5.1.5 Plant and Animal Response Flashcards

1
Q

what are ways that plants can sense changes in environment, and adapt to them

A
  • can sense the direction of light (and grow towards in to maximise light absorption for photosynthesis)
  • can sense gravity, so their roots and shoots grow in the right direction
  • climbing plants can sense touch, so they can find things to climb and reach sunlight
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2
Q

what do plants need to respond to, in order to increase chances of survival

A
  • herbivory
  • abiotic stress
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3
Q

what is herbivory

A

being eaten by plants
- plants are more likely to survive if they can respond to this

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4
Q

what toxic chemicals can plants produce, in response to being eaten

A
  • ALKALOIDS: chemicals with bitter taste, noxious smells or poisonous characteristics
  • so able to deter or kill herbivores
  • e.g. tobaccos plants produce alkaloid nicotine in response to tissue damage, and nicotine is highly poisonous to many insects
  • TANNINS: taste bitter, and in some herbivores (e.g. cattle and sheep), can bind to proteins in the gut, making the plant hard to digest
  • both deter animals from eating the plant
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5
Q

what are pheromones

A

signalling chemicals that produce a response in other organisms

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6
Q

what pheromones do plants release in response to herbivory

A
  • some release ALARM PHEROMONES in the air, in response to herbivore grazing, which causes nearby plants to detect these chemicals to start making chemical defences such as tannins
  • corn plants after being eaten by caterpillars, produce pheromones which attract parasitic wasps, which then lay their eggs in the caterpillars, which eventually kills them
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7
Q

what is a physical response of a plant to being touched

A
  • the plants fold up
  • e.g. if a single leaflet of plant Mimosa pudica is touched, the signal spreads through the whole leaf
  • this causes it to quickly fold up
  • helps protect it against herbivory
  • as knocks off any small insects feeding on the plant
  • scares off animals trying to eat it
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8
Q

what is abiotic stress to plants

A
  • anything living that is natural but non-living
  • e.g. drought
  • e.g. extreme cold
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9
Q

how do some plants respond to extreme cold

A
  • produce their own form of antifreeze
  • e.g. carrots
  • produce antifreeze proteins at low temperature
  • the proteins bind to ice crystals and lower the temperature water freezes at
  • stops more ice crystals from growing
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10
Q

what is a tropism

A

the response of a plant to a directional stimulus (stimulus coming from a particular direction), by regulating their growth

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11
Q

what is positive tropism

A

growth towards the stimulus

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12
Q

what is negative tropism

A

growth away from the stimulus

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13
Q

what is phototropism

A

the growth of a plant in response to light
- shoots are positively phototropic and grow towards the light
- roots are negatively phototropic, and grow away from the light

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14
Q

what is geotropism

A

the growth of a plant in response to gravity
- shoots are negatively geotropic and grow upwards
- roots are positively geotropic and grwo downwards

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15
Q

what is hydrotropism

A

plant growth in response to water
- roots are positively hydrotropic

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16
Q

what is thermotropism

A

plant growth in response to temperature

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17
Q

what is thigmotropism

A

plant growth in response to contact with an object

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18
Q

what brings about some of the responses to stimuli of plants

A

growth hormones (also called growth substances)
- chemicals that speed up or slow down plant growth

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19
Q

where are growth hormones produced

A

in the growing regions of the plant
- shoot tips
- leaves
but they move to where they are needed in other parts of the plant

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20
Q

what does growth hormone gibberellin do

A

stimulates:
- seed germination
- stem elongation
- side shoot formation
- flowering

