18 – Equine GI 1 & 2 Flashcards

1
Q

General approach to patient

A
  • Signalment
  • History
  • PE
  • Further diagnostic tests (diagnostic plan)
  • Differential diagnoses
  • Diagnosis
  • Treatment plan
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2
Q

What are the 3 ‘parts’ that could cause dysphagia?

A
  • Prehension
  • Mastication
  • deglutition
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3
Q

What are the clinical signs of dysphagia?

A
  • Feed/water exiting nostrils
  • Ptyalism
  • Coughing
  • Weight loss
  • +/- aspiration pneumonia
  • +/- CN deficits
    o Decreased tongue tone
    o Facial nerve paralysis
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4
Q

What are the causes of dysphagia?

A
  • Pain
    o Dental disease
    o Foreign body
    o TMJ osteoarthritis
  • Obstruction
    o DDSP
    o (retro)pharyngeal abscess (strangles)
    o Choke
  • Neurologic disorder
    o Guttural pouch mycosis
    o Botulism
    o EPM (equine protozoal myoencephalitis)
    o Facial nerve trauma
    o THO
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5
Q

How do you diagnose dysphagia?

A
  • History
  • PE
  • Neurologic exam
  • Oral exam
  • Bloodwork
  • Endoscopy
  • Radiographs
  • CT
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6
Q

What are some examples of esophageal disorders?

A
  • *Choke
  • Esophageal stricture
  • Esophageal diverticula
  • Esophagitis
  • Esophageal ulcers
  • Esophageal rupture
  • Megaesophagus
  • *many of these can predispose them to choke or a secondary to choke
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7
Q

What are the clinical signs of choke?

A
  • Dysphagia
  • Coughing
  • Inappetence
  • Ptyalism
  • Colic signs
  • Anxiety
    o Tachycardia
    o Tachypnea
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8
Q

How do you diagnose choke?

A
  • Signalment
    o Friesian: megaesophagus (predisposing to choke)
  • History
    o Feed: unsoaked beet pulp
    o Previous choke
  • PE
  • *nasogastric intubation
  • Oral exam
  • Endoscopy
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9
Q

What are examples of gastric disorders?

A
  • *Gastric ulcers
  • Gastric impactions
  • Gastric rupture
  • Neoplasia
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10
Q

What are the clinical signs of gastric ulcers?

A
  • *non-specific
  • Mild acute colic
  • Recurring colic
  • Poor body conditions
  • Parital anorexia
  • Poor performance
  • Attitude changes
  • Frequent stretching to urinate
  • “girthy”; behaviour change
    • NO bleeding! (NO anemia), no melena
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11
Q

How do you diagnosis gastric ulcers?

A
  • Gastroscopy
    o Need to be fasted for 18 hours
    o **equine squamous gastric ulcer syndrome=most common (along margo plicatus)
    o Equine glandular gastric ulcer syndrome (usually localized in the pylorus)
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12
Q

Colic signs

A
  • *manifestation of visceral abdominal pain
  • Frequently getting up and down
  • Pawing
  • Rolling
  • Flank watching
  • Posturing to urinate
  • Downward dog
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13
Q

How do you diagnose colic?

A
  • Signalment
  • History
  • PE
  • Nasogastric intubation
  • Rectal palpation
  • Abdominal ultrasound
  • Abdominocentesis
  • Blood work
  • Fecal examination
  • (radiology)
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14
Q

What are the DDx for colic?

A
  • Laminitis
  • Rhabdomyolysis
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15
Q

What are the common causes of colic: surgical and medical: SMALL intestine?

A
  • Strangulating lipoma
  • Mesenteric rent
  • Epiploic foramen entrapment
  • Ascarid impaction
  • Intussception
  • Hernia
  • Meckel’s diverticulum
  • Mesenteric torsion
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16
Q

What are the common causes of colic: surgical and medical: LARGE intestine?

