10 - Feline Dentistry Flashcards
What are three common oral conditions felines experience whereas in other species they are rare
Chronic ulcerative gingivostomatitis (CUGS)
Tooth resorption (TR)
Eosinophilic granuloma complex
What is CUGS?
feline chronic ulcerative gingivostomatitis
an inflam of mucous lining of any of the sutures in the mouth
more severe than regular gingivitis associated with PD dz
can be an individual inappropriate immune response from cat to a variety of antigenic triggers (ex. pasteurella bact, plaque back, and calicivirus)
Why is CUGS so debilitating? What does it result in
leads to loss of all teeth in cats at a very early age
heightened immune response -> severe oral inflam, oral ulcers, foul breath, resorption of hard dental tissue, dysphagia
some patients have large areas of their oral cavity affected w/ painful, raw areas
tx is extensive, long-term, w/ dangerous side effects and some drugs being used off-label
important to note that whole mouth extractions may be necessary
What is an off-label drug?
using a drug not for its intended purpose
What are the clinical signs of CUGS?
gingival bleeding is earliest sign
inflamed gingiva + mucosa may appear swollent, cobblestone-textured,b right red or rasberry-like
light touch of gingiva = spontaneous hemorrhage
hypersalivation -. saliva is ropy and thick
many stop eating bc of pain
How do we diagnose CUGS?
full mouth exam inc dental rads and PD charting is required
good quality photographs also aid ability to make comparisons and gauge the success of the tx regime set out
may reveal info about the cause of the individual’s stomatitis
complete hx and PE required
A complete blood count, thyroid, biochem tests, rule out FeLV + FIV
Autoimmune testing + food testing to rule out allerg reaction to diet
How do we help diagnose CUGS under anesthesia?
exam all surfaces for color, shape, texture, pain response and gingival bleeding
biopsy to determine probably etiology
intraoral rads necessary to eval teeth w/ erosive dz
How do we manage stage 1 of CUGS?
Complete oral health assessmet + tx inc intraoral rads
all teeth affected by resorption or periodontitis must be extracted
any retained root tips extracted
Ab’s + NSAID’s and pain meds prescribed
after procedure, aggressive home care is instituted (tooth brushing, dental diet, antiseptic oral gels + rinses, dental checks every 6mo, frequent dental cleanings every 6-12mo)
How do we manage stage 2 CUGS
If stage 1 tx fails, Owner unwilling/unable to do homecare
involves numerous tooth extractions - minimally all of caudal teeth (P’s + M’s), must ensure all tooth roots + PD ligament is removed, may require extraction of I’s and C’s in some patients if gingivitis is significant
still dental rechecks every 6mo
How do we manage stage 3 of CUGS?
extract all remaining teeth
remove all roots + PD ligaments
trials show promise w/ omega interferon (local injection and daily dosing) but its not available in CAD or US except by Emergency Drug Release
What is stage 4 management of CUGS?
up to 30% of cats continue to have significant oral inflam despite extraction of all teeth and removal of PD ligament
long term antibiotic and steroid therapy, may have gold salt therapy and Interferon’s
euthanasia is unable to adequately control dz
What is CUPS in dogs?
Chronic ulcerative proliferative stomatitis
uncommon in dogs
also involves an overexaggerated immune response to plaque, may be immune mediated
causes severe gingivitis and gingival recession
classic “kissing lesions” on mucosa that touches plaque-covered teeth
similar aggressive tx as for CUGS in cats
What is tooth resorption in cats? What % of all cats are affected by it?
AKA feline odontoclastic resoprtive lesion (FORL), neck lesions, cervical line lesions and cat cavities
~50% affected with TR
effects of TR is resorption of tooth, proliferation of gingiva or pulp to cover resulting lesion
Clin signs is paid, behave changes, dec appetite, drops food or hiss at it
What is the etiology of TR?
dunno why it occurs
results from activation of cells called odontoclasts responsible for normal remodeling of tooth structure, in dz process - continue to resorb tooth structure until entire tooth is lost, at same time have proliferation of gingiva which covers resulting lesion
tend to occur at or just below gumline but may affect only root structure
rads necessary to dx and tx TR
What teeth are most commonly affected in TR?
premolars -> molars -> canines
majority of lesions on buccal surface
most common is 307/407