2023 Cardio Residents Exam Flashcards

1
Q

Most common cause of mitral regurgitation (Webb Thoracic Imaging, 3rd ed, p2464)

A

Annular calcification

BRANT, 5th ed
Mitral valve prolapse (MVP) is defined as bowing or prolapse of the mitral leaflet of 2 mm or more beyond the annular plane into the left atrium in ventricular systole. MVP is the most common cause of severe nonischemic mitral regurgitation.

Webb, 3rd
PROLAPSE VS FLAIL
Prolapse is ballooning of the middle of the valve beyond the annulus during systole; the tips of the leaflets to which the chordae attach do not pass beyond the annulus.

Flail is indicated by passage of the tips of the mitral leaflets beyond the annulus and into the left atrium during systole.

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2
Q

The most common clinical feature of dilated cardiomyopathy is:

A

Left ventricular failure

Other choices:
-Ventricular enlargement
-Increased wall thickness of the left ventricle (FALSE: Thickness is often mildly thinned or normal)
-End-diastolic volume is decreased (FALSE: INCREASED end-diastolic volume, DECREASED ejection fraction)

Brant, 5th
Nonischemic-dilated cardiomyopathies are characterized by cardiac chamber dilation coupled with IMPAIRED CONTRACTILITY OF THE LEFT VENTRICLE or BOTH THE LEFT VENTRICLE AND RIGHT VENTRICLE. Approximately 50% of dilated cardiomyopathies are IDIOPATHIC or genetic in origin

Dilated cardiomyopathies all have:
1. Increased systolic and diastolic volumes with decreased ejection fractions
2. Ventricular thickness is often mildly thinned to normal and is associated with increased end-diastole volumes, greater than 140 mL for the LV and greater than 150 mL for the RV, indexed to body surface area

SSPF cine imaging is used to assess global or regional ventricular dysfunction and decreased LV and/or RV ejection fractions.

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3
Q

The second most frequent cause of asymmetric distribution of pulmonary edema

A

Chronic obstructive pulmonary disease

Webb, 3rd
Most frequent: GRAVITATIONAL
Patients with heart disease frequently sleep lying on their right side because of consciousness of the prominent left-sided pulsation (prominent point of maximum impulse in the presence of cardiomegaly)

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4
Q

What is the most common primary pericardial tumor? (Webb & Higgins, Thoracic Imaging 3rd ed., p 2602)

A

Mesothelioma

Secondary tumors of the pericardium are far more common. These occur from local invasion of lung and mediastinal malignancies and from distant metastases. Lymphomas, melanomas, lung, and breast carcinomas are the most common primary tumors that involve the pericardium

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5
Q

Most common cause of constrictive pericarditis in developing countries (Webb & Higgins, Thoracic Imaging 3rd ed., p 2570)

A

Tuberculosis

Most common cause of constrictive pericarditis in industrialized countries: CARDIAC SURGERY

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6
Q

On chest x-ray there was a right cardiophrenic mass, what will be your impression? (Webb & Higgins, Thoracic Imaging 3rd ed., p 2596)
a. Pericardial diverticulum
b. Pericardial cyst
c. Both but most likely B.
d. None of the above.

A

c. Both but most likely B.

Webb, 3rd
Pericardial cysts are caused by developmental abnormalities and are alleged to occur when a small portion of the pericardium is pinched off during embryonic development. Pseudocyst of the pericardium may develop after surgical pericardiotomy. Ninety percent of pericardial cysts are located in the cardiophrenic angles (70% on the right and 20% on the left side).

Pericardial cysts usually do not communicate with the pericardial cavity. In contrast, diverticula have a narrow communication with the pericardial cavity. With increase or decrease in pericardial fluid, diverticula can change in size.

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7
Q

Which among the following is the most common vascular anomaly? (Swischuk p. 305)

A

Aberrant right subclavian artery

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8
Q

The most common pericardial tumor in childhood. (Swischuk p. 327)

A

Teratoma

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9
Q

The most common cause of arterial aneurysm in the pediatric age group. (Swischuk p. 333)

A

Mycotic aneurysm

Most are encountered in neonates and young infants undergoing umbilical artery catheterization

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10
Q

Which of the following is not an associated finding of chronic venous thrombi? (Swischuk p. 331)
a. Compensatory dilatation of the azygos vein
b. Oval or bullet-shaped vascular calcification
c. Development of collateral vessels
d. None of the above

A

d. NOTA

Predisposing factors, much as with arterial thromboembolism, include sepsis, dehydration, and mechanical injury secondary to indwelling catheters.

Mature thrombus:
1. Calcify then produce characteristic oval or bullet-shaped calcifications in the various vessels involved
2. With IVC obstruction, compensatory dilatation of the azygos vein is noted. This occurs because collateral venous return must be accomplished through the azygos or hemiazygos venous systems when the inferior vena cava is obstructed.

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11
Q

Myxoma is the most common benign cardiac tumor. In which chamber is it most commonly found?