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21
Q

what do growth hormones auxins stimulate in a plant

A
  • growth of shoots by stem elongation
  • where the cell walls become loose and stretchy
  • so the cells get longer
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22
Q

what can a high concentration of auxins cause

A

inhibition of growth in the roots

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23
Q

where are auxins produced

A

the tips of shoots of flowering plants

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24
Q

what is an example of an auxin, and what does it do

A
  • indoleacetic acid
  • IAA
  • works by stimulating cell elongation
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25
Q

how does IAA move around the plant

A
  • via diffusion and active transport over short distances
  • via phloem over long distances
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26
Q

why does IAA move around a plant

A
  • to control tropisms
  • but its movement means different parts of the plant have different amounts of IAA
  • and an uneven distribution of IAA means that there is uneven growth of the plant
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27
Q

how does IAA respond to phototropism

A
  • IAA moves to the more shaded parts of shoots and roots
  • so in SHOOTS: the cells ELONGATE and bend towards the light
  • and in ROOTS: the growth is INHIBITED and the roots bend away from the light
  • means there is uneven growth
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28
Q

how does IAA respond to geotropism

A
  • IAA moves to the underside of the shoots and roots due to gravity
  • so in SHOOTS: the cells ELONGATE and the shoot grows upwards
  • and in ROOTS: the growth here is INHIBITED, so the root grows downwards
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29
Q

PRACTICAL: how would you carry out an investigation into phototropism

A
  • via showing how plant shoots respond to light:
    1) take 9 wheat shoots, all planted in individual pots, but same type of soil and roughly equal in height
    2) cover the tips of the 3 shoots with a foil cap, leave 3 without foil and wrap the base of the final 3 with foil, so only the tip is exposed
    3) set up the shoots in front of a light source and leave for 2 days
  • need to make sure all shoots are same distance away from the light source, and experience same intensity of light, and all other variables (temperature, exposure to moisture) should be controlled
    4) at end of experiment, should get results
  • you can record the amount of growth (in mm) and direction of growth to give you quantitative results
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30
Q

PRACTICAL: what should be the results of your phototropism investigation

A
  • the shoots with exposed tips: grown towards the light source (as positive tropism)
  • this includes completely exposed and the one with the covered base, and it should still grow towards the light
  • covering the tip with foil cao would’ve prevented growth towards the light
  • AS it is the tip (where the IAA is produced) that is most sensitive to light
  • so if covered, the shoot should just grow straight up
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31
Q

PRACTICAL: how could you perform a practical investigation into geotropism

A

1) line 3 petri dishes with moist (not soaking) cotton wool
2) should use the same volume of water and the same amount of cotton wool in each dish
3) space out 10 cress seeds on the surface of the wool in each dish, then push each one into the wool
4) tape a lid on each dish and wrap in foil (prevents light reaching the wool and affecting your results)
5) store you dishes somewhere with a warmish, pretty constant temperature, e.g. a cupboard
6) prop one dish up, at a 90°, label it and mark which way is up. place another dish on a slope at 45° angle and the third on a flat, horizontal surface
7) make sure you label the dishes carefully, so you know which way is up when you unwrap the dishes at the end of the experiment
8) leave seeds for 4 days
9) observe the seeds’ shoot and root growth
- to get quantitative results, measure the amount of growth of the roots and shoots, as well as the angle of growth

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32
Q

PRACTICAL: what should be your results of the geotropism practical

A
  • whatever the angle the dishes were places, the shoots have grown away from gravity (negative geotropism) and the roots have grown towards gravity (positive geotropism)
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33
Q

what is an apical bud

A

the shoot tip at the top of a flowering plant

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34
Q

what is apical dominance

A

auxins stimulate the growth of the apical bud and inhibit the growth of the side shoots from lateral buds

  • means that the apical bud is dominant over the lateral buds
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35
Q

what does apical dominance prevent

A

prevents side shoots from growing

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36
Q

why is apical dominance good

A
  • saves energy
  • and prevents side shoots from the same plant competing with the shoot tip for light
  • as energy isn’t being used up to grow side shoots, apical dominance allows a plant in an area where there are loads of plants to grow tall very fast
  • grows past the smaller plants, and can reach the sunlight
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37
Q

what happens if you remove the apical bud from a plant and leave it like this

A
  • the plant won’t produce auxins
  • so side shoots will start growing
  • via cell division and cell elongation
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38
Q

what happens if you remove the apical bud from a plant and replace it with a source of auxin

