4th year Flashcards

1
Q

4 components of a full periodontal charting

A

Periodontal pocket depths
Bleeding on probing
Plaque index
Mobility

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2
Q

Define BPE and describe how it is conducted

A

Screening tool for periodontal disease
Conducted by using light probing force (20-25grams) WHO probe (‘ball end’ 0.5mm in diameter and a black band from 3.5mm to 5.5mm) to assess pocket depths in sextants

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3
Q

3 things that must be included in the record when managing a periodontal condition

A

Condition of the periodontal tissues
Risk factors
Periodontal advice and treatment

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4
Q

Define periodontal health

A

Clinical gingival health on an intact or reduced periodontium

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5
Q

Define periodontitis

A

Chronic inflammation of the supporting tissues around the teeth

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6
Q

6 investigations to stage periodontal disease

A

Assessment of greatest site of clinical attachment loss
Assessment of radiographic bone loss
Assessment of tooth loss due to periodontitis
Maximum pocket depth
Furcation involvement
Occlusal trauma

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7
Q

4 step approach to periodontal treatment

A

Step 0: Prerequisite to therapy
Step 1: Risk factor control
Step 2: Intervene
Step 3: Check/review
Step 4: Exit, plan longer-term care

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8
Q

Describe the 4 components of step 0 in the S3 treatment guidelines for periodontitis

A

Educate
Diagnose
Risk assess
Plan

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9
Q

Describe the 3 components of step 1 in the S3 treatment guidelines for periodontitis

A

OHI
PMPR
Supra-gingival scaling

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10
Q

Describe the 2 components of step 2 in the S3 treatment guidelines for periodontitis

A

Sub-gingival biofilm, calculus removal
Adjunct therapy

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11
Q

Describe the 2 components of step 3 in the S3 treatment guidelines for periodontitis

A

Periodontal pocket chart
Re-treatment of non-responder sites

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12
Q

Describe the 2 components of step 4 in the S3 treatment guidelines for periodontitis

A

Plan longer-term, supportive care
Rehabilitation

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13
Q

Define an engaging patient

A

Favourable improvement in OH
Reduce plaque and bleeding scores by 50%
Plaque scores ≤ 20% and bleeding scores ≤ 30%
Progresses to step 2

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14
Q

Describe the management of a non-engaging patient

A

Remain in step 1 until engaged

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15
Q

Describe the evidence-based recommendations for the choice of toothbrush

A

The use of a powered toothbrush may be considered as an alternative to manual tooth brushing for patients in supportive periodontal care

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16
Q

Describe the evidence-based recommendations for interdental cleaning

A

Recommend that tooth brushing should be supplemented by the use of interdental brushes (where anatomically possible) for patients in supportive periodontal care

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17
Q

Describe the evidence-based recommendations for the use of floss

A

Do not suggest the use of floss as the first-choice method of interdental cleaning for patients in supportive periodontal care

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18
Q

Describe the evidence-based recommendations for the use of other interdental cleaning devices

A

Suggest the use of other interdental cleaning devices in interdental areas, not reachable by interdental brushes, for patients in supportive periodontal care

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19
Q

Describe the evidence-based recommendations for the use of adjunctive locally administered antiseptics

A

Locally administered sustained-release chlorhexidine may be considered as an adjunct to sub-gingival instrumentation in patients with periodontitis

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20
Q

Describe the evidence-based recommendations for the timing of delivery of sub-gingival root
instrumentation

A

Supportive periodontal care should be scheduled for intervals of 3 to a maximum of 12 months, with the frequency determined by the patient’s risk profile and periodontal status after active therapy

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21
Q

Epidemiology of periodontitis

A

8-12% of population

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22
Q

Define a risk factor

A

Factor that is biologically linked to the disease that can be modified

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23
Q

6 risk factors for periodontal disease

A

Smoking
Diabetes
Stress
Drugs
Systemic diseases
Nutrition

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24
Q

6 systemic diseases that can have an effect on periodontal health

A

Ehlers Danlos Syndrome
Hypophosphatasia
Down syndrome
Chediak higashi syndrome
Papillion-Lefevre syndrome
Leucocyte adhesion deficiency

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25
Q

1 mechanisms behind why Down syndrome increases risk of periodontal disease

A

Underlying neutrophil disorder

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26
Q

1 mechanisms behind why Papillon-Lefevre syndrome increases risk of periodontal disease

