5. Molar Incisor Hypomineralisation Flashcards

1
Q

Definition of MIH (2)

A

Hypomineralisation of systemic origin of one to four permanent molars, frequently associated with affected incisors
Usually a developmental condition, found in the period of development between birth and 2yrs

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2
Q

Definition of enamel hypomineralisation

A

Disturbances of enamel formation, resulting in a reduced mineral content (normal enamel thickness but less mineralised so softer)
Qualitative

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3
Q

Definition of enamel hypoplasia

A

Reduced bulk or thickness of enamel (normal but less)

Quantitative

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4
Q

Types of enamel hypoplasia (2)

A

True

Acquired

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5
Q

Definition of true enamel hypoplasia

A

Enamel never formed

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6
Q

Definition of acquired enamel hyperplasia

A

Post-eruptive loss of enamel bulk

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7
Q

Clinical periods of enquiry for MIH (3)

A

Pre-natal (Pre-eclampsia, gestational diabetes)
Natal (fullterm, specific birth trauma)
Post-natal (resp., varicella, MMR)

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8
Q

Incubation period of measles

A

10-14 days

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9
Q

Duration of illness of measles

A

7-10 days

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10
Q

Signs and symptoms of measles (6)

A
Fever
Rash
Conjunctivitis
Cough
Coryza
Koplik's spots
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11
Q

Untreated measles can cause (7)

A
Secondary infection
Otitis media
Bronchopneumonia
Corneal ulcers
Stomatitis
Gastroenteritis
Appendicitis
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12
Q

Duration of illness of rubella

A

8-10 days

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13
Q

Signs and symptoms of rubella (5)

A
Mild fever
Maculopapular rash
Generalised lymphadenopathy (especially sub occipital nodes)
Malaise
URTI
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14
Q

Untreated rubella can cause (3)

A

Encephalitis
Arthritis
Purpura

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15
Q

Duration of illness of varicella/chicken pox

A

6-10 days

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16
Q

Signs and symptoms of varicella (3)

A

Low grade fever
Rash
Crops of spots progressing from macule-papule-vesicle

17
Q

Untreated varicella can cause (3)

A

Secondary infection of lesion
Encephalitis
Pneumonia

18
Q

Features of hypomineralised molars (3)

A

Increased pulp horn and subodontoblastic region neural density
Significant increases in immune cell accumulation (especially with post-eruptive enamel loss)
Significant increase in vascularity in sensitive MIH

19
Q

MIH pain mechanisms (3)

A

Dentine hypersensitivity
Peripheral sensitisation (of C-fibres)
Central sensitisation

20
Q

Clinical problems of MIH(3)

A

Loss of tooth substance (enamel breakdown, tooth wear, secondary caries)
Sensitivity
Appearance

21
Q

MIH treatment (4)

A

Composite/GIC restorations
Stainless steel crowns
Adhesively retained copings
Extraction (8.5-9.5yrs)

22
Q

Treatment of affected incisors (5)

A
Acid pumice microabrasion
External bleaching
Localised composite placement
Full composite veneers
Full porcelain veneers