A

e.g. agar block containing auxin
- the side shoot development will still be inhibited
- SHOWS THAT apical dominance is controlled by auxin

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39
Q

why may side shoots still grow towards the bottom of a plant

A
  • auxins become less concentrated as they move away from the apical bud to the rest of the plant
  • so if a plant grows very tall
  • the bottom of the plant has a low auxin concentration
  • so side shoots will start to grow near the bottom
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40
Q

PRACTICAL: how could you investigate the roles of auxins in apical dominance

A

1) plant 30 plants, e.g. pea plants, that are similar age, height and weight in pots
2) count and record the number of side shoots growing from the main stem of each plant
3) for 10 plants, remove the tip of the shoots and apply a paste containing auxins to the top of the stem
3) for other 10, remove tip of the shoot and apply a paste without auxins to the top of the stem
4) leave the final 10 plants as they are: untreated controls [they are needed for comparison so you know the effect you see is likely to be due to the hormone, and not any other factor]
5) let each group grow for 6 days
- keep all plants in the same conditions: light intensity, water [makes sure that any variables that may affect your results are controlled, making the experiment valid]
6) after 6 days, count the number of side shoots growing from the main stem of each of your plants
7) should get results, where without any auxin, the most amount of more side shoots have grown
= show that auxins inhibit the growth of side shoots, and they are involved in apical dominance

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41
Q

where are gibberellins produced

A

in young leaves and seeds

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42
Q

what do gibberellins produce again

A

seed germination, stem elongation, side shoot formation, flowering

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43
Q

how do gibberellins stimulate the stems of plants to grow

A
  • via stimulating stem elongation
  • this helps plants to grow very tall
  • e.g. if a dwarf variety of a plant is treated with gibberellin, it will grow to the same height as the tall variety
  • UNLIKE auxins, gibberellins don’t inhibit plant growth in any way
44
Q

how do gibberellins stimulate seed germination

A
  • trigger the breakdown if starch into glucose in the seed
  • the plant embryo in the seeds can then use the glucose to being respiring
  • can release energy as it needs to grow
  • gibberellins and seed germination can be inhibited and prevented by hormone abscisic acid
45
Q

how have scientists been able to prove the role of gibberellins in seed germination

A
  • have produced genetically altered seeds
  • that are unable to produce gibberellins
  • these seeds are unable to germinate
  • unless given gibberellins
46
Q

PRACTICAL: how could you investigate the role of gibberellins in stem elongation

A

1) plant 40 plants, e.g. dwarf pea plants, that are similar age, height and mass in pots
2) leave 20 plants to grow as they are, watering them all in the same way and keeping them all in same conditions [your controls]
3) leave the other 20 in the same conditions, but water them with a dilute 100 mg dm-3 solution of gibberellins
4) let the plants grow for 28 days and measure the lengths of all the stems once each week
5) results would show that stems grow more when watered with the dilute solution of gibberellin
6) suggesting gibberellin stimulates stem elongation

  • can also calculate the rate of growth of plants too
47
Q

how can gibberellins and auxins be synergistic

A

they work together to have a really big effect, e.g. working together to help a plant grow very tall

48
Q

how can gibberellins and auxins be antagonistic

A

they oppose each others actions, e.g. gibberellins stimulating the growth of side shoots, but auxins inhibiting this

49
Q

what are deciduous plants

A

plants that lose their leaves in winter

  • technical term for leaf loss = abscission
50
Q

why do deciduous plants lose their leaves in the winter

A
  • help to conserve water (which is lost from leaves) during cold parts of the year
  • as here, it may be difficult to absorb water
    from the soil (may be frozen)
  • and there is less light for photosynthesis
51
Q

when is leaf loss triggered

A

by the shortening day length in the autumn

52
Q

which two hormones control leaf loss

A

auxins and ethene

53
Q

how do auxins inhibit leaf loss

A

AUXINS INHIBIT leaf loss:
- auxins are produced by young leaves
- as the leaf gets older, less auxin is produced
- leading to leaf loss