A

Mutation in cathepsin C gene and enzyme required for activation neutrophil-derived antimicrobial peptide

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27
Q

6 effects of smoking on periodontal status

A

Increases risk of developing periodontal disease
Greater frequency of diseased sites
Greater reduction of periodontal bone height
Poorer response to periodontal therapy
Increased risk of continuing loss of attachment
More prone to gingival recession/furcation defects

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28
Q

4 mechanisms behind why smoking increases risk of periodontal disease

A

Impaired PMN function
Reduced gingival blood flow
Impaired healing
Effects plaque microbial flora

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29
Q

Effect of age on periodontal status

A

Prevalence of periodontitis increases with age

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30
Q

Effect of diabetes on periodontal status

A

Increases risk of developing periodontal disease

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31
Q

4 mechanisms behind why diabetes increases risk of periodontal disease

A

Dysregulation of PMNs function
Altered collagen metabolism
Micro vascular damage

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32
Q

Describe the relationship between periodontal disease and diabetic control

A

Significant periodontal disease increases risk of worsening glycaemic control

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33
Q

Effect of psychological stress on periodontal status

A

Increases risk of developing periodontal disease

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34
Q

Effect of scurvy on periodontal status

A

Increases gingivitis and loss of attachment leading to tooth loss

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35
Q

1 mechanism behind why scurvy increases risk of periodontal disease

A

Lack of vitamin C affects formation of collagen fibres

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36
Q

Effect of pregnancy on periodontal status

A

Increased gingival inflammation progressively during pregnancy

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37
Q

Effect of IL-1 gene polymorphisms on periodontal status

A

Associated with advanced periodontal disease

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38
Q

3 drugs associated with drug induced gingival overgrowth

A

Phenytoin
Cyclosporin
Calcium channel blocker amlodipine

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39
Q

5 tooth related risk factors for periodontal disease

A

Anatomical risk factors
Tooth position
Iatrogenic risk factors
Xerostomia
Occlusal trauma
Incompetent lip posture

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40
Q

Describe a risk assessment tool for periodontal disease

A

PreViser uses smoking history, diabetic status and clinical findings to assess a patients risk of developing periodontal disease

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41
Q

4 properties of chlorhexidine

A

Anti-plaque
Anti-microbial
Substantivity
Highly effective

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42
Q

Describe the substantivity property of chlorhexidine

A

Ability to bind to soft and hard tissues, increasing working time

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43
Q

Concentration of standard chlorhexidine mouthwash UK

A

0.2% chlorhexidine gluconate, 10ml rinse (20mg)

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44
Q

Concentration of Corsodyl Daily Defence mouthwash

A

0.06% Chlorhexidine Digluconate

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45
Q

3 indications for the use of chlorhexidine mouthwash

A

Post-scaling and root instrumentation
Post periodontal surgery
Acute gingival infections

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46
Q

4 disadvantages of chlorhexidine mouthwash

A

Interacts with sodium lauryl sulphate in toothpaste decreasing chlorohexidine activity
Brown/black staining of teeth
Altered taste sensation
Potential to cause parotid swelling

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47
Q

Indication for use of Corsodyl Daily Defence mouthwash

A

Part of a maintenance program to limit staining

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48
Q

Indication for use of chlorhexidine gel

A

Patients with localised gingivitis associated with mouthbreathing

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49
Q

1 reason antibiotics are used in periodontal therapy

A

Facilitate removal of sub-gingival bacteria

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50
Q

4 reasons against of the use of antibiotics in periodontal therapy

A

No penetration of antibiotics without mechanical disruption of the plaque biofilm
Potential systemic upset
Potential bacterial resistance
Non-surgical periodontal therapy is successful without antibiotics

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51
Q

6 topical antimicrobials

A

Dentomycin
Atridox
Actisite
Arestin
Elyzol
Periochip

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52
Q

4 advantages of topical antimicrobials

A

Direct to site of action
Low dosage required
No systemic upset
Reduced risk of resistance

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53
Q

5 indications for the use of topical antimicrobials

A

Localised disease
Sites showing poor response to mechanical treatment in otherwise stable patients
Localised molar/incisor periodontitis
Chronic, recurrent periodontal abscesses
Adjunctive to debridement in deep or recurrent periodontal sites

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54
Q

Describe Periochip

A

Slow release 2.5mg chlorhexidine from bovine gelatin carrier
Designed to be used in periodontal pockets ≥5mm