A
  • Gas
  • Spasmodic
  • Impaction
  • Displacement
  • Enterolith/fecalith
  • Sand
  • Volvulus
  • Cecal impaction
  • *diarrhea=hind gut problem in horses
17
Q

Colic signalment: age importance

A
  • Strangulating lipoma: typically older
  • Proliferative enteropathy: weanling, yearling
  • Ascarid impactions: typically less than 1 year old
18
Q

Colic signalment: sex

A
  • Stallion: check scrotum always!
  • Mare: pregnant; pre vs. post partum
19
Q

Colic signalment: breed

A
  • Different temperatures
    o Stoic (drafts, donkeys) vs. ‘drama queens’
20
Q

Colic history importance

A
  • Duration of colic signs (treatments and response)
  • Passing feces/urinating normally
  • Fever
  • Current diet, water
  • ANY changes
  • Purpose/use of horse
  • Exercise routine
  • Weight loss
  • Deworming and vaccination history
  • Dental care
  • Travel history
  • Any other animals sick
  • Sand paddocks/pastures
  • Medications (NSAIDs: R. dorsal colitis)
  • Orthopedic injury
  • Previous colic or surgery
21
Q

Horse with fever and colic: what should you think?

A
  • Infection or inflammation
  • Lean away from impaction
22
Q

Colic: physical exam

A
  • Mentation
  • Heart rate: suggestive of disease severity
  • Respiratory rate
  • Temperature
  • MM colour and moisture: CRT
  • Digital pulses (ex. may help DDx laminitis)
  • Ventral or peripheral edema
    o Hypoalbuminemia?=colitis; enteropathy
  • Borborygmi; cecal flush
  • Distance exam: Abrasions on head
23
Q

**Nasogastric intubation: colic

A
  • DO FIRST IF TACHYCARDIA
  • Diagnostic and therapeutic
  • Normal reflux <2L
  • *if >2L suggestive of SI issue (strangulation AND duodenal-proximal jejunitis (DPJ))
    o Relief of pain
    o If strangulation=less painful but will still be painful (unchanged pain)
    o If DPJ=visible relax
  • *no reflux=NO SI obstruction (could be distal jejunum)
  • *horses with DPJ usually less painful post reflux, unlike surgical lesion (ex. strangulation)
24
Q

Rectal palpation: colic

A
  • *normal or NOT?
  • If abnormal: ID exact abnormality ideal, but NOT critical to case management
  • Ex. impaction: pelvic flexure
  • Ex. small or transverse colon impaction
  • Ex. L dorsal displacement
25
Abdominocentesis: colic
- Help to differentiate strangulating (vascular=transudate) VS. non-strangulating (inflammation=exudate) lesion - NORMAL= straw-colour and transparent - Maybe not in late stage pregnancy OR if concerned about a large impaction
26
Abdominocentesis: evaluation
- *gross examination - *lactate (normal <2mmol/L) - Total solids - Nucleated cell count - PCV (beware of inadvertent splenic puncture) - Cytology
27
Peritoneal fluid lactate
- Visceral/intestinal ischemia o Peritoneal fluid lactate increases BEFORE blood/plasma lactate o *higher with strangulating obstructions compared to non-strangulating
28
What are some abnormal findings that can be found on ultrasonography (colic)?
- EXCESSIVE free fluid - Stomach: distension - SI o Increased diameter o Increased wall thickness o Absent or increased motility o “target” sign (intussusception) - LI o Nephrosplenic ligament entrapment o Colon torsion o Colitis - *limitations=gas artifacts
29
Radiography (colic)
- Abdomen of adult horse o Portable x-ray units not powerful enough o *referral clinic - *2 main indications: sand impaction and enteroliths
30
Radiography: sand impaction
- Large colon - Along ventral abdomen - Diagnosis and monitoring of resolution
31
Radiography: enteroliths
- Solid concentration of material o Varied mineral composition=different opacities - Generally formed around nidus (ex. foreign body) - Mid-abdominal radiography o Radio-opaque o +/- adjacent air - 96.4% PPV in high prevalence areas
32
Clinical pathology: colic
- PCV/total solids - Lactate
33
*Blood gas (venous): colic
- Acid/base imbalances - Electrolyte concentrations - Lactate concentration - Glucose concentration - +/- creatinine concentration
34
What are other tests you could do but that you don’t routinely need?
- Biochemistry - CBC
35
Blood lactate concentration
- Marker of tissue perfusion - Normal=<2mmol - Ischemia and cellular hypoxia = anaerobic glycolysis = increased lactate - Dehydration = decreased perfusion = increased lactate - Portable analyzer
36
Diarrhea: etiological diagnosis;
- ID cause o Culture o PCR o Toxin assays o Fecal egg count o Sand - Serology: o Lawsonia intracellularis o Neorickettsia risicii - *hang it in a glove and leave overnight