1 Flashcards

1
Q

Travel thru CTR and SOF

A

NOA
Nasociliary (V1)
Oculomotor (3)
Abducens (6)

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2
Q

Travel thru SOF, above CTR

A
FLighT
Frontal (V1)
Lacrimal (V1)
Trochlear (4)
And superior ophthalmic vein
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3
Q

Travel thru IOF, below CTR

A

Inferior ophthalmic vein

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4
Q

Travel thru optic canal

A

Optic nerve

Ophthalmic artery

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5
Q

Which layer of the eye is not found in the limbus as it is defined histologically

A

Bowman’s

By definition, is anterior to the border of the limbus

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6
Q

Which structure is composed of modified skin and contains goblet cells

A

Caruncle

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7
Q

What is responsible for the production of the lens capsule

A

Lens epithelium

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8
Q

Which nerves innervate the conj

A

Lacrimal (V1)
Long posterior ciliary
Infraorbital (V2)

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9
Q

Innervation of iris sphincter

A

PNS fibers in SPCNs

PNS from EW nucleus travel with CN3, synapse at ciliary ganglion (e.g. ADies)

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10
Q

Innervation of iris dilator

A

SNS fibers in LPCNs and SPCNs

SNS from superior cervical ganglion (without synapsing) travel with L/SPCNs to dilator

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11
Q

Eyelid epidermis vs other parts of body

A

Lacks adipose tissue

Thinner

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12
Q

Cell bodies of somatic system lie in

A

Ventral horn

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13
Q

Which structure within the lower MB is responsible for reflex responses of the head and neck to auditory stimuli

A

Inferior colliculus

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14
Q

Circle of Willis composed of

A

Anterior and posterior communicating
Anterior and posterior cerebrals
Internal carotid

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15
Q

Oral bays vs dentate processes

A

OB: pLana extending into retina

DP: retina extending into plana

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16
Q

Areas of sclera containing elastic fibers (3)

A

Lamina cribrosa
Scleral spur
Lamina fusca

17
Q

NTMs

  • most widely distributed
  • who uses glutamate
  • who uses glycine
A

GABA

Glut: PR, BP, GC

Glyc: retina, brain stem, spinal cord

18
Q

Frontal lobe (4)

A

Personality
Planning
Production of speech = Broca’s
FEFs = saccades (contral), near pupillary response

19
Q

Parietal lobe (2)

A

Pursuits (ipsi)

Sensory - e.g. pt knows a pen is used for writing, but unable to call it a pen

20
Q

Temporal lobe (2)

A

Hippocampus = short-term memory, spatial recognition
Wernicke’s = speech recognition
-“wordy speech”: perfect sounding speech, but words do not make any sense

21
Q

Medulla

  • contents (2)
  • functions (3)
A

MLF
CNs: 9-12

Autonomic: breathing, heart rate, digestion

22
Q

Telencephalon and Diencephalon

A

Separated by optic chiasm
T = behind
D = in front of chiasm, incr eye (outgrowth)

23
Q

Hypothalamus regulates (3)

A

Body temp
Eating
Sleeping

24
Q

Unhealthy optic nerve

  • normal app
  • edematous
  • atrophic
A

Norm: retrobulbar optic neuritis

Edema:

  • unilateral = inflammation (TB, sarcoid), vascular (DM), ischemic (N/AAION), compression (TED)
  • bilateral = HTN, tumor, inflammation, pseudotumor cerebri

Atrophic:

  • primary = toxic (alcohol, ethambutol), hereditary (Leber’s), trauma, ortho/retrograde
  • secondary = anything after edematous
25
Q

What to think of for:

  • AAION
  • NAION
A

AAION = temporal arteritis = GCA

  • jaw claudication
  • anorexia

NAION

  • assoc with meds (VIAgra), apnea, nocturnal brady, DM
  • disc at risk
26
Q

AAION

  • who
  • pathophys
A

> 55yo

Secondary to occlusion of SPCAs
Most common cause GCA/temporal arteritis
-less commonly polyarteritis nodosa, SLE, HZV

27
Q

AAION

-signs/symp

A

Signs: unilateral disc edema + APD + decr vision

Symp:

  • ocular: AMAUROSIS FUGAX, sudden vision loss
  • systemic: temporal HA, JAW CLAUDICATION, NECK PAIN, ANOREXIA, tender/nodular temporal artery, malaise, fever, myalgia, scalp tenderness
28
Q

AAION

-diagnostics

A

Evaluate STAT for elevated:

  • ESR: > age/2 men, > (age + 10)/2 women
  • CRP: >2.45 mg/dL
  • Platelets: >400k

Elevated ESR and CRP are 97% specific for GCA

29
Q

NAION

  • who
  • pathophys
A

> 50yo with hx of HTN, DM, and/or hypercholesterolemia
90% have small, crowded disc with C/D <0.3 (“disc at risk”)

Secondary to irreversible ischemia and anterior optic nerve due to unknown etiology
Nocturnal hypotension may contribute to development

30
Q

NAION

-signs/symp

A

Signs: decr vision, APD, unilateral disc edema, VF loss (esp inferior altitudinal)

Symp: sudden, painless, NON-PROGRESSIVE vision loss

  • often occurs upon awakening
  • rarely improves after initial onset
31
Q

NAION

-diagnostics

A

No assoc with systemic symptoms (vs AAION/GCA)

Normal ESR/CRP/platelets

DX OF EXCLUSION