3. Abdominal mass/distension Flashcards

1
Q

causes of abdominal swelling

A

6 Fs
flatus
fat
fluid
feces
fatal growth

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2
Q

what is flatus made up of

A

increased intestinal gas
nitrogen and oxygen are swallowed while CO2, hydrogen and methane are produced intraluminally by bacterial fermentation

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3
Q

aerophagia is

A

the swelling of air - can result in an increased amount of oxygen and nitrogen in the small intestine (flatus) and lead to abdominal swelling

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4
Q

aerophagia results from

A

gulping food, chewing gum, smoking, or as a response to anxiety which can lead to repetitive belching.

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5
Q

increase in production of intestinal gas is caused by

A

bacterial metabolism of excess fermentable substances such as lactose and other oligosaccharides, which can lead to production of hydrogen, carbon dioxide, or methane.

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6
Q

impaired transit of gas

A

In some persons, particularly those with irritable bowel syndrome and bloating, the subjective sense of abdominal pressure is attributable to impaired intestinal transit of gas rather than increased gas volume.

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7
Q

fat

A

Abdominal fat may be caused by an imbalance between caloric intake and energy expenditure associated with a poor diet and sedentary lifestyle; it also can be a manifestation of certain diseases, such as Cushing’s syndrome.

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8
Q

fluid

A

The accumulation of fluid within the abdominal cavity (ascites) often results in abdominal distention

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9
Q

grades of ascites

A

Grade 1 ascites is detectable only by ultrasonography; grade 2 ascites is detectable by physical examination; and grade 3 ascites results in marked abdominal distention.

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10
Q

fetus

A

Typically, an increase in abdominal size is first noted at 12–14 weeks of gestation, when the uterus moves from the pelvis into the abdomen. Abdominal distention may be seen before this point as a result of fluid retention and relaxation of the abdominal muscles.

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11
Q

feces

A

In the setting of severe constipation or intestinal obstruction, increased stool in the colon leads to increased abdominal girth. These conditions are often accompanied by abdominal discomfort or pain, nausea, and vomiting and can be diagnosed by imaging studies.

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12
Q

fatal growth

A

Neoplasms, abscesses, or cysts can grow to sizes that lead to increased abdominal girth. Enlargement of the intraabdominal organs, specifically the liver (hepatomegaly) or spleen (splenomegaly), or an abdominal aortic aneurysm can result in abdominal distention. Bladder distention also may result in abdominal swelling.

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13
Q

excessive alcohol and jaundice suggests

A

ascites

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14
Q

Hx of heart failure or TB suggests

A

ascites

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15
Q

increased eructation and flatus suggests

A

aerophagia or increased intestinal production of gas

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16
Q

inability to pass stool or flatus together with nausea and vomiting suggests

A

bowel obstruction

17
Q

weight loss, night sweat and anorexia indicates

A

neoplastic

18
Q

presence of lymphadenopathy, especially supraclavicular (Virchow’s node) suggests

A

metastatic abdominal malignancy

19
Q

kussmaul’s sign is

A

elevation of JVP during inspiration

20
Q

elevated JVP, kussmauls sign, a murmur of tricuspid regurgitation or pericardial knock indicate

A

ascites

21
Q

spider angiomas, palmar erythema, caput medusae or gynecomastia indicate

A

liver disease

22
Q

caput medusae

A

dilated superficial veins around the umbilicus

23
Q

absence of bowel sounds or presence of localised high pitched bowel sounds indicates

A

ileus or intestinal obstruction

24
Q

umbilical venous hum suggests

A

portal hypertension

25
Q

harsh bruit over the liver suggests

A

heard rarely in patients with hepatocellular carcinoma or alcohol-associated hepatitis

26
Q

abdo swelling by fluid/gas/solid mass can be differentiated by

A

percussion

27
Q

abdomen filled with gas on percussion

A

tympanic

28
Q

abdomen containing mass or fluid on percussion

A

dull

29
Q

absence of abdominal dullness on percussion

A

cannot exclude ascites

minimum of 1500ml of ascitic fluid is required for detection on physical examination

30
Q

nodular liver suggests

A

cirrhosis or tumour

31
Q

pulsations on palpation of the liver

A

Light palpation of the liver may detect pulsations suggesting retrograde vascular flow from the heart in patients with right-sided heart failure, particularly tricuspid regurgitation.

32
Q

abdo x-ray in intestinal obstruction of ileus

A

dilated loops of bowel

33
Q

abdo US can detect

A

as little as 100 mL of ascitic fluid, hepatosplenomegaly, a nodular liver, or a mass.

often inadequate to detect retroperitoneal lymphadenopathy or a pancreatic lesion because of overlying bowel gas.

34
Q

if malignancy or pancreatic disease is suspected

A

If malignancy or pancreatic disease is suspected, CT can be performed. CT may also detect changes associated with advanced cirrhosis and portal hypertension

35
Q

laboratory evaluation

A

should include (to assess hepatic function):

  • liver biochemical testing
  • serum albumin level measurement
  • prothrombin time determination (international normalized ratio)
    and:
  • complete blood count to evaluate for the presence of cytopenias that may result from portal hypertension or of leukocytosis, anemia, and thrombocytosis that may result from systemic infection.
  • Serum amylase and lipase levels should be checked to evaluate the patient for acute pancreatitis.
  • Urinary protein quantitation is indicated when nephrotic syndrome, which may cause ascites, is suspected.
36
Q

if liver cirrhosis is suspected

A

In selected cases, the hepatic venous pressure gradient (pressure across the liver between the portal and hepatic veins) can be measured via cannulation of the hepatic vein to confirm that ascites is caused by cirrhosis. In some cases, a liver biopsy may be necessary to confirm cirrhosis.