A

Left atrium

Webb, 3rd
Myxoma is the most common benign cardiac tumor. It is located in the left atrium in 75% of cases and in the right atrium in 20% of cases. Multiple atrial myxomas may occur rarely, especially in Carney’s syndrome.

Left atrial myxomas are typically attached by a narrow pedicle to the area of the fossa ovalis. Myxomas can grow through a patent foramen ovale and extend into both atria, a condition that has been described as a “dumbbell” appearance.

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12
Q

What is the second most common benign cardiac tumor? (Webb 2634)

A

Lipoma

Webb, 3rd
Lipomas are reported to be the second most common benign cardiac tumor in adults but may actually be the most common.

If the mass projects into the right atrium, it is called a lipoma, while lipomatous hypertrophy is confined to the atrial septum.

Lipomas are typically located in the right atrium or atrial septum

Lipomatous hypertrophy is defined as a deposition of fat in the atrial septum around the fossa ovalis that exceeds 2 cm in transverse diameter. It spares the fossa ovalis, a characteristic feature that is clearly delineated with T1- weighted SE images

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13
Q

What is the most common cardiac tumor in children? (Webb 2637)

A

Rhabdomyoma

Webb, 3rd
Rhabdomyomas are the most common cardiac tumors in children, representing 40% of all cardiac tumors in this age group. Thirty to fifty percent of rhabdomyomas occur in patients with tuberous sclerosis.

They are characterized by an intramural location and involve equally the left and right ventricles (RV).

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14
Q

Pheochromocytoma are predominantly encountered within and around the left atrium. What is the most common clinical symptom for patients with pheochromocytoma? (Webb 2642)

A

Hypertension

Webb, 3rd
Pheochromocytomas arise from neuroendocrine cells clustered in the visceral paraganglia in the wall of the left atrium, roof of the right and left atrium, atrial septum, behind the ascending aorta, and along the coronary arteries.

Hypertension, the most common symptom, is related to catecholamine overproduction by the mass. The average age at diagnosis is 30 to 50 years.

Cardiac pheochromocytomas are usually benign. Pheochromocytomas are generally highly vascularized.

Pheochromocytomas are hyperintense to the myocardium on T2-weighted images and isointense or hyperintense on T1-weighted images

After Gd-DTPA administration, they show strong signal enhancement because of their high vascularity.

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15
Q

Angiosarcoma is the most common malignant cardiac tumor in adults. Where is it most commonly located? (Webb 2654)

A

Right atrium

Webb, 3rd
Angiosarcomas are the most common malignant cardiac tumors in adults and constitute one third of malignant cardiac tumors

This entity has been divided into two clinicopathologic forms.
-Most frequently, angiosarcomas are found in the right atrium. In this form, no evidence of Kaposi sarcoma is found.
-Another form is characterized by involvement of the epicardium or pericardium in the presence of Kaposi sarcoma. This form is associated with the acquired immunodeficiency syndrome.

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16
Q

What is the most common intracardiac mass? (Webb 2665)

A

Thrombus

Thrombus is the most common intracardiac mass, involving most frequently the LV or left atrium. It is most often located in the LV at the site of myocardial infarction or at the apex in dilated cardiomyopathy.

Atrial thrombus is encountered in patients with mitral valve disease or atrial fibrillation.

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17
Q

What is the most common cause of dilated cardiomyopathy? (Brant 4th ed., p. 603)

A

Ischemic cardiomyopathy

Specific causes for dilated cardiomyopathies should be pursued as the specific therapy may vary:
(1) Ischemic cardiomyopathy (the most common cause) because of chronic ischemia, prior infarction, or anomalous coronary arteries
(2) Acute myocarditis (Coxsackie virus most commonly) or long-term sequelae of myocarditis
(3) Toxins (ethanol and doxorubicin [Adriamycin])
(4) Metabolic (mucolipidosis, mucopolysaccharidosis, glycogen storage disease)
(5) Nutritional deficiencies (thiamin and selenium)
(6) Infants of diabetic mothers
(7) Muscular dystrophies

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18
Q

What is the most common type of atrial septal defect? (Webb Thoracic Imaging: Pulmonary and Cardiovascular Radiology Third Edition, Sec. 8 Ch. 31 p. 2379)

A

Secundum

There are four types of atrial septal defects:
1. Secundum (most frequent) - region of the fossa ovalis, which is approximately the middle of the septum
2. Primum - lower part of the septum and bordering on the atrioventricular valve
3. Sinus venosus (superior and inferior vena caval locations) - in either the upper part of the septum and bordering on the ostium of the superior vena cava or the lower septum and bordering on the ostium of the inferior vena cava
4. Coronary sinus (least frequent)

The primum type is usually part of an atrioventricular septal defect (AVSD), which was formerly called endocardial cushion defect

The coexistence of large primum and secundum defects constitutes a common atrium.

