10. Shortness of Breath Flashcards
How can you classify the causes of SOB by timing?
Sudden- PE, pneumothorax, foreign body
Days- pneumonia, heart failure and pleural effusion
Weeks- COPD, chronic asthma
What are some of the risk factors to ask about in someone with SOB?
Smoking Occupation DVT/ PE risk factors Drugs PMHx- autoimmune conditions like rheumatoid arthritis and SLE can cause interstitial lung disease and pleural effusions
What associated symptoms should you ask someone with SOB?
Chest pain, cough, wheeze, FLAWS, muscle weakness/ fatigue
What is COPD?
Bracket term covering chronic bronchitis and emphysema
What causes COPD?
Smoking- >20 pack years usually
Occupational exposure to lung irritants
Alpha 1 antitrypsin deficiency
What are signs of COPD?
Hyperexpanded chest, breathing through pursed lips, reduced air entry/ chest expansion, prolonged expiratory phase, hyper-resonant percussion
What are some key features in the HPC for asthma?
Wheeze, cough that is worse at night, other atopies, symptoms worse with NSAIDs, aspirin, beta blockers
What signs might you see on examination in someone with interstitial lung disease?
Clubbing, reduced chest expansion, late inspiratory fine crackles
What are some of the key features in the HPC for heart failure?
History of CVD, hypertension, valve disease, cardiomyopathies
Orthopnea, paroxysmal nocturnal dyspnoea, swelling, sleeping with multiples pillows, pink frothy sputum
What are some of the symptoms of congestive heart failure and the reasoning behind them?
SOB- heart isn’t pumping enough blood. Back pressure forces fluid from the pulmonary vasculature into the alveoli.
Orthopnoea- lying down increases venous return to the heart while it is already struggling to pump. Blood becomes more congested in pulmonary vaculature, forcing fluid into the lungs.
Displaced apex beat due to dilation of the heart.
Crackles in lungs due to build up of blood in pulmonary vessels, forcing fluid into alveoli.
Peripheral oedema due to back pressure in the venous system and salt + water retention due to activation of RAAS by a low cardiac output.