by pathogenic microorganims in roots

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4
Q

what do alkaloids do

A

taste bitter
in tips and flowers and peripheral cells in stems, roots

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5
Q

what do pheromones do

A

chemicals released that can affect anothers behaviour/physiology

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6
Q

define tropism

A

a directional growth response determined by an external stimulus

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7
Q

name the different types of tropisms as a response to abiotic stress

A

photo - light
geo - gravity
chemo - chemicals
thigmo - physical contact eg. ivy growing around structures
hydro - water

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8
Q

define nastic response

A

a non directional response to external stimuli

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9
Q

example of nastic response

A

folding in when touched

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10
Q

name the plant hormones

A

cytokinins
abscisic acid
auxins
gibberellins
ethene

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11
Q

effects of cytokinins

A

promote cell division
delay leaf senescence
overcome apical dominance
promote cell expansion

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12
Q

effects of abscisic acid

A

inhibits seed germination/ growth
causes stomatal closure when low water availablity

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13
Q

effects of auxin

A

promote cell elongation
inhibit growth of side shoots
inhibit leaf abscission (leaf fall)

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14
Q

where is auxin made

A

apex of the shoot

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15
Q

effects of gibberellins

A

promote seed germination
stem growth

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16
Q

effects of ethene

A

promotes fruit ripening

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17
Q

general role of plant hormones

A

coordinate plant responses to environmental stimuli

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18
Q

how do hormones move in a plant

A

active transport
diffusion
mass flow

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19
Q

leaf senescence vs leaf absission

A

senescence = development of leaves
absission = leaf fall

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20
Q

define apical dominance

A

inhibition of lateral buds further down the shoot due to chemical produced by the apical bud at the tip of the shoot

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21
Q

explain the relationship between auxin concentration and growth

A

normal auxin conc. in lateral buds inhibits growth
where as low auxin conc. promotes growth

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22
Q

how are auxins, abscisic acid and cytokinins involved in plant growth

A

abscisic acid = inhibits bud growth - high auxin levels in shoot keeps the AA levels high = when the tip is removed AA levels drop and the bud starts to grow
cytokinins = promotes bud growth = high auxin levels = sink for cyto. = apical dominance = when tip removed cytokinin spreads evenly around plant = lateral growth

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23
Q

role of gibberellins in seed germination

A

seed absorbes water = releases gibberellins = stimulates amylase production = breaks starch into glucose = substrate for respiration/ protein synthesis = embryo can grow

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24
Q

how do gibberellins cause growth

A

causes growth in internodes by stimulating cell elongation (loosening of cell walls) and cell division (stimulates protein production that controls cell cycle)

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25
Q

explain the experiment about gibberellins in dwarf and tall plants

A

tall plants = high concentrations of Ga1 (gibberellins)
tall plants = have homo. dominant Le allele
dwarf = have homo. recessive le allele
Le is responsible for converting G20 to GA1 (gibberellins)
plant with mutant (na-1) blocks gibberellin production in first step
grafted this shoot onto homozygous le plant = grew tall as shows it has the enzyme toconvert G20 to GA1 and can use the G20 provided from the other plant
therefore proves GA1 causes stem elongation/ growth

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26
Q

where does the growth occur in plants

A

meristem
in roots/ shoots

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27
Q

explain Darwins results in investigating plant hormones

A

the shoot tip is responsible for phototropic responses

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28
Q

explain Boysen-Jensons results in investigating plant hormones

A

substance in the tip needs to be able to move backwards for phototropism to happen
- permeable block = shoot still showed phototropism
- impermeable block = no phototropic response

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29
Q

explain Went’s results in investigating plant hormones

A

chemical messenger doesn’t need tip to be present to allow phototropism

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30
Q

how was auxin proved to be the chemical messenger for growth in plants in investigating plant hormones

A

agar blocks with with different auxin concentrations on gave the same results
using a series of blocks with diff. conc. of auxin (IAA) created by serial dilution gave shoot curvature in proportion to the amount of auxin
equal light = equal growth all around
unequal light = auxins move to shade = cells elongate quicker = bends to light
extent of cell elongation = con. of auxin

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31
Q

mechanism of auxin

A

causes active tranpsort of H+ by ATPase enzymes on plasma membrane into cell wall
results in low pH
provides optimum pH for enzymes to break bonds within the cellulose
wall becomes less rigid and can expand as cell takes in water

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32
Q

explain the effect of auxin in the roots and shoots

A

shoots - light on one side = auxin moves to shaded side = zone of elongation = cells elongate
roots - auxin accumulates on lower side and inhibits cell elongation = upper side continues to grow = root bends down

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33
Q

explain the effect of auxin concentration on stimulation and inhibition of growth in the roots and shoots

A

at low concentrations = root growth is stimulated
at high concentrations = shoot growth stimulated and root growth inhibited