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19
Q

Which of the following is the most common cyanotic congenital cardiac anomaly? (Webb Thoracic Imaging: Pulmonary and Cardiovascular Radiology Third Edition, Sec. 8 Ch. 31 p. 2403)

A

Transposition of the great arteries

TGA is the most frequent cyanotic heart lesion. Without surgical intervention, most of the infants would die in the first year of life.

TGA is the most frequent anomaly causing pulmonary overcirculation in a cyanotic infan

Group IV
A lesion is included in this group when the radiograph displays pulmonary arterial overcirculation in the presence of cyanosis. The heart size is usually increased. The observation of increased pulmonary vascularity in a patient with cyanosis is an incongruous finding and should alert the observer to the presence of an admixture lesion rather than a strictly left-to-right shunt. An aid to remembering the major diagnoses in this category is the letter T. The most common diagnosis in this category is TGA, which is the most frequent cyanotic congenital heart lesion at birth

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20
Q

Which of the following congenital heart defects is most commonly associated with right aortic arch? (Webb Thoracic Imaging: Pulmonary and Cardiovascular Radiology Third Edition, Sec. 8 Ch. 31 p. 2417)

A

Tetralogy of Fallot

Swischuck, p305
Right aortic arch with right descending aorta and mirror image branching is one of the few aortic arch anomalies frequently associated with congenital heart disease. Tetralogy of Fallot is most common.

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21
Q

In terms of hemodynamics, what is the most critical component of the anomaly below?

A

Pulmonary stenosis

Swischuck, p268
Tetralogy of FaIlot is the most common cause of cyanotic congenital heart disease beyond the first 30 days of life.

Hemodynamically, the critical component of tetralogy of Fallot is pulmonary stenosis

Webb, 3rd
The major components of this anomaly are caused by a displacement of the outlet septum (conal septation) toward the right ventricle, resulting in a diminutive right ventricular outflow region and failure of alignment of the outlet portion with the remainder of the ventricular septum. The latter abnormality causes a large VSD (infracristal), and the aorta is located immediately over the defect

The extreme form of tetralogy is pulmonary atresia with a nonrestrictive VSD.

Early in life, the thoracic radiograph may not be typical, but it becomes characteristic later. Regression of the thymus reveals the concave main pulmonary artery segment, which is characteristic for tetralogy of Fallot.

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22
Q

T/F: High-velocity jet flow produces bright signal within the dark signal of the blood pool on GRE cine (Webb Thoracic Imaging, 3rd ed, p2438)

A

False

High-velocity jet flow, such as occurs with the flow across a valvular stenosis or the retrograde flow across a regurgitant orifice, produces a SIGNAL VOID (low-signal region within the bright signal of the blood pool) on GRE cine.

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22
Q

Part of Williams’ syndrome, which is characterized by hypercalcemia, elfin facies, variable mental retardation, and characteristic “cocktail party” personality (Webb Thoracic Imaging, 3rd ed, p2436)

A

Supravalvular aortic stenosis

Supravalvular stenosis nearly always is a congenital anomaly, either as an isolated lesion or as part of Williams’ syndrome.

The supravalvular narrowing has roughly three configurations: focal constriction at the sinotubular junction (hourglass configuration) with poststenotic dilation of the mid and distal ascending aorta, focal membrane at the sinotubular junction, and tubular narrowing from the sinotubular junction to just below the origin of the innominate artery.

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22
Q

It is indicated by the passage of the tips of the mitral leaflets beyond the annulus and into the left atrium during systole (Webb Thoracic Imaging, 3rd ed, p2464)

A

Mitral valve flail

Prolapse is ballooning of the middle of the valve beyond the annulus during systole; the tips of the leaflets to which the chordae attach do not pass beyond the annulus.

Flail is indicated by passage of the tips of the mitral leaflets beyond the annulus and into the left atrium during systole.