54
Q

how does ethene stimulate leaf loss

A
  • ethene is produced by aging leaves
  • as leaves get older, a layer of cells (called the abscission layer) develops at the bottom of the leaf stalk (where the leaf meets the stem)
  • the abscission layer separates the leaf from the rest of the plant
  • ethene stimulates the cells in the abscission layer to expand, breaking their cell walls
  • causes the leaf to fall off
55
Q

how are ethene and auxins related

A

they are antagonistic to each other

56
Q

why do plants need to close their stomata

A

in order to reduce water loss through respiration

57
Q

what do plants use to close their stomata

A

guard cells
- found either side of the stomatal pore

58
Q

how do guard cells open and close the stomata

A
  • when the guard cells are full of water
  • they are plump and turgid, so the pore is open
  • when the guard cells lose water, they become flaccid, making the pore close
59
Q

which hormone triggers stomatal closure

A

abscisic acid ABA

60
Q

how does ABA trigger stomatal closure

A
  • ABA binds to receptors on the guard wall membranes
  • this causes specific ion channels to open
  • which allows calcium ions to enter the cytosol from the vacuole
  • this increased concentration of Ca+ ions in the vacuole causes other ion channels to open, allowing ions, like K+, to leave the guard cells
  • this raises the water potential of the cells
  • water then leaves the guard cells via osmosis
  • guard cell becomes flaccid and the stomata closes
61
Q

how is ethene used commercially

A

used via the fruit industry to control how different fruits develop:
- ethene stimulates enzymes that break down cell walls, break down chlorophyll and convert starch into sugars
- this makes fruit soft, ripe and ready to eat
- so e.g. bananas are harvested before they are ripe, as less likely to get damaged this way
- then exposed to ethene on arrival
- so they all ripen at the same time
- on shelves and in peoples homes

62
Q

how are auxins used commercially

A
  • as SELECTIVE WEEDKILLERS (HERBICIDES):
  • auxins make weeds produce long stems instead of lots of leaves
  • this makes the weeds grow too fast
  • so they can’t get enough water/nutrients
  • so they die
  • AS ROOTING HORMONES (in ROOTING POWDER):
  • auxins make a cutting (part of a plant) grow roots
  • this cutting can then be planted and grown into a new plant
  • many cuttings can be taken from just one original plant
  • treated with rooting hormones
  • so lots of the same plant can be grown quickly and cheaply from just one plant
63
Q

why do animals respond to changes in their environment, and give examples

A
  • respond to external environment: avoid places that are too hot or cold
  • respond to internal environment: control blood glucose concentration
  • to increase their chances of survival
64
Q

what is a stimuli, and what are the chain of events it causes

A
  • a change in the internal or external environment
  • receptor detects stimuli
  • effectors bring about a response to stimuli
  • receptors will communicate with effectors via the nervous system or the hormonal system
  • sometimes both
65
Q

what are the 2 main structures of the nervous system

A

central nervous system CNS
peripheral nervous system

66
Q

what is your central nervous system

A

your brain and spinal chord

67
Q

what is your peripheral nervous system

A

made up of the neurones that connect the CNS to the rest of the body

68
Q

what are the two different function systems of the peripheral nervous system

A

somatic nervous system and autonomic nervous system

69
Q

what is the somatic nervous system

A

controls your conscious activities, e.g. running or playing a video game

70
Q

what is your autonomic nervous system

A

controls your unconscious activities, e.g. digestion

71
Q

what are the 2 divisions of the autonomic nervous system

A

sympathetic and parasympathetic

72
Q

what is your sympathetic nervous system

A

gets the body ready for action
- the fight or flight system
- releases neurotransmitter noradrenaline

73
Q

what is your parasympathetic nervous system

A

calms the body down
- rest and digest system
- neurones release neurotransmitter acetylcholine