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55
Q

4 properties of Periochip

A

Delivers peak concentration at 2 hours
Continued release over 10 days
Self-retentive in pocket
No bacterial resistance

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56
Q

3 indications for use of systemic microbials

A

Generalised disease
Adjunctive to excellent mechanical debridement
For access to periodontal and other oral sites

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57
Q

6 stages in the treatment of generalised advanced chronic or aggressive periodontitis

A

Extraction of hopeless teeth
Endodontic therapy
Non-surgical periodontal therapy
Systemic antibiotic therapy
Periodontal surgery
Maintenance

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58
Q

2 systemic microbials used in periodontal therapy

A

Amoxycillin 250mg tid 5-7 days + Metronidazole 200mg tid 5-7 days
Tetracycline 250mg qid 7 days

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59
Q

Describe Periostat

A

Low-dose doxycyclineused as part of maintenance programme for patients with generalised, aggressive periodontitis

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60
Q

4 features of Periostat

A

Sub-MIC levels
Anti-collagenase effect
Single dose daily over 9 months
No bacterial resistance problems

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61
Q

4 features of localised molar-incisor periodontitis in young adults

A

Affects younger age group (<35 years)
Rapid attachment loss and bone destruction
Patients otherwise healthy
Microbial and plaque deposits inconsistent with levels of destruction

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62
Q

1 risk determinant for periodontitis in young adults

A

Family history

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63
Q

1 causative bacteria linked to periodontitis in young adults

A

Aggregatibacter actinomycetemcomitans

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64
Q

5 mechanisms of Aggregatibacter actinomycetemcomitans leading to periodontitis in young adults

A

Releases lethal leucotoxin directed against neutrophils and monocytes
LPS induces bone resorption
Produces enzymes such as collagenase
Chemotactic inhibition factors
Resists phagocytosis

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65
Q

5 stages in the treatment of periodontitis in young adults

A

OHI, plaque control, smoking cessation advice
Extraction of hopeless teeth
Sub-gingival scaling and root instrumentation
Local delivery of Dentomycin, Atridox
Periodontal surgery
Maintenance, follow-up

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66
Q

Describe dentomycin

A

2% minocycline hydrochloride local anti-microbial with anti-collagenase activity

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67
Q

Describe atridox

A

10% doxycycline hyclate local anti-microbial with anti-collagenase activity
Liquid polymer, hardens on contact with fluid

68
Q

4 acute gingival conditions

A

Periodontal abscesses
Necrotising gingivitis
Necrotising periodontitis
Acute herpetic gingivostomatitis

69
Q

Define acute periodontal abscesses

A

Sudden build up of pus within a pocket that will track until it is able to drain either through pocket or gingival tissues

70
Q

4 signs of an acute periodontal abscess

A

Well localised pain to a vital tooth
Tooth ’elevated’ in its socket
Redness / swelling around gingiva
Pointing on gingiva

71
Q

Management of an acute periodontal abscess

A

Drainage through the pocket by thorough subgingival debridement with LA or lanced with a scalpel to allow pus drainage
Follow-up

72
Q

Define chronic periodontal abscesses

A

Long-standing suppurating pocket

73
Q

3 potential causes of traumatic gingival lesions

A

Toothbrushing
Flossing
Toothpicks

74
Q

1 sign of trauma from toothbrushing

A

Elongated superficial lesions predominantly found buccally

75
Q

1 sign of trauma from flossing

A

Interdental lesion with linear cleft on buccal or lingual gingiva

76
Q

Management of traumatic gingival lesions

A

Removal of the offending agent and symptomatic therapy

77
Q

Define necrotising gingivitis

A

Opportunistic infection commonly affecting interdental papillae

78
Q

3 predisposing factors to necrotising gingivitis

A

Smoking
Stress
Immuno-compromised

79
Q

Bacterial aetiology of necrotising gingivitis

A

Anaerobic bacteria
Gram -ve motile rods
Spirochaetes

80
Q

4 signs of necrotising gingivitis

A

Acute pain in gingivae
Punched out ulceration of tips of interdental papillae
Gingival ulcers bleed spontaneously/readily
Foetor oris

81
Q

Define necrotising periodontitis

A

Infection characterised by necrosis of gingival tissues, PDL and alveolar bone

82
Q

2 signs of necrotising periodontitis

A

Deep interproximal craters with denudation of alveolar bone
Sequestration of interdental or buccal/lingual bone