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23
It is usually acquired and nearly always is caused by rheumatic fever. It is the salient lesion of rheumatic heart disease. (Webb Thoracic Imaging, 3rd ed, p2460)
Mitral stenosis ## Footnote Mitral stenosis usually is acquired and nearly always is caused by rheumatic fever. It is the salient lesion of rheumatic heart disease. Other etiologies are rare; these include congenital valvular, subvalvular (parachute mitral valve), or supravalvular stenoses; left atrial myxoma; and exuberant mitral annular calcification. Mitral stenosis often is accompanied by a variable degree of mitral regurgitation.
24
Valvular stenosis (Webb Thoracic Imaging, 3rd ed, p2435)
It exerts a pressure overload, which involves compensatory myocardial hypertrophy. ## Footnote Valvular stenosis exerts a pressure overload, which involves the compensatory mechanism of myocardial hypertrophy. Regurgitation exerts a volume overload, which involves chamber dilatation.
25
Severe aortic stenosis is defined as an aortic valve area less than (Webb Thoracic Imaging, 3rd ed, p2446)
1.0 cm2 ## Footnote Severe AS is defined as an aortic valve area less than 1.0 cm2 (less than 0.60 cm2/m2). For this measurement, the area of flow through plane at the level of the origin of the flow jet is used
26
In patients with aortic stenosis, there is usually post-stenotic enlargement of the proximal ascending aorta which is greater than (Webb Thoracic Imaging, 3rd ed, p2451)
4.0 cm ## Footnote In patients with aortic stenosis, there is usually poststenotic enlargement of the proximal ascending aorta (greater than 4.0 cm or 2.2 cm/m2)
27
The following inherited diseases are associated with aortic regurgitation, except (Webb Thoracic Imaging, 3rd ed, p2453) a. Marfan syndrome b. Ehlers-Danlos syndrome c. Leys-Dietz syndrome d. Osteogenesis imperfecta e. None of the above
e. None of the above
28
# [](http://) T/F: In the normal subject, RV stroke volume is nearly equivalent to LV stroke volume (Webb Thoracic Imaging, 3rd ed, p2456)
True
29
The following are MR features of mitral stenosis except (Webb Thoracic Imaging, 3rd ed, p2461) a. Diastolic signal void emanating from mitral valve b. Thickened and limited excursion of leaflet c. Left ventricular enlargement d. Normal LV end-diastolic and end-systolic volumes e. High velocity flow on VEC
c. Left ventricular enlargement ## Footnote Left ATRIAL enlargement
30
In the severe form of this disease, the cardiac silhouette extends from one lateral chest wall to the other or “wall to wall” (Webb Thoracic Imaging, 3rd ed, p2472)
Tricuspid regurgitation ## Footnote Tricuspid regurgitation produces cardiomegaly with right atrial and RV enlargement. The most remarkable degree of cardiomegaly may occur with chronic severe tricuspid regurgitation. A cardiac silhouette extending from one lateral chest wall to the other (“wall to wall”) usually is due to severe tricuspid regurgitation. This lesion does not produce pulmonary venous hypertension or edema. In severe cases, the pulmonary vascularity may appear attenuated.
31
The following are true of asymmetric septal hypertrophic cardiomyopathy: a. Hallmark is dynamic subvalvular aortic stenosis b. During diastole, the LV outflow tract appears normal or slightly narrowed because of the presence of upper septal hypertrophy c. None of the above d. All of the above
d. AOTA ## Footnote Webb, p2486 Asymmetric septal hypertrophic cardiomyopathy can cause obstruction of the LVOT. The hallmark is dynamic subvalvular aortic stenosis. During diastole, the LVOT appears normal or slightly narrowed because of the presence of upper septal hypertrophy
32
True about pulmonary venous hypertension (PVH) a. Ossific nodules appear in the lungs only after multiple episodes of edema and chronic PVH. b. The pulmonary venous pressure in acute disease is approximately 5 mm Hg greater for each grade of PVH compared to that in chronic disease. c. There is no correlation between left atrial pressure and radiographic signs of pulmonary edema because of phase lag between rapid pressure changes and slower changes in radiographic alterations. d. All of the above
a. Ossific nodules appear in the lungs only after multiple episodes of edema and chronic PVH. ## Footnote Webb, p2295 Ossific nodules are small foci of bony metaplasia that appear in the lungs only after multiple episodes of edema and chronic PVH. The venous pressure in CHRONIC disease is approximately 5 mm Hg greater for each grade of PVH compared to that in acute disease. Left atrial mean pressure is usually inferred from the mean pulmonary wedge pressure. Correlation between left atrial pressure and radiographic signs of pulmonary edema is only fair because of phase lag between rapid pressure changes and slower changes in radiographic alterations.
32
# [](http://) Which of the following salient radiographic features of myocardial infarction is true? a. Normal chest x-ray in about 70% of first acute infarctions b. Cardiomegaly is usually indicative of Restrictive Cardiomyopathy c. Normal heart size with pulmonary venous hypertension or pulmonary edema in about 50% of first acute infarctions d. All of the above
c. Normal heart size with pulmonary venous hypertension or pulmonary edema in about 50% of first acute infarctions
