12 - ECG Abnormalities Flashcards

1
Q

When are the PR, QRS and QT interval abnormal?

A
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2
Q

What are the three different types of heart block and explain what they look like on the ECG?

A

- 1st degree: longer PR

- Mobitz Type I/Wenkebach: Increasing PR and then drop of QRS

- Mobitz Type II: Normal PR and then random drop

- 3rd degree (complete block): P and QRS not linked with ventricular rate very slow

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3
Q

What are the causes of heart block?

A
  • MI or degenerative changes
  • Due to failure of conduction either from AV node, or Bundle of His
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4
Q

Why would a myocardial infarction lead to heart block?

A

Coronary artery occlusion in the right so no blood supplying the SAN etc

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5
Q

What is the issue with Type II Mobitz?

A
  • High risk of progression to complete heart block
  • Dangerous rhythm so need pacemaker to prevent this
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6
Q

How do you diagnose and treat 3rd degree heart block?

A
  • Complete failure of atrioventricular conduction
  • Ventricular escape rhythm so wide QRS
  • Slow ventricular conduction rate
  • Urgent pacemaker
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7
Q

What is bundle branch block?

A
  • P wave and PR interval normal
  • Wide QRS >3 small boxes as takes longer for depolarisation past delayed conduction in R or L bundle branches

WILLIAM MARROW

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8
Q

What are the two classifications of arrhythmias?

A
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9
Q

What will supraventricular and ventricular rhythms look like on an ECG?

A
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10
Q

What is atrial fibrillation and what would it appear as on the ECG?

A
  • Supraventricular rhythm where rhythm comes from multiple atrial foci
  • No P waves, wavy baseline
  • Not all atrial are conducted as some in refractory period so irregular R-R and narrow QRS
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11
Q

What is a ventricular ectopic beat?

A
  • Slower depolarisation so wide QRS as impulse not spreading down normal His-Purkinje system
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12
Q

What is ventricular tachycardia?

A
  • Run of more than 3 ventricular ectopics
  • Regular broad tachycardia
  • Dangerous rhythm that needs urgent treatment as risk of VF
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13
Q

What is ventricular fibrillation?

A
  • Abnormal irregular fast ventricular depolarastion
  • Multiple ectopic sites so no co-ordinated contraction
  • No cardiac output and cardiac arrest
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14
Q

How do you treat ventricular fibrillation and how does it work?

A
  • Defibrillator as it is a shockable rhythm
  • Puts all of the ventricular myocytes into repolarisation so they all depolarisation at the same time
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15
Q

What are the questions you should ask yourself when looking at an ECG?

A
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16
Q

Draw a tree diagram of the classifications of arrythmias?

A
17
Q

By looking at an ECG how would you see which coronary artery there has been an occlusion in?

A
18
Q

What is an acute STEMI?

A

- Complete occlusion of the coronary artery by thrombus and the full thickness of the myocardium is involved

- Sub-epicardium damage causes ST elevation in the leads facing the occlusion

  • Need instant treatment to stop necrosis
19
Q

How does a STEMI evolve on an ECG over time?

A
  • Q wave due to necrosis so lead looks through the infarction to the other side so signal travelling away from it
  • T-waves invert due to reversal of repolarisation
20
Q

What is a pathological Q wave?

A

>1 small square wide

>2 small squares deep

Depth more than 1/4 of the height of following R wave

21
Q

What does an old MI look like on an ECG?

A

Pathological Q waves as some areas of necrosis

22
Q

What does a non-stemi ECG look like and why?

A
  • Subendocardial injury leading to ST depression and/or T wave depression
  • Cardiac ischaemia or infarction not affecting full thickness of the wall
23
Q

How do you tell the difference between unstable angina (severe ischemia) and NSTEMI on an ECG?

A

Can’t tell a difference, have to differentiate using troponin as marker for muscle necrosis

24
Q

What does stable angina look like on ECG?

A
  • ST depression only occurs when exercising and reverses at rest
  • Could have narrowed coronary artery
25
Q

What happens to the ECG during hyperkalaemia and why?

A
  • Resting m.p less negative which inactivates some voltage gated Na channels
  • Heart less excitable
  • Conduction problems occur
26
Q

What happens to the ECG during hypokalaemia?

A

Heart becomes more excitable

27
Q

What is the QU interval?

A

Start of R to end of U

28
Q

What leads do you use to look at the following sites?

A
29
Q

What does a prolonged QT interval suggest?

A
  • Prolonged depolarisation and repolarisation
30
Q

What is the path of the femoral artery traced back to the heart?

A
  • External iliac
  • Common iliac
  • Aorta
31
Q

Where does the left coronary artery arise from?

A
  • Left coronary sinus above the aortic valve flap
32
Q

What drug is a positive inotrope but will slow conduction at the AV node?

A

Cardiac glycoside

33
Q

What murmur will you hear in a patent ductus arteriosus?

A

Machinery murmur as pressure in aorta always greater than in pulmonary artery

34
Q

Where can you listen for the different heart sounds in the heart? e.g where would you listen for an issue with the aortic valve?

A
35
Q

When are the different regurgitation and stenosis heart sounds heard and what type of sound are they? e.g mitral valve stenosis

A
36
Q

A woman had an MI in her LAD 6 months ago, what would her ECG look like now and what leads would this be seen in?

A

Q waves in V2-V4

37
Q

A GP can hear a murmur in the right second intercostal space, what is this suggestive of?

A

Aortic stenosis