74
Q

what is the location and function of the hypothalamus

A
  • just beneath the middle part of your brain
    1) automatically maintains body temperature to a normal level
    2) produces hormones that control the pituitary gland
75
Q

what is the location and function of your cerebrum

A
  • largest part of your brain
  • divided into two parts called the cerebral hemispheres
  • has a thin outer layer called the cerebral cortex, which is highly folded
    1) involved in vision, hearing, learning and thinking
76
Q

what is the location and function of your pituitary gland

A
  • found beneath the hypothalamus
  • controlled by the hypothalamus
    1) releases hormones and stimulates other glands, e.g. the adrenal gland, to release their hormones
77
Q

what is the location and function of the medulla oblongata

A
  • at the base of your brain, at the top of the spinal chord
    1) automatically controls breathing and heart rate
78
Q

what is the location and function of the cerebellum

A
  • underneath the cerebrum
  • also has a folded cortex
    1) important in muscle coordination, posture and coordination of balance
79
Q

what is a reflex

A

where the body responds to a stimulus without making a conscious decision to respond
- as the pathway of communication doesn’t involve conscious parts of your brain

80
Q

why are reflexes so fast, and why is this good

A
  • you don’t have to spend time deciding how to respond
  • so information travels very fast from receptors to effectors
  • so help an organisms avoid damage to the body
  • as so fast
81
Q

what are 2 types of reflexes

A
  • blinking reflex
  • knee-jerk reflex
82
Q

when does a blinking reflex occur

A
  • when your body detects something that could damage your eye
  • you automatically blink
  • so you quickly close your eyelid to protect your eye
  • then open it again
  • e.g. if eyelid is touched
83
Q

how does the blinking nerve reflex occur, if something touched your eye

A
  • the sensory nerve endings in your cornea (front part of your eye) are stimulated by touch
  • the nerve impulse is sent along the sensory neurone to a relay neurone in the CNS
  • the impulse is then passed from the relay neurone to motor neurones
  • the motor neurones send impulses to the effectors
  • the orbicularis oculi muscles that move your eyelids
  • these muscles contract
  • cause your eyelid to close quickly
  • to prevent your eyelid from being damaged
84
Q

when does a knee-jerk reflex occur, and why

A
  • it works to quickly straighten your leg if the body detects your quadriceps is suddenly stretched
  • helps to maintain posture and balance
  • e.g. if your knees buckle after landing from a jump, the reflex causes your quadriceps to contract to keep you upright
85
Q

how does the knee-jerk reflex take place

A
  • stretch receptors in the quadriceps muscles detect the muscle is being stretched
  • a nerve impulse is passed along the sensory neurone, which communicated directly with a motor neurone in the spinal chord (no relay neurone involved)
  • the motor neurone carries to nerve impulse to the effector (quadricep muscle) causing it to contract, so the lower leg moves forward quickly
86
Q

what happens when an organism is threatened

A
  • it responds by preparing the body for action, e.g. fighting or running away
  • called the “fight or flight” response
  • nerve impulses from the sensory neurones arrive at the hypothalamus, activating both the hormonal system and the sympathetic nervous system
87
Q

how is the hormonal system activated during fight or flight

A
  • the pituitary gland releases a hormone called ACTH
  • this causes the cortex of the adrenal glands to release steroidal hormones
88
Q

how is the sympathetic nervous system activated during fight or flight

A
  • triggers the release of adrenaline from the medulla region of the adrenal gland
89
Q

what are the effects of adrenaline and the sympathetic nervous system on fight or flight