33
True about radiographic features of Aortic stenosis except: a. Enlargement of the ascending aorta due to post-stenotic dilatation b. Mild or no cardiomegaly in compensated stage c. Pulmonary venous hypertension or pulmonary edema d. Calcification of aortic valve may be discernible on radiograph but is more readily shown on CT
c. Pulmonary venous hypertension of pulmonary edema
34
Double density sign
LA enlargement
35
Grade II sign of pulmonary ventricular hypertension: a. Perihilar alveolar filling b. Subpleural edema c. Large upper lobe vessels d. Confluent acinar shadows
b. Subpleural edema ## Footnote a. Perihilar alveolar filling - Grade III c. Large upper lobe vessels - Grade I d. Confluent acinar shadows - Grade III
35
One of the following x-ray findings is not a radiographic sign of Left Atrial enlargement: a. Left retro-cardiac double density b. Splaying of the carina c. Posterior displacement of the left upper lobe bronchus d. Horizontal orientation of the distal portion of the left bronchus
a. Left retro-cardiac double density ## Footnote Should be RIGHT retrocardiac double density
36
Hoffman-rigler sign
LV enlargement
36
# [](http://) Walking man sign
LA enlargement
37
A semiquantitative estimation of pericardial effusion can be obtained by measuring the width of the pericardial space in front of the right ventricle. A moderate effusion is associated with a width of more than _ mm. (Webb & Higgins, Thoracic Imaging 3rd ed., p 2564)
5 mm
37
Shmoo sign
LV enlargement ## Footnote Brant, 4th ed, p575 Left ventricular or “Shmoo” configuration (after Al Capp’s Shmoo) describes lengthening and rounding of the left heart border with a downward extension of the apex resulting from left ven- tricular enlargement.
37
Findings which can differentiate hemorrhagic pericardial effusion from nonhemorrhagic effusion except: (Webb & Higgins, Thoracic Imaging 3rd ed., p 2564) a. Homogenenous, high signal on T2W SE b. Higher than 40-45 density units on CT c. If chronic, low signal on all types of MR sequences d. High intensity on T1W SE
a. Homogenenous, high signal on T2W SE
38
Which of the following is not included in the diagnostic features of acute pericarditis? (Webb & Higgins, Thoracic Imaging 3rd ed., p 2570) a. Early contrast enhancement of pericardium b. Pericardial thickness of > 2 mm on CT and MRI c. Pericardial effusion d. All may be seen in acute pericarditis
b. Pericardial thickness of > 2 mm on CT and MRI ## Footnote > 4 mm
39
True or False: The classic picture of constrictive pericarditis is classified as chronic fibrous constrictive pericarditis and consists of fibrotic changes and must have associated calcifications consequent to a previous chronic inflammatory process. (Webb & Higgins, Thoracic Imaging 3rd ed., p 2571)
False ## Footnote The classic picture is classified as chronic fibrous constrictive pericarditis and consists of fibrotic changes WITH OR WITHOUT associated calcifications consequent to a previous chronic inflammatory process
40
A 30-year old male underwent chest x-ray for pre-employment. On x-ray you noted leftward deviation of the cardiac silhouette with normal lung parenchyma. What will be your impression on your report? (Webb & Higgins, Thoracic Imaging 3rd ed., p 2571) a. Cardiac findings, as detailed above. Please correlate clinically. b. Consider absent pericardium. CT correlation may be done for further evaluation. c. (Descriptive) d. No significant chest findings.
b. Consider absent pericardium. CT correlation may be done for further evaluation. ## Footnote The most reliable signs of complete absence of the left pericardium are interposition of lung between the aorta and main pulmonary artery, in the aortopulmonary window, and a rotation of the cardiac axis to the left side
41
You suggested a chest CT with contrast for the patient below. The patient had no history of cardiac surgery. You noted a narrow communication of the mass with the pericardial cavity, what will be your diagnosis? (Webb & Higgins, Thoracic Imaging 3rd ed., p 2596)
Pericardial diverticulum
42
# [](http://) True of pericardial effusion: (Brant and Helms, Fundamentals of Diagnostic Radiology 4th ed., p. 580) a. Water bottle appearance on lateral view b. Negative for differential density sign PA c. Differential density sign on AP d. Positive epicardial fat pad sign on lateral view
d. Positive epicardial fat pad sign on lateral view ## Footnote Webb, p2341 The varying density sign is also sometimes present on the frontal radiograph (see Fig. 30.40). This consists of a lesser density at the periphery of the cardiac contour compared to the central portion of the cardiac contour. The cause of this varying density is that the x-ray beam encounters only fluid toward the periphery of the pericardial effusion, while in the center of the pericardial effusion, the radiographic beam must pass through both water anteriorly and the cardiac substance more centrally. Brant, 4th Pericardial Effusion. Between the visceral and parietal lay- ers is the pericardial space, which usually contains 20 mL of serous fluid. More than 50 mL of fluid is clearly abnormal, but 200 mL is required for detection by plain film radiography. Mediastinal and epicardial