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34
Q

the commercial use of auxin

A

prevent leaf and fruit drop
promote flowering
herbicides - promotes growth too much that cell cannot support= breaks = plant dies
seedless fruit
encouraging root growth/ taking cuttings

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35
Q

the commercial use of cytokinins

A

delay leaf senescence / prevent yellowing of leaves
mass produce plants in tissue culture
promote bud and shoot growth

36
Q

the commercial use of gibberellins

A

fruit production
-delay senescence - makes fruits available for longer
-can elongate apples to improve shape
-grape stalks elongate = less compact so bigger grapes
brewing
- speeds up germination of barely
sugar production
- stimulates growth in nodes = stems elongate = more sugar can be stored
plant breeding
- speed up process by inducing seed formation

37
Q

commercial use of ethene

A

speeds up fruit ripening
promotes fruit drop
promotes lateral growth
promotes female sex expression in cucumbers

38
Q

central nervous system

A

central part of the nervous system composed of the brain, spinal cord and relay neurones

39
Q

peripheral nervous system

A

the sensory and motor neurones connecting the sensory receptors and effectors to the cns

40
Q

what does the PNS divide into

A

somatic and autonomic

41
Q

describe the somatic nervous system

A

conduct action potentials from the cns to the effectors
under voluntary control
mostly myelinated = responses are rapid
one single neurone connecting

42
Q

describe the autonomic nervous system

A

conduct action potentials from the cns to the effectors
under involuntary control
mostly non myelinated = responses are not rapid
two neurones are connected by a ganglia

43
Q

what can the autonomic nervous system be divided into

A

sympathetic and parasympathetic

44
Q

describe the sympathetic nervous system

A

prepares the body for activity
fight or flight response
noradrenaline as neurotransmitter
increases activity
during times of stress

45
Q

describe the parasympathetic nervous system

A

conserves energy
relaxing responses
acetylcholine as neurotransmitter
decreases activity
during sleep/ relaxation

46
Q

effects of the sympathetic and parasympathetic nervous systems (opposite)

A

sympathetic
- increase heart rate
- dilates pupils
- increase ventilation rate
- reduces digestive activity
parasympathetic
- decrease heart rate
- constricts pupils
- reduce ventilation rate
- increase digestive activity

47
Q

Name the structures of the brain

A

Cerebrum
Cerebellum
Medulla oblongata
Hypothalamus
Pituitary

48
Q

Function of the cerebrum

A

Conscious thought
Conscious actions
Emotional responses
Intelligence, decision making etc.
Memory
Divided into two cerebral hemispheres (left side controls right side of body)

49
Q

Function of the cerebellum

A

Coordinates balance and muscle control/ coordination

50
Q

Function of the medulla oblongata

A

Controls physiological processes
Eg. Cardiac centre , Vasomotor centre, Respiratory centre

51
Q

Function of the hypothalamus

A

Coordinates homeostasis responses
Temperature regulation
Osmroregulation

52
Q

Functions of the pituitary

A

endocrine gland
anterior lobe - secretes hormones
posterior lobe - stores hormones

53
Q

define reflex action

A

a response that doesn’t involve any processing by the brain

54
Q

what neurones make up the reflex pathway

A

sensory - relay - motor

55
Q

explain the blinking reflex

A

temporary closure of the eye
is a cranial reflex - passes through the brain

56
Q

explain the knee jerk reflex

A

relex that straightens the leg
is a spinal reflex - passes through spinal cord
response is quick as only uses sensory and motor neurone

57
Q

where is the fight or flight response coordinated

A

hypothalamus

58
Q

how does the hypothalamus coordinate a response

A

increases activity in the sympathetic nervous system
stimulates release of hormones from the anterior pituitary gland

59
Q

what does the hypothalamus secrete and their role

A

releasing hormones into the blood
passes into the pituitary which causes release of tropic hormones from anterior pituitary
stimulates endocrine glands

60
Q

what does CRH cause

A

from the hypothalamus causes the release of ACTH from the pituitary

61
Q

what does TRH cause

A

from the hypothalamus causes release of TSH

62
Q

what does ACTH cause

A

adrenal cortex to release cortisol hormones

63
Q

what does TSH cause

A

thyroid stimulating hormone
the thyroid to release more thyroxine

64
Q

explain what happens in response coordination in the nervous pathway

A

sympathetic nervous system
activates adrenal medulla - adrenaline released into the bloodstream
impulses activate glands/ smooth muscles
fight or flight response initiated

65
Q

explain what happens in hormonal coordination in the nervous pathway

A

hypothalamus releases hormones to stimulate the pituitary gland
pituitary secretes ACTH - adrenal cortex releases corticoid hormones - into the bloodstream
pituitary secretes TSH - thyroid gland secretes thyroxine - into the bloodstream
initiates fight or flight response

