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MAP III final > wound healing > Flashcards

wound healing Flashcards

1
Q

what are the three primary stages of wound healing?

A

inflammation, proliferation, remodeling

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2
Q

what are the three stages of inflammation?

A

vasoconstriction, hypoxia, and perfusion

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3
Q

what mediates vasoconstriction?

A

norephinephrine

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4
Q

what happens if you have too much hypoxia during inflammation stage of healing?

A

it impairs macrophages and increases risk of infection

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5
Q

what occurs during perfusion during the inflammation stage of healing?

A

vasodilation occurs and this allows for inflammatory cells to get to