fat enable the pericardium to be visualized as a thin arcuate line paralleling the anterior heart border in the retrosternal region. A pericardial stripe exceed- ing 2 to 3 mm is indicative of pericardial thickening or effu- sion. Unfortunately, the thickened pericardial stripe can be seen on the lateral radiograph in only about 15% of patients with pericardial effusion. The “differential density sign” refers to a lucent margin along the left heart border on the PA radio- graph or along the posterior cardiac border on the lateral radiograph. It is seen in up to 63% of patients with pericar- dial effusion but is less specific than the thickened pericardial stripe. Large pericardial effusions cause the heart to appear on frontal radiographs in the shape of a sac of water sitting on a tabletop (Fig. 21.21).
43
True of cardiac tamponade: (Brant and Helms, Fundamentals of Diagnostic Radiology 4th ed., p. 580) a. Rapid enlargement of cardiac silhouette with normal vascularity b. Diastolic collapse of the LV on 2D echo c. Appreciated radiographically with at least 50 mL of pericardial fluid d. Paradoxical narrowing of the SVC and IVC
a. Rapid enlargement of cardiac silhouette with normal vascularity
44
# [](http://)[](http://) The pericardial space normally contains ____ ml of fluid. (Webb & Higgins, Thoracic Imaging 3rd ed., p 2551)
up to 50 mL ## Footnote The pericardial cavity normally contains up to 50 mL of serous fluid that distributes diffusely around the heart surface and inside the pericardial recesses.
45
Which of the following is false regarding atrial septal defects? (Swischuk p. 243) a. The secundum defect results from the absence of, or a defect in, the flap of tissue derived from the septum primum b. Ostium primum defects are low defects resulting from maldevelopment of the primitive endocardial cushions. c. The secundum defect results from the absence of, or a defect in, the flap of tissue derived from the septum secundum d. These defects cause left-to-right shunting of blood
Answer in the answer key: a. The secundum defect results from the absence of, or a defect in, the flap of tissue derived from the septum primum ## Footnote Choice A is true Choice C is false
46
In cases of double aortic arch, what is seen on barium esophagram? (Swischuk p. 308) a. Reverse S-shaped indentation of the esophagus b. Indentation of the anterior border of the esophagus c. Indentation of the posterior border of the esophagus d. None of the above
a. Reverse S-shaped indentation of the esophagus
46
# [](http://) In cases of aberrant left pulmonary artery, what is seen on barium esophagram? (Swischuk p. 315) a. Indentation of the posterior border of the esophagus b. Indentation of the anterior border of the esophagus and posterior border of the trachea c. Reverse S-shaped indentation of the esophagus d. None of the above
b. Indentation of the anterior border of the esophagus and posterior border of the trachea
47
Which of the following is not an associated finding of pulmonary embolism? (Swischuk p. 332) a. Cardiomegaly with right ventricular hypertrophy b. Prominence of the pulmonary artery c. Diminution of the pulmonary vasculature d. None of the above
d. NOTA ## Footnote Swischuk, p332 Roentgenographically, there will be cardiomegaly with right ventricular hypertrophy, prominence of the pulmonary artery, and diminution of the pulmonary vasculature. Pulmonary emboli also can calcify lain films, when embolism is massive, tend to show decreased pulmonary vascularity and cardiomegaly with a right-side enlargement pattern. Most often, however, with pulmonary embolism, plain films are normal.
48
Identify the MISMATCHED sequence (Webb 2622) a. Cine MRI: movement b. Spin echo: white blood c. Gradient echo: white blood d. Steady-state free precession (SSFP): white blood
b. Spin echo: white blood
49
# [](http://)[](http://) Which of the following characteristics are suggestive of a MALIGNANT cardiac tumor? (Webb 2654) a. Combined intramural and intracavitary components b. Simple pericardial effusion c. Narrow attachment to the cardiac wall d. None of the above
a. Combined intramural and intracavitary components
50
Which of the following are suggestive of a BENIGN cardiac tumor? (Webb 2654) a. Mediastinal extension b. Pulmonary metastasis c. Narrow attachment to the cardiac wall d. Tumor necrosis
c. Narrow attachment to the cardiac wall
51
True or false: Rhabdomyosarcomas typically appear as single tumors. (Webb 2657)
False ## Footnote Rhabdomyosarcomas are the most common malignant cardiac tumors in children. They can arise anywhere in the myocardium and are often multiple. Their signal intensity on MRI is variable. Rhabdomyosarcomas may be isointense to the myocardium on T1- and T2-weighted images, but areas of necrosis can exhibit heterogeneous signal intensity and patchy hyperenhancement after Gd-DTPA administration (see Fig. 35.22). Extracardiac extension into the pulmonary arteries and descending aorta has been clearly delineated with MRI.
52
True or false: On imaging alone, it IS possible to differentiate among PRIMARY CARDIAC fibrosarcoma, osteosarcoma, leiomyosarcoma, or liposarcoma. (Webb 2657)
False
53
True or false: Primary cardiac lymphoma is more common than secondary lymphoma involving the heart.