66
Q

mechanism of adrenaline

A

protein hormone = can’t enter cell
adrenaline (first messanger)
binds to receptor = associated with a G protein inside cell = activates enzyme adenyl cyclase
converts ATP to cAMP
(second messanger)
cyclic AMP causes activation of enzymes

67
Q

how can the heart generate its own heartbeat

A

myogenic cardiac muscle - initiate its own beat
SAN - pacemaker - controls wave of excitation/ action potential by nerves (accelerater, vagus)

68
Q

describe the role of the accelerator and vagus nerve

A

accelerator = increases heart rate
vagus = reduces heart rate

69
Q

what is the cardiovascular centre located

A

medulla oblongata

70
Q

how does the cardiovascular centre recieve information

A

stretch receptors = in muscles = detect movement = send impulses to the cardiovascular centre = extra 02 needed= hr increases
chemoreceptors = in carotid arteries, aorta = monitor pH = exercise produces co2/ carbonic acid
baroreceptors in carotid sinus = detect blood pressure

71
Q

3 types of muscle

A

smooth (involuntary)
skeletal (voluntary)
cardiac

72
Q

explain the structure of cardiac muscle and its benefits

A

cells form long fibres
branch to form cross bridges
ensure electrical stimulation spreads evenly over the walls cells joined by intercalated discs - allow movement of ions
doesn’t fatigue easily, myogenic

73
Q

explain the structure of smooth muscle

A

spindle shaped cells
contracts slowly and regularly
doesn’t tire easily
controlled by autonomic nervous system
in longitudinal and circular layers

74
Q

explain the structure of skeletal muscle (sarco)

A

striated
sacorlemma - membrane that surrounds the fibres that contain many nuclei
sarcoplasm - muscle cell cytoplasm that contains lots of mitochondria and
sarcoplasmic reticulum - specialised endoplasmic reticulum
myofibrils - contents of the fibres are arranged in
sarcomeres - myofibrils are divided into chain of subunits that contain protein filaments, are the contractile units of muscle

75
Q

what proteins are in muscles to allow contraction

A

actin
myosin

76
Q

neuromusclar junction

A

junction where a neurone and muscle meet

77
Q

how is a muscle contraction stimulated at a NMJ

A

action potential arrives at the end of the axon
opens calcium ions channels = move into axon
causes vesicles of acetylcholine to move to the membrane
exocytosis = acetylcholine molecules diffuse across the gap = fuse with receptors on the sarcolemma
opens sodium ion gates = enters muscle fibres = causes depolarisation
wave of depolarisation passes along sarcolemma = spreads down tubule (T system)
causes calcium ions to be released from sarcoplasmic reticulum = causes muscle contraction

78
Q

name the 4 proteins found in muscles

A

actin
myosin
tropomyosin
troponin

79
Q

explain what the H zone, A band, I band and Z lines are

A

H zone = area in the centre where only myosin is, dark band
A band = the whole length of myosin
I band = area where there is only actin, light band
Z lines = hold together actin

80
Q

what are the thin filaments and thick filaments of a muscle

A

thin = actin
thick = myosin

81
Q

structure and function of the proteins of thin filaments

A

actin = two strands that wind around each other, twisted double string of beads
tropomyosin winds around the actin = blocks binding sites at rest
troponin = complex of 3 polypeptides - one binds to actin, one to tropomyosin and one to calcium

82
Q

structure and function of thick filaments

A

bundle of myosin molecules
has two potruding heads
heads are mobile and bind to actin when sites are exposed

83
Q

briefly explain the mechanism of muscle contraction

A

caused by movement of myosin heads
muscle stimulated = tropomyosin moves exposing binding sites = myosin binds to sites on actin = causes actin to slide past myosin

84
Q

control of muscle contraction

A

action potential passes to sarcoplasmic reticulum = calcium ions released into sarcoplasm
calcium ions bind to the troponin = alters the shape and tropomyosin is pulled aside = exposes binding sites on the actin
myosin heads bind to the actin = forms cross bridges between the filaments
myosin heads move = pulls actin past past the myosin
myosin heads detach and bind again further up the actin

85
Q

how is a muscle relaxed

A

calcium ions pumped back into sarcoplasmic reticulum

86
Q

role of ATP in muscle contraction

A

myosin head attaches to the actin filament = cross bridge
myosin head tilts backwards = causing actin to move past = power stroke = ADP +Pi are released
after the power stroke a new ATP joins = breaks cross bridge
myosin head returns to original position as ATP is hydrolysed to ADP +Pi = as it releases energy
myosin can now form new cross bridge

87
Q

maintaing the supply of ATP

A

aerobic respiration = high oxygen supply = lots of ATP released = limited to how much 02 is supplied
aerobic respiration = release less ATP but lactic acid builds up = fatigue
creatine phosphate = reserve store = phosphate transferred to ADP molecules creating ATP = supports contraction for another 2-4 seconds

module 5 Communication, homeostasis and energy (7 decks)

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