2020 AHA ACLS P2 Flashcards

1
Q

To function effectively a high performance team needs to focus on ____x4?

A
  • Timing
  • Quality
  • Coordination
  • Administration
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2
Q

What are some ways to increase Chest Compression Fraction (CCF)?

A
  • Precharge the defibrillator
  • Perform a pulse check during the precharge phase
  • Have compressor hover over the chest during shock in order to immediately get back to compression
  • Have the next compressor ready to take over immediately
  • Intubate and deliver meds during compressions
  • use cpr and vents continuously together
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3
Q

Describe the role of the Team Leader

A

The Team Leader should focus on comprehensive pt care, not just one role. They must ensure everything is being done at the right time and the right way.
The Team Leader:
- organizes the group
- monitors individual performance
- backs up team members
- models excellent team behavior
- trains and coaches (even future team leaders)
- Facilitates understanding
- Focuses on comprehensive pt care
- Temporarily designates another team member to take over at Team Leader if an advanced procedure is required.

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4
Q

Describe the Role of a Team Member

A

A Team Member:

  • Proficient in performing the skills in their scope
  • Clear about role assignments
  • Prepare to fulfill their role
  • Well practiced in resuscitation skills
  • Knowledgeable about the algorithms
  • committed to success
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5
Q

Describe the Role of the CPR Coach

A

The CPR Coach’s main responsibility is to help members provide high quality CPR and minimize pauses in compressions. They can deliver feedback about:

  • Compressors rate, depth, recoil
  • Rate and Volume of Ventilations
  • Compression Pauses

They should have clear line of site to the compressor and should therefore stand by the defibrillator. It is because of this that often the CPR Coach is also in charge of the Monitor/Defibrillator; they don’t have to only be the CPR Coach.

An overview of their job is:

  • Coordinate the start of CPR
  • Coach to improve the quality of chest comps
  • State midrange targets (say 110 comp/min instead of 100-120)
  • Coach to the midrange targets
  • Help minimize the length of pauses in compressions
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6
Q

What are the elements of Effective Team Dynamics as part of a High-Performance Team (8x)

A
  • Clear Roles and Responsibilities
  • Knowing your Limitations
  • Constructive Interventions
  • Knowledge Sharing
  • Summarizing and Reevaluating
  • Closed Loop Communications
    (1. give order 2. get eye contact and response that they understand 3. confirm that order is completed before giving that member another)
  • Clear Messages (don’t yell, speak calm and clear)
  • Mutual Respect
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7
Q

What are the 6 possible roles when enough people are present?

A
  • Team Leader
  • Monitor/Defibrillator/CPR Coach
  • Compressor
  • Airway
  • IV/IO/Medications
  • Timer/Recorder
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8
Q

Tidal Volume and Hypoventilation according to AHA

A

Tidal volume is 6-8mL/kg for normal oxygenation (usually just go by 500mL outside of AHA)

Hypoventilation is anything below 6 breaths/min and requires a bag-mask device or advanced airway with O2

Tachypnea and bradypnea are still considered anything above or below 12-20

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9
Q

Respiratory Distress (mild to severe) VS Respiratory Failure VS Respiratory Arrest

A

Resp DISTRESS is when there is an abnormal rate or effort present at rest. Usually if it is only increased rate or effort alone it is mild but when you have signs of tachycardia, skin changes, significant rate or effort increase, changes in mental status ect, it is considered severe respiratory distress.

Resp FAILURE is when the pt is in a state of inadequate oxygenation, ventilation, or both. Usually this is once the pt is so exhausted they are no longer trying to vent as much

Resp ARREST is the absence of breathing
This is when you should deliver 500-600mL of ventilation via bag valve mask. Sometimes a higher volume is needed so be sure you can bypass or block off the pressure relieve valve on a BVM. Remember that an adult BVM has way more than 500mL so you do not want to squeeze the whole bag!

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10
Q

What are the dangers of Hyperventilation, either too much volume or too many breaths/min?

A
  • It can cause gastric inflation and lead to regurgitation and aspiration
  • It increases intrathoracic pressure
  • decreases venous return to the heart
  • diminishes cardiac output and survival
  • may cause cerebral vasoconstriction, reducing blood flow to the brain
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11
Q

How does AHA want you to deliver breaths in a respiratory arrest WITH a pulse?

A

1 breath every 6 seconds, OR 10 breaths/min with a bag-valve mask or any advanced airway.

Deliver breath over 1 second and watch for chest rise and fall

Check pulse every 2 minutes only taking about 5-10 seconds to asses. If no pulse then start CPR

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12
Q

What are your O2 sat goals for ACS, Respiratory Arrest/Acute Cardiac Symptoms, stroke, and Post-Cardiac Arrest care?