False ## Footnote Primary cardiac lymphoma is less common than secondary lymphoma involving the heart, which usually represents the spread of non-Hodgkin’s lymphoma. Primary lymphoma of the heart most often occurs in immunocompromised patients and is highly aggressive. Almost all primary cardiac lymphomas are B-cell lymphomas.
54
Which of the following cardiac MR sequences is acquired for the purpose of quantifying ventricular volumes, mass, and global function? (Webb 3rd ed., p. 2478)
Cine MRI with FIESTA ## Footnote Cine MR images are acquired for the purpose of quantifying ventricular volumes, mass, and global function. Cine MR images are now usually done with some form of balanced steady-state free precession sequence. A set of these images at multiple levels encompassing the heart provides a volumetric data set for the direct measurement of the end-diastolic, end- systolic, and stroke volumes, mass, and ejection fraction of both the left ventricle and right ventricles. The blood pool is bright on cine MRI.
55
All of the following are imaging features of myocarditis, EXCEPT? (Webb 3rd ed., p. 2526) a. Increased LV +/- RV volumes b. Decreased LV ejection fraction c. Focal early GAD hyperenhancement in the LV subepicardium d. Global LV hypokinesis
c. Focal early GAD hyperenhancement in the LV subepicardium
56
Which of the following imaging features will differentiate restrictive cardiomyopathy from constrictive pericarditis? (Webb 3rd ed., p. 2505) a. Normal pericardial thickness b. Non-enhancing myocardium c. Decreased LV volume and ejection fraction d. Increased LV wall thickness
a. Normal pericardial thickness ## Footnote In constrictive pericarditis, the pericardium is nearly always thickened, whereas restrictive cardiomyopathy does not have this feature.
57
Which type of cardiomyopathy typically presents with a normal ventricular wall thickness? (Brant 4th ed., p. 602) a. Hypetrophic b. Dilated c. Restrictive d. All of the above e. None of the above
c. Restrictive
58
Which of the following types of cardiomyopathies may present with decreased cardiac compliance? (Brant 4th ed., p. 602) a. Hypetrophic b. Dilated c. Restrictive d. All of the above e. None of the above
d. All of the above
59
This rare form of cardiomyopathy is limited to the RV, with dilation of the RV chamber, marked thinning of the anterior right ventricular wall and abnormal RV wall motion: (Brant 4th ed., p. 605)
“Arrhythmogenic right ventricular dysplasia” (ARVD)
60
Which of the following is characterized by loose networks of trabeculations in the endocardial wall of the left ventricle, LV enlargement and decreased ejection fraction? (Webb 3rd ed., p. 2534) a. Cardiac sarcoidosis b. Cardiac amyloidosis c. Cardiac Noncompaction d. Dilated cardiomyopathy
c. Cardiac Noncompaction
60
Apical aneurysm of the left ventricle is a characteristic chronic finding in: (Webb 3rd ed., p. 2532)
Chagas disease ## Footnote hagas’ disease is caused by the protozoan Trypanosoma cruzi. Cardiac involvement is frequent. This infection is characterized by acute, intermediate, and chronic phases. In chronic Chagas’ disease, the heart is the most frequently affected organ, and lymphocytic infiltration can be observed. Regions of focal inflammation have been shown to enhance strongly on T1-weighted images after the administration of Gd-DTPA and on DE-MR. The regional distribution of LGE is typically inferolateral basal and apical. Apical aneurysm of the left ventricle is a characteristic of chronic Chagas disease. LGE is seen more frequently in more advanced diseases and worse clinical statuses.
61
Regional dysfunction, with no delayed hyperenhancement and acute transient decrease in myocardial perfusion are indicative of which type of myocardial injury? (Webb 3rd ed., 2769)
Stunned myocardium ## Footnote Stunned myocardium shows regional myocardial dysfunction, which recovers to normal in hours to days; there is no irreversible injury (acute infarctions). Hibernating myocardium is chronic ischemia that causes persistent myocardial dysfunction but is not so severe that infarctions occur. Reduction in myocardial blood flow below a critical threshold for a sufficiently long time causes myocardial infarction.
62
# [](http://) All of the following are delayed enhancement patterns characteristic of myocardial infarction, EXCEPT? (Webb 3rd ed., 2771) a. Always involves at least the subendocardial layer of the myocardium b. Corresponds to the distribution of one or more coronary arteries or branch arteries c. Both d. None of the above
d. None of the above ## Footnote The delayed enhancement caused by acute or chronic myocardial infarction has two features that are characteristic: (1) always involves at least the subendocardial layer of myocardium and (2) corresponds to the distribution of one or more coronary arteries or branch arteries
63
It is defined as right ventricular failure secondary to pulmonary parenchymal or pulmonary arterial disease: (Brant 4th ed., p 604)
Cor pulmonale
64
An acyanotic patient presents with evidence of increased pulmonary vascularity, left atrial enlargement, biventricular enlargement. What is your primary consideration? (Webb Thoracic Imaging: Pulmonary and Cardiovascular Radiology Third Edition, Sec. 8 Ch. 31 p. 2389)
