Complicated OB pt2 Flashcards

(41 cards)

1
Q

What is the most prominent symptom associated with umbilical cord compression from cord prolapse?

A
  • Fetal bradycardia (from FHT)
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2
Q

What are the possible causes of umbilical cord prolapse?

A
  • Multiple gestation
  • Abnormal presentation (breech, shoulder, etc.)
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3
Q

What is the initial primary management of cord prolapse?

A

Manual elevation of the presenting part to offload the umbilical cord.

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4
Q

Other than manual displacement of the fetus, what other option is available to manage cord prolapse?

A

Retrograde bladder filling w/ 500 - 600 mls.

If this doesnt work or fetal distress, then c-section.

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5
Q

What are the two different types of twins?

A
  • Monozygomatic twins (identical)
  • Dizygotic twins (fraternal)

monozygomatic: conceived from one egg

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6
Q

In what type of situation is it more likely to have twin-twin transfusion to the other in the womb?

A

Monochorionic (rare in dichorionic)

vascular communications

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7
Q

Monochorionic monoamniotic refers to what?

A
  • Same placenta
  • Same amniotic fluid
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8
Q

What are the two body systems we typically worry about with mom when she is multiple-gestation?

A

CV & pulmonary issues

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9
Q

How much does CO increase for a multiple-gestation mom?

A

20% greater than a typical parturient

primarily related to increased stroke volume

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10
Q

multiple-gestation parturients are at increased risk of hypoxemia. Why?

A

↓ TLC & FRC near gestation due to increased uterine size

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11
Q

What systems do not change in a multiple-gestation parturient vs a woman having only one baby?

A
  • Renal
  • Hepatic
  • CNS

All the same whether you have one baby or multiple.

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12
Q

What are the consequences of the stomach being displaced cephalad in a multiple-gestation patient?

A

↓ LES competence = ↑ aspiration risk

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13
Q

What is the maternal estimated blood volume of a multiple-gestation patient?

A

105 mL/kg

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14
Q

multiple-gestation delivery EBL is typically _____ greater than a single baby delivery.

A

500 mL

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15
Q

How much more plasma volume does a multigestational patient have than a monogestational patient?

A

additional 750 mL plasma volume

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16
Q

Regarding twin-to-twin transfusion syndrome, what would be likely to be seen from the donor twin?

A
  • Smaller size
  • ↑ risk of IUGR
  • ↑ risk of anemia

giving away all his nutrients

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17
Q

Regarding twin-to-twin transfusion syndrome, what would be likely to be seen from the recipient twin?

A
  • Larger size
  • ↑ risk of volume overload
  • ↑ risk of cardiac failure
18
Q

Multiple-gestation patients tend to be at risk for _____ delivery.

A

early (preterm)

>50% delivery before 37 weeks.

19
Q

Planned delivery for twins typically occurs at ____ weeks.
Planned delivery for triplets typically occurs at ____ weeks.

A

38 weeks

35 weeks

to decrease perinatal mortality

20
Q

Increased fetal weight and larger volume of amniotic fluid in a multiple gestation mom can lead to?

A
  • increased risk aortocaval compression and supine HoTN syndrome
21
Q

Increased uterine distention from multiple gestation increases the risk of ______ and ______.

A

uterine atony & PPH

Have methergine & hemabate ready.

22
Q

Delivery with GETA usually poses what risks?

A
  • increased risk of difficult airway
  • increased EBL
23
Q

How are PIH (pregnancy induced hypertension) and preeclampsia differentiated?

A

Proteinuria = Preeclampsia

24
Q

What is the BP criteria for pregnancy induced HTN?

A
  • BP elevated > 139/89 mmHg x2 (visits)
  • after 20 weeks gestation
  • without proteinuria
25
___% of parturients with gestational HTN will develop preeclampsia
25%
26
What makes a definitive diagnosis of PIH?
Delivery of baby and return to normotension (resolves by 12 weeks post-partum)
27
A new onset HTN & proteinuria after 20 weeks would be suspect for _______.
Preeclampsia *key feature: proteinuria*
28
What alternate s/s can develop with preeclampsia (besides HTN & proteinuria)?
- Persistent epigastric/RUQ pain - Persistent cerebral s/s (syncope, blurry vision) - IUGR - Thrombocytopenia - ↑ LFT's
29
What are the characteristics of preeclampsia **without severe** features?
- BP ≥ 140/90 after 20 weeks gestation - Renal Insufficiency: [Proteinuria ≥ 300mg/24hrs or Protein:Creatinine ≥ 0.3 or 1+ on urine dipstick]
30
What features are typical of **severe** preeclampsia?
- BP ≥ 160/110 - Thrombocytopenia (PLT < 100k) - Creatinine > 1.1 or 2x baseline - Pulmonary edema - New onset cerebral/vision disturbances - Impaired liver function
31
How can chronic HTN be distinguished from pregnancy induced?
* Prepregnancy HTN >140/90 * HTN does not resolve postpartum
32
Review: Pregnancy HTN Disorders Table
33
Is a fetus necessary to develop preeclampsia?
Technically no, can be in a molar pregnancy
34
Abnormal placental implantation and impaired remodeling of spiral arteries is indicative of what?
Preeclampsia *small and constricted spiral arteries unable to adequately provide O2 and nutrients to fetus*
35
What are the vascular features of preeclampsia?
36
Preeclampsia is early onset if it occurs before ____ weeks. Is this associated with better or worse outcomes?
Before 34 weeks ⇒ worse outcomes
37
What are the features of postpartum-onset of preeclampsia?
- Occurs within one week of delivery. - Proteinuria - Seizures
38
What is prophylaxis on preeclampsia patients?
Aspirin
39
Why is aspirin a prophylactic for preeclampsia?
- Inhibits synthesis of prostaglandins & synthesis of thromboxane α2
40
When should aspirin be initiated for patients at high risk of preeclampsia?
16 weeks or earlier for best benefit
41
________ is increased relative to prostacyclin in preeclamptic patients.
Thromboxane