A
  • heart rate is increased: so blood is pumped around the body faster
  • muscles around the bronchioles relax: so breathing is deeper
  • glycogen is converted to glucose (glycogenolysis): so more glucose is available for muscles to respire
  • muscles in the arterioles supplying the skin and the gut constrict, and muscles supplying the heart, lungs and skeletal muscles dilate: so that blood is diverted to the latter
  • erector pili muscles in the skin contract: makes the hair stand on end so the animal looks bigger
90
Q

what systems does control of heart rate involve

A

nervous AND hormonal systems

91
Q

how is heart rate controlled

A
  • the sinoatrial node SAN generates electrical impulses that cause the cardiac muscles to contract
  • the rate at which the SAN fires, and therefore the heart rate, is unconsciously controlled
  • by part of the brain, called the medulla
92
Q

why do organisms need to alter their heart rate

A
  • to respond to internal stimuli
  • e.g. to prevent fainting due to low blood pressure or make sure the heart rate is high enough to supply the body with enough oxygen
93
Q

what 2 receptors detect stimuli to do with heart rate

A

pressure receptors and chemical receptors

94
Q

what are the cardiac pressure receptors

A
  • called baroreceptors
  • in the aorta and the vena cava
  • they’re stimulated by high and low blood pressure
95
Q

what are the cardiac chemical receptors

A
  • called chemoreceptors
  • in the aorta, carotid artery (major artery in the neck), and in the medulla
  • they monitor the oxygen level in the blood
  • also monitor CO2 and pH, which are both indicators of O2 levels
96
Q

what happens once receptors detect a stimuli to do with the heart

A
  • electrical impulses are sent to the medulla along the sensory neurones
  • the medulla processes the information and sends impulses to the SAN along motor neurones
97
Q

what happens during high blood pressure

A
  • baroreceptors detect high blood pressure
  • impulses are sent to the medulla, which sends impulses along the vagus nerve, which secretes acetylcholine, which binds to receptors in the SAN
  • the effector is the cardiac muscles
  • which respond by slowing down the heart rate, so blood pressure reduces down to normal
98
Q

what happens during low blood pressure

A
  • baroreceptors detect low blood pressure
  • impulses are sent to the medulla, which sends impulses along the accelerator nerve, which secretes noradrenaline, which binds to receptors on the SAN
  • the effector is the cardiac muscle
  • which responds by speeding up the heart rate to increase blood pressure back to normal
99
Q

what happens during high blood O2 levels (or low CO2 or high pH)

A
  • chemoreceptors detect chemical changes in the blood
  • impulses are sent to the medulla, which sends the impulses along the vagus nerve, which secretes acetylcholine, which binds to receptors on the SAN
  • the effector is the cardiac muscle
  • and the response is the heart rate decreasing to return O2, CO2 and pH levels back to normal
100
Q

what happens during low O2 (or high CO2, or low pH)

A
  • the chemoreceptors detect chemical changes in the blood
  • the impulses are sent to the medulla, which sends impulses along the accelerator nerve, which secretes noradrenaline, which binds to receptors on the SAN
  • the effector is the cardiac muscle
  • which responds by increasing the heart rate, returning O2, CO2 and pH levels to normal
101
Q

how does your hormonal system assist in controlling heart rate

A
  • releases adrenaline, e.g. during a fight or flight response
  • adrenaline binds to specific receptors in the heart
  • which causes the cardiac muscle to contract more frequently and with more force
  • so heart rate increases
  • and the heart pumps more blood
102
Q

PRACTICAL: how would you measure your own heart rate

A
  • find your pulse on your wrist by placing your index and middle finger where the base of your thumb meets your forearm
  • count the number of beats in 15 seconds
  • then multiply by 4 to get the number of beats per minute
103
Q

PRACTICAL: how would you investigate the effect of exercise on heart rate

A

1) measure heart rate at rest and record it on a table
2) do some gentle exercise, e.g. stepping on and off a step for 5 minutes
3) immediately afterwards, measure your heart rate again
4) return to a resting position
5) measure your heart rate every minute until it returns to the starting rate
6) record how long it takes to return to normal

  • could also use an electrical heart rate monitor instead, which contain sensors to measure your heart rate
104
Q

what is a statistical test you could use for calculating the effect of exercise on a group of people with, or without endurance training

A

a student’s t-Test