Ventricular septal defect
65
What congenital heart defect shows pulmonary plethora, right atrial and ventricular enlargement, and a diminutive ascending aorta and aortic arch? (Webb Thoracic Imaging: Pulmonary and Cardiovascular Radiology Third Edition, Sec. 8 Ch. 31 p. 2380)
Atrial septal defect
66
The NICU intern messaged Radio Telegram to request for a wet read of the chest radiograph of a preterm infant. You noted evidence of pulmonary arterial overcirculation, left atrial and ventricular enlargement, and an enlarged aortic arch. You suspect a significant left-to-right shunt. What is your primary diagnosis? (Webb Thoracic Imaging: Pulmonary and Cardiovascular Radiology Third Edition, Sec. 8 Ch. 31 p. 2392)
Patent ductus arteriosus
67
A cyanotic patient presents with decreased pulmonary vascularity, right ventricular enlargement, a concave pulmonary arterial segment, and a prominent ascending aorta and aortic arch. Because you’ve already seen several similar studies you quickly come up with a primary diagnosis, noting that it can also present with asymmetric blood flow towards which structure? (Webb Thoracic Imaging: Pulmonary and Cardiovascular Radiology Third Edition, Sec. 8 Ch. 31 p. 2397)
Right lung ## Footnote Branch pulmonary arterial stenosis, especially at the origin of the left pulmonary artery, may cause asymmetric pulmonary blood flow. However, even in the absence of branch stenosis, preferential flow occurs to the right lung due to the orientation of the right ventricular outflow tract.
68
Based on the Collett and Edward Classification, what type of truncus arteriosus is shown when the right and left pulmonary arteries arise separately from the posterior aspect of the truncus? (Webb Thoracic Imaging: Pulmonary and Cardiovascular Radiology Third Edition, Sec. 8 Ch. 31 p. 2415)
Type II
69
The snowman sign on chest radiographs is commonly attributed to total anomalous pulmonary venous return. Which specific type do we expect to see this? (Webb Thoracic Imaging: Pulmonary and Cardiovascular Radiology Third Edition, Sec. 8 Ch. 31 p. 2417)
Supracardiac ## Footnote Swischuck, p251 Roentgenographically, the changes in total anomalous pulmonary venous retUrn are varied and depend on the specific type ofvenous connection. The most characteristic and diagnostic plain film configuration in the type I anomaly group is that of the so-called "snowman" or "figure 8" heart.
70
This cardiac deformity presents with displacement of one or more triscuspid valve leaflets into the inflow portion of the right ventricle. Notable findings include decreased pulmonary vascularity, enlargement of the right atrium and ventricle, a small main pulmonary arterial and hilar segments, and a small thoracic aorta. (Webb Thoracic Imaging: Pulmonary and Cardiovascular Radiology Third Edition, Sec. 8 Ch. 31 p. 2420)
Ebstein anomaly
71
Which of the following is true in patients with corrected transposition of the great arteries (L-TGA)? (Webb Thoracic Imaging: Pulmonary and Cardiovascular Radiology Third Edition, Sec. 8 Ch. 31 p. 2404-5) a. Blood from the pulmonary veins drains into the left atrium, to the right ventricle, and into the aorta b. The morphologic right ventricle is located to the left of the morphologic left ventricle c. There is a prominent convexity of the upper left cardiac border on radiographs d. All of the above
d. AOTA
72
Diagnosis
VENTRICULAR SEPTAL DEFECT
73
A: Which type of partial anomalous pulmonary venous return would this radiograph be typically attributed? B: What is your diagnosis? (Separate answers for A and B with a comma)
INFRACARDIAC, SCIMITAR SYNDROME INFRACARDIAC TYPE, SCIMITAR SYNDROME
74
Diagnosis? Specify specific phenotype/classification
ASYMMETRIC HYPERTROPHIC CARDIOMYOPATH ASYMMETRIC SEPTAL HYPERTROPHIC CARDIOMYOPATHY HYPERTROPHIC CARDIOMYOPATHY
75
Representative cut of a delayed T1W-contrast enhanced cardiac MR study, short axis view. Diagnosis?
MYOCARDIAL INFARCTION
76
In terms of hemodynamics, what is the most critical component of the anomaly below?
PULMONARY STENOSIS
77
Diagnosis?
MYOCARDIAL BRIDGING BRIDGING OF THE LEFT ANTERIOR DESCENDING ARTERY BRIDGING OF THE LAD
78
Gradient echo images. Pointed mass has NO early enhancement on post-contrast sequences. Impression?
THROMBUS INTRACARDIAC THROMBUS
79
Impression, ECG-gated T1 spin-echo images (left); same image with fat saturation (right); (Webb 2622
LIPOMA
80
Identify the location of mass; Intracavitary, intramural, pericardial, paracardiac (Webb 2623)
INTRAMURAL
81
Name the vascular anomaly associated with these imaging findings
ABERRANT LEFT PULMONARY ARTERY
82
Name the vascular anomaly associated with these imaging findings
DOUBLE AORTIC ARCH
83
Diagnosis. (Pointed by the arrows)
PERICARDIAL EFFUSION
84
What do you call this sign? (Same image as previous)
OREO COOKIE SIGN
85
Diagnosis.
CONSTRICTIVE PERICARDITIS
86
Diagnosis
AORTIC REGURGITATION
87
Diagnosis
MITRAL STENOSIS
88
Diagnosis
TRICUSPID REGURGITATION
89
Complication of myocardial infarction
TRUE ANEURYSM LEFT VENTRICLE TRUE LEFT VENTRICULAR ANEURYSM TRUE ANEURYSM OF THE LEFT VENTRICLE
90
What disease entity is this? (arrows)
PULMONARY ARTERIAL HYPERTENSION
91
Identify lesion along the right side of the heart.
PERICARDIAL CYST