83
Q

Management of necrotising gingivitis and periodontitis

A

Initial debridement under LA
Prescribe chlorhexidine mouthwash, symptomatic management
Severe cases 3 day course 200mg metronidazole x3 daily or amoxycillin 250 mg x3 daily for 5 days
Follow-up

84
Q

Define acute herpetic gingivostomatitis

A

Infection of the oral cavity which is caused by the herpes simplex virus (HSV)

85
Q

2 signs of acute herpetic gingivostomatitis

A

Multiple lesiosn with rupture to form ulcers (<5mm) over gingivae and oral mucosa with erythematous margin
Swollen gingivae

86
Q

Management of acute herpetic gingivostomatitis

A

Supportive and symptomatic management as condition is self-limiting over 7 days

87
Q

Define gingival recession

A

Apical migration of the gingival margin below the cement-enamel junction leading to exposure of the root surface accompanied with osseous/bone recessions

88
Q

Prevelance of gingival recession

A

20-60% of the population

89
Q

4 anatomical predispositions to gingival recession

A

Bone dehiscence
Bone fenestration
Thin gingival biotype
High frenal attachment

90
Q

Define bone dehiscence

A

Defect that results in lowering of the crestal bone margin to expose the root surface

91
Q

Common site of bone dehiscence

A

Labial/buccal aspect of maxillary canines and mandibular anterior teeth

92
Q

Define bone fenestration

A

Window of bone loss on the lingual/buccal aspect of a tooth that exposes root surface

93
Q

Describe the assessment of periodontal phenotype

A

Periodontal probe inserted into the sulcus:
 Probe visible: thin (≤1 mm)
 Probe not visible: thick (> 1 mm)

94
Q

Describe the relationship between high frenal attachment and gingival recession

A

High frenal attachment can cause an apical pull of gingival tissues and gingival recession

95
Q

5 acquired factors linked to gingival recession

A

Aggressive toothbrushing
Occlusion, parafunctional habits
Orthodontic treatment
Trauma
Restorations impinging on the biologic width

96
Q

Describe the relationship between occlusion and parafunctional habits and gingival recession

A

Repeated gingival trauma due to occlusion or parafunctional activity may lead to gingival recession

97
Q

Describe the relationship between orthodontic treatment and gingival recession

A

Tooth/root movements through bony envelope may lead to dehiscence and risk of recession

98
Q

Define biological width

A

The distance from the junctional epithelium and connective tissue attachment to the root surface of a tooth (2mm)

99
Q

4 consequences of crown margin impinging on biologic width

A

Gingival inflammation
Pain
Gingival recession
Underlying bone resorption

100
Q

3 aquired pathological factors linked to gingival recession

A

Periodontal disease
Periodontal treatment
Factitious trauma

101
Q

Describe the relationship between periodontal treatment and gingival recession

A

Treatment may induce gingival recession as healthy gingival tissues migrate to sit 2-3mm above bone or recession becomes more noticeable as gingival swelling decreases

102
Q

Describe the relationship between factitious trauma and gingival recession

A

Self-induced injury of the periodontal tissues as a result of repeated voluntary trauma to localised areas may lead to gingival recession

103
Q

Describe Miller’s classification of gingival recession

A

Presence of interdental papilla is the most important factor for determining root coverage

104
Q

Define Miller’s I classification

A

Recession short of muco-gingival junction, normal papillary height, no periodontal bone loss in the interdental area, 100% root coverage

105
Q

Define Miller’s II classification

A

Recession at or apical to mucogingival junction, normal papillary height, no periodontal loss in the interdental area, 100% root coverage

106
Q

Define Miller’s III classification

A

Recession at or apical to mucogingival junction, some reduction in papillary height, bone or soft tissue loss in the interdental area or malpositioning of the teeth preventing 100% root coverage

107
Q

Define Miller’s IV classification

A

Recession at or apical to mucogingival junction; significant loss of papillary height to a level apical to marginal soft tissue recession

108
Q

4 consequences of gingival recession

A

Root caries
Abrasion cavities
Dental hypersensitivity
Poor aesthetics

109
Q

Describe level 1 management of gingival recession

A

Prevention: OHI, atraumatic toothbrushing technique, scaling and root debridement