A

ACS = at least 90%
Acute Cardiac Symptoms/Resp Arrest = at least 95%
Stroke = 95-98%
Post Cardiac Arrest Care = 92-98%

(AHA says normal is 95-98%)

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13
Q

When someone is choking and you can’t remove the object via the Heimlich maneuver and you cant see it to remove with your fingers, what should you do as they begin to go into respiratory arrest and unconsciousness?

A

START CPR, check for obstruction visibility in the mouth every time you give ventilations

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14
Q

Measuring and sizing OPAs and NPAs

A

OPA:
- After clearing mouth of secretions, pick an OPA that is from corner of the mouth to the angle of the mandible. They do not care if you go 180 or 90 degree turn

NPA:
- Use width of pinky finger as guide to size, but you should not see blanching of the nostril upon insertion.
Length is judged by tip of pts nose to the earlobe. If you experience resistance upon insertion try either twisting is or using other nostril. Always lubricate before insertion to ease the passage.

(Picture shows bevel facing lateral aka NOT medial)

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15
Q

Wall mounted suction units should be able to provide airflow of more than _____ and suction of more than _____ when clamped at full suction?

A

Wall mounted suction units should be able to provide airflow of more than 40 L/min and suction of more than -300mmHg when clamped at full suction?

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16
Q

When to use Rigid VS Flexible Catheters

A

FLEXIBLE:

  • in mouth or nose
  • for ET suctioning
  • For aspiration of thin secretions from the oro or nasopharynx
  • to perform intratracheal suctioning
  • to suction through an in place airway to access the back of the pharynx in a pt with clenched teeth

RIGID:

  • for more effective suctioning of the oropharynx
  • for thick secretions or particulate matter
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17
Q

Suctioning procedure list

A
  • measure catheter before suctioning
  • place catheter into oropharynx past tongue, DO NOT inset beyond the distance from the tip of nose to earlobe
  • Apply suction with a rotating or twisting motion
  • Limit each suction attempt to 10 seconds or less
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18
Q

What is the ideal minimum of CCF percentage?

A

Ideally Chest Compression Fraction should be at Least 80%

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19
Q

Is agonal gasping a sign of breathing?

A

NO, pts in cardiac arrest may have Agonal Gasps for the first few minutes but this is NOT BREATHING it is a sign of cardiac arrest

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20
Q

What is another way to calculate CCF when an automated feedback device is not available?

A

Use 2 timers, one that starts and stops when the code begins and ends (total code time) and another that starts and stops when chest compressions are being done. Then use the formula:

CCF = Actual Chest Comp Time + Total Code Time

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21
Q

Monophasic VS Biphasic Defib Joule recommendations

A

If using a MONOPHASIC defibrillator give a signal 360-J shock and use that same joule for subsequent shocks

If using a BIPHASIC defibrillator use the recommended range via the manufacturer, this is usually displayed on the front of the device. Typically it is between 120-200 J. IF YOU DO NOT KNOW, then give the max energy dose for first and all shocks

If you attain a successful conversion on a certain shock Joules, and the pt later goes back into VF or pVT then use the previously successful Joules to shock again

22
Q

What exactly is the goal of using a defibrillator? Is it to restart the heart?

A

A defibrillator DOES NOT restart the heart. It STUNS the heart, effectively stopping all electrical activity for a moment in the hopes that the heart’s normal pacemakers will have a chance to take back over.

Typically if you have a successful conversion, the spontaneous rhythm will be slow and may not produce a pulse or adequate perfusion (b/c the heart has just taken a serious beating) and therefore CPR should be continued

23
Q

AHA time ranges of “Clinical Death”, “Biological Death”, and “Irreversible Brain Damage”

A

Clinical Death occurs in 4-6min after arrest, at this time frame NO damage to the brain has occurred.

Biological Death occurs in 6-10min after arrest, at this time frame Brain damage is LIKELY

Irreversible Brain Damage occurs at the 10+ min mark after arrest ; EXCEPT in cold water drownings or hypothermia emergencies b/c the O2 demand of metabolic process has been severely reduced.

***This is where the phrase “they’re not dead till they’re warm and dead” comes from

24
Q

What does pVT quickly deteriorate into? and what does the second rhythm turn into if left untreated?

A

pVT quickly turns into VF which left untreated will turn into Asystole

25
Q

What is the MOST EFFECTIVE WAY to treat VF?

A

DEFIBRILLATION is the most effective treatment for VF!!
CPR does not lead to ROSC, it is merely priming the heart for a higher chance of a successful defib by giving a small amount of circulation and perfusion to the heart and brain

26
Q

Every minute that passes between collapse and defib the chances of survival drop by _____% ; but if CPR is immediately started it can change that to _____%?

A

Every minute that passes between collapse and defib the chances of survival drop by 7-10% ; but if CPR is immediately started it can change that to 3-4%

This means that by giving immediate CPR, you can increase a pts chances of survival by 2-3 times

27
Q

What is the max time it should take to clear the pt and deliver a shock?