110
Q

Describe level 2 management of gingival recession

A

Surgical correction if indicated

111
Q

Describe level 3 management of gingival recession

A

Maintenance and monitoring

112
Q

6 factors improving success of surgical treatment

A

Gingival margin is on the CEJ (class I,II)
PPD<3mm
No BOP
Highly motivated patients with excellent plaque control
Adequate width of attached gingiva
Thick gingival biotype

113
Q

4 treatment options for gingival recession

A

Maintain current gingival position and prevent progression
Composite additions to teeth
Gingival veneer
Mucogingival surgery

114
Q

Describe 3 features of free gingival graft tissue

A

Generally from firm keratinised tissue of the palate
Includes epithelium and underlying connective tissue lamina propria
Should be 1-2mm thick

115
Q

Describe 3 features of a connect tissue graft

A

Underlying connective tissue lamina propria only
Healing is improved, heals by primary intention
Improved aesthetic outcome

116
Q

Describe 3 features of a coronal advancement flap

A

One surgical site
Improved vascularisation and colour match
Predictable only in shallow recessions

117
Q

Describe 4 features of periodontal surgery with repositioning

A

Flap surgery with apically repositioned flap
Allows access for bone removal
Preserves keratinised gingival tissue
Heals more rapidly than Gingivectomy

118
Q

Give an alternative to grafts

A

Synthetic materials

119
Q

3 examples of synthetic materials

A

Mucograft
Mucoderm
Aloederm

120
Q

2 features of synthetic materials

A

Increase thickness of keratinised tissue
Can be used where there is no suitable donor site to harvest material

121
Q

Describe Brannstrom’s theory of dentine sensitivity

A

Fluid movement in dentinal tubules stimulates nerve endings and causes short, sharp pain which resolves immediately on removal of stimulus
Level of sensitivity related to number of exposed dentinal tubules

122
Q

3 symptomatic treatments for dentine hypersensitivity

A

Topical fluoride
Unfilled resin eg Seal and Protect
Restoration with GI or composite

123
Q

5 indications for flap surgery

A

Access for root surface instrumentation
Modification of osseous furcation defects
Apical repositioning of gingival tissues/crown lengthening
Periodontal regeneration
Root resection/hemisection

124
Q

3 indications for excisional surgery

A

Management of drug-induced gingival overgrowth
Tissue biopsy
Crown lengthening

125
Q

1 indication for mucogingival surgery

A

Management of recession

126
Q

3 indications for crown lengthing

A

Aesthetic reasons
Complications with existing restorative work
Toothwear

127
Q

5 features to consider when selecting patients suitable for periodontal flap surgery

A

Good compliance and response but some residual pockets ≥5mm and bleeding
Re-instrumentation unsuccessful
Bleeding on probing (from bottom of pockets) Residual deep periodontal pocketing
Non-smoker

128
Q

4 stages of the surgical flap procedure

A

Crevicular incision that splits periosteum using firm pressure from scalpel
Careful mucoperiosteal flap elevation
Scaling and root planning
Suturing

129
Q

2 important features in surgical flap design

A

Ensure broad base of flap so as to allow good vascular supply to tissue
Include papilla in flap

130
Q

2 options for surgical flap design

A

Envelope flap
Relieving incisions

131
Q

3 features of post-operative care after periodontal flap surgery

A

Chlorhexidine mouthwash start next day
Use soft brush
Review in 1 week for suture removal

132
Q

3 potential responses to periodontal flap surgery

A

Gingival recession
Exposed root surface
Minimal residual pocketing

133
Q

Describe 3 stages of Modified Widman approach internal bevel incision

A

Initial incision splits the periosteum
Crevicular incision
Incision to remove wedge of gingival tissue

134
Q

2 types of resective gingival surgery

A

Gingivectomy
Gingivoplasty

135
Q

Define gingivectomy

A

Surgical excision of gingival tissue to eliminate soft tissue pockets

136
Q

Define gingivoplasty

A

Surgical re-contouring or reshaping of gingival tissue

137
Q

Describe 2 stages of external bevel incision

A

Blade at 45 degree angle to gingival tissue, use Blake’s or Kirkland scapel
Recontour the gum or to excise areas of gingival overgrowth

138
Q

Describe 1 feature of excisional surgery

A

Leaves a broad wound which requires a dressing to allow healing by primary intention