A

It should only take 5 seconds max, and while doing so you should face the pt and not the defibrillator. This ensures that the compressor did not start compressions too soon. ALSO DO NOT LET THEM TOUCH THE STRETCHER!!

28
Q

When should you check a rhythm once you have a 4 or 12 lead established. How long should this take?

Can/should you palpate a pulse while doing this?

A

Check the rhythm after 2 minutes of CPR, but DO NOT exceed 10 seconds in pausing chest compression

Palpate for a pulse during a rhythm check, ONLY if the rhythm appears organized

29
Q

What is the aim in using Vasopressors? What is the main one used in FV and pVT?

A

Vasopressors DO NOT increase survivability from VF or pVT! They DO improve aortic diastolic BP, coronary artery perfusion pressure, and the rate of ROSC.

Epinephrine is the main vasopressor used b/c of its A-adrenergic effects (vasoconstriction). Vasoconstriction increases cerebral and coronary blood flow during CPR by increasing mean arterial pressure and aortic diastolic pressure.

Give 1mg of Epi after the SECOND shock and during CPR, repeated every 3-5 minutes (or 4 minutes)

30
Q

What are the 2 antiarrhythmics that can be considered for VF or pVT either before OR after the shock?

A

AMIODARONE
- 300mg IV/IO bolus initial
150mg for secondary
- Amiodarone is a class III antiarrhythmic but has other class properties. It blocks sodium channels (Class I effect) and exerts noncompetitive antisympathetic action (class II effect). It also has a prolongation of the cardiac action potential (class III effect)

LIDOCAINE
- 1.0-1.5mg/kg IV/IO initial
0.5-0.75 mg/kg at 5-10intervals for secondary+ to max of 3mg.
- It suppresses automaticity of conduction tissue in the heart by increasing the electrical stimulation threshold of the ventricles. His-Purkinjie system, and spontaneous depolarization of the vents during diastole by a direct action on the tissue.
It also blocks permeability of the neuronal membrane to sodium ions, which inhibits depolarization and the blockade of conduction

31
Q

What drug should be considered for Torsades de Pointes?

A

MAGNESIUM SULFATE

  • loading dose of 1-2g IV/IO diluted into 10mL solution given as a bolus over 20 minutes
  • It is a sodium/potassium pump agonist
32
Q

What are 2 extra steps you can take post IV drug admin to help get the drug to the central circulation faster?

A
  • admin 20mL fluid bolus after (flush it)

- hold the extremity up for about 10-20 seconds

33
Q

What are 4 important points about IO access?

A
  • You can achieve it in all age groups
  • You can achieve it in about 30-60 seconds
  • Any drug you can give IV you can give IO
  • It is preferable to ET and may be easier to establish
34
Q

What are 5 important things to keep in mind if giving a drug via ET tube? (Not preffered over IV/IO)

A
  • The optimal dosage for ET route admin is unknown
  • Dosage is usually about 2-2.5 times more than IV/IO
  • You have to stop CPR briefly so that the meds are not regurgitated
  • Drugs like Epi can negatively effect the colorimetric devices detectors

*** When giving via ET, dilute the medication into 5-10mL of NS and inject it directly into the ET tube

35
Q

What is PEA and how is it different than VF and pVT?

A

PEA IS ANY ORGANIZED RHYTHM WITHOUT A PULSE

So any rhythm, even sinus rhythm, that is organized and should have a pulse on the QRS’s (wide or narrow) but does not is PEA.

This is because there is still electrical activity but the heart is mechanically functioning so weak that it is not producing a pulse, aka not pumping. This is why PEA use to be called Electromechanical Dissociation.

So literally anything that does not produce a pulse EXCEPT for VF and pVT is PEA. THIS IS BECUASE VF AND PVT RESPOND BEST TO IMMEDIATE ELECTRICAL THERAPY.

So if there is NO pulse present you should ask yourself “is this a VF or pVT rhythm?” and if not then it is PEA

Asystole is also not considered PEA only because it has it’s own definition of no electrical activity. PEA HAS electrical activity but no pulse like asystole. So PEA requires no pulse but STILL HAS electrical activity

36
Q

Normally in PEA there is electrical activity but the heart is not contracting? What is another way you can have electrical activity AND adequate contractility but still no pulse, qualifying it as PEA?

How can you tell which is happening?

A

You can also get PEA when there is not enough preload to the left ventricle. So the electrical activity and contractility are fine but there is nothing to pump so you technically still have PEA.