139
Q

4 indications for resective gingival surgery

A

To eliminate supra bony pockets after completion of non surgical treatment
To improve the aesthetics
Crown lengthening to facilitate restorative procedures
Management of drug-induced gingival overgrowth

140
Q

1 feature of post-operative care after resective gingival surgery

A

Coe-pak for 5 days following surgery

141
Q

Define occlusal trauma

A

Injury to the periodontium occurring as a result of occlusal forces in excess of the reparative or adaptive capacity of periodontium

142
Q

Does occlusal trauma commonly occur with or independently of periodontal disease

A

Commonly with periodontal disease

143
Q

Define primary occlusal trauma

A

Due to excessive occlusal forces applied to teeth with normal periodontal support, periodontally healthy teeth

144
Q

Define secondary occlusal trauma

A

Due to normal or excessive forces applied to teeth with reduced periodontal support

145
Q

Define grade I mobility

A

Tooth movement less than 1mm in buccal-lingual direction

146
Q

Define grade II mobility

A

Tooth movement greater than 1mm in buccal-lingual direction

147
Q

Define grade III mobility

A

Tooth movement greater than 1mm in buccal-lingual direction and can be depressed in the socket

148
Q

Give a primary indicator of occlusal trauma

A

Tooth mobility

149
Q

Describe the relationship between secondary occlusal trauma and tooth mobility

A

In secondary occlusal trauma a smaller non-axial force causes the same horizontal movement because the position of fulcrum is lower due to reduced supporting bone

150
Q

4 stages in pathogenesis of occlusal trauma

A

Increased occlusal forces overwhelm the physical limitations of the PDL
PDL vessels undergo haemorrhage, thrombosis oedema and increased vascular permeability
Periodontal fibres become disorganised and collagen destruction follows
Increased osteoclastic activity and cemental resorption leads to bone resorption in pressure zones

151
Q

Define fremitus

A

Tooth displacement caused by the patients own occlusal forces

152
Q

Describe how to assess fremitus

A

Place a finger on the buccal aspect of the maxillary teeth
Ask the patient to tap the teeth together in inter-cuspal position and lateral and protrusive contacts
Palpate and visualise for displacement

153
Q

Describe 3 common clinical signs of traumatic occlusion

A

Class II incisor relationships and with deep overbites
Stripping of upper incisor palatal gingiva or lower incisor buccal gingiva
Increased probing depths on the palatal aspect of upper incisors

154
Q

Describe 4 common radiographic signs of traumatic occlusion

A

Alveolar bone loss due to periodontitis
Widening of PDL
Funneling of periodontal ligament
Reduced crown - root ratio

155
Q

Describe the relationship between stress and periodontal disease

A

Increased risk for progressive periodontitis in adults with psychological traits of stress

156
Q

Describe the relationship between stress and bruxism

A

Increased stress is associated with clenching/grinding causing increased occlusal load and subsequent PDL lidening

157
Q

3 managements of occlusal trauma

A

Grind teeth out of occlusion
Splint if mobility persists despite resolution of pocketing
Restore missing posterior support with RPD to reduce occlusal load

158
Q

Describe aTwistflex wire splint

A

Passive wire splint cemented in place with flowable composite

159
Q

Describe a fibre splint

A

Fibre mesh placed on teeth and cemented with composite

160
Q

Describe a composite only splint

A

Composite flowed around teeth, rigid splint

161
Q

2 disadvantages of a composite only splint

A

Bulky and interferes with cleaning
Rigid, high chance of fracture

162
Q

Describe a cast metal resin-retained splint

A

Specific lab made metal splint

163
Q

1 disadvantages of cast metal resin-retained splint

A

Rigid, very high chance of debond

164
Q

4 considerations for occlusal splinting

A

Must provide with an occlusal splint
Preferable for splint to incorporate stable canines
Must be be sufficient enamel for etching, crowns not suitable
Not suitable for teeth with Class III mobility

165
Q

Define natural pontics

A

Use crown of compromised tooth as bridge pontic

166
Q

Indication for natural pontics

A

Technique used for mobile teeth with very poor prognosis

167
Q

5 stages of natural pontic provision

A

Compromised tooth extracted and root sectioned off at previous level of gingiva + 2mm
Extirpate pulp through apical foramen and clean pulp chamber with sodium hypochlorite
Seal apical portion of crown with composite
Splint cemented onto remaining teeth
Crown of extracted tooth cemented onto splint