This can be caused by things like hypovolemia, PE causing decrease venous return, cardiac tamponade, or tension pneumothorax

To tell if it is a lack of preload and nothing in the L Vent or if it is the heart not contracting that is causing PEA, the focus needs to be on the output of ETCO2 and pulse presence upon CPR

If you are doing CPR and you get a good pulse with good ETCO2 then you probably have fluid in the L Vent and it is the contractility that is failing. Adversely, if you are doing CPR and you do not get a good pulse or ETCO2 reading then you probably don’t have any blood getting to the L Vent

37
Q

You should consider stopping resuscitation efforts if the ETCO2 level is below ____ after ____ minutes of CPR?

A

You should consider stopping resuscitation efforts if the ETCO2 level is below 10 mmHg after 20minutes of CPR?

38
Q

Additional thoughts for Hypothermic Cardiac Pts

A

Hypothermia for AHA is a core body temp below 30degrees C or 86F

Drugs will not metabolize as fast and may rise to toxic levels with normal dosage and timing intervals

Vasopressors are still recommended but Antiarrhythmics are NOT

The aim of ACLS for hypothermic pts is ACTIVE REWARMING

39
Q

What are the new or old versions of an LVAD?

A

Old = Pulsatile flow

New = Continuous flow

40
Q

What are the 2 most common causes of pump failure on a VAD?

A

Disconnection from the power supply or from the driveline. This is why the first thing you do when assessing a pt with a VAD in place is ensure good connections!

An important note to remember with a VAD pt is that they normally have back up batteries, controllers, etc so when you transport a pt to a hospital KEEP THE PT CLOSE TO THEIR BACK UP EQUIPMENT b/c the receiving facility might not have the equipment.

Usually when the equipment is close to failure for whatever reason it will beep! (unless the batteries are completely dead). When functioning properly there should be a “VAD Hum” that you can auscultate to see if it is on/working

Pts that are having a cardiac event that have a VAD are usually suffering from something other than a VAD malfunction but always assess both the machine and the pt

41
Q

At how many weeks into pregnancy does the uterus size begin to adversely affect an attempted resuscitation? How many weeks till the fetus may survive a C-Section, aka a Hysterotomy?

A

At 20 weeks the uterus size begins to affect resuscitation efforts.

At 24-25 weeks they baby should be able to survive a C-Section

42
Q

Why do you want to place a pregnant pt in a Left-lateral Recumbent or Decubitus position?

If you cannot use the Left Lateral Decubitus position b/c you need to do CPR, what are 2 techniques you can use to shift the Gravid Uterus?

A

It takes pressure off of the Vena Cava’s to allow for better venous return

To shift a Gravid Uterus (uterus that is pressing on the Vena Cava’s and maternal vessels) you can:

  • Ideally sit on the left side of the supine pt, in line with the uterus. Reach across and pull the uterus leftward and upward toward your own abdomen with 2 hands
  • If you cannot sit on the left side, use one hand to push it left and upward
43
Q

At what blood pressure is a pregnant pt considered hypotensive and warrants therapy?

A

Less than 100mmHg or less than 80% baseline

44
Q

If there is no sign of ROSC possible for the mother within 5 minutes what should you do for the fetus?

A

Immediately assemble/call upon the Care Team for this situation b/c they may want to do a Perimortem Caesarean delivery

45
Q

How do you calculate MAP?

A

MAP = (diastolic) blood pressure is doubled and added to the (systolic) blood pressure and that composite sum then is divided by 3 to estimate MAP.

((Dia X 2) + Sys) / 3)

46
Q

What is the TTM range goal for post cardiac care?

A

32-36C using a feedback loop device

47
Q

Most death after ROSC occur within ____?

A

The first 24 hours

48
Q

How should you position the pt after ROSC?

A

Elevate the head of the bed 30 degrees if tolerated to reduce the incidence of cerebral edema, aspiration, and ventilatory associated pneumonia

49
Q

What are the major factors that determine survivability after a cardiac arrest? What is the best way to treat them?

A

Brain Injury and Hemodynamic Instability are the major factors that determine survivability after a cardiac arrest.

TTM for at least 24 hours is the best treatment to fight this, ONLY when unconscious after ROSC

TTM for 24 = Improved Neuro Prognosis

50
Q

What are the 2 types of capnography devices?

A

MAINSTREAM:
- measure the CO2 directly on the airway and sends the signal back to the device to display

SIDESTREAM:
- samples gas from the airway and measures the CO2 within the device

51
Q

What are the drugs used for Post Cardiac Care which are subsequently all used for management of hypotension (SBP less than 90mmHg)?

A
  • 1-2L IV Bolus of NS or LR
  • Norepinephrine 0.1-0.5mcg/kg/min
  • Epinephrine 2-10mcg/min (not per kilo)
  • Dopamine 5-20mcg/kg/min
52
Q

What is the leading cause of death in OHCA survivors?

A

Ischemic Brain Injury due to withdrawal of life sustaining treatment on the basis of a predicted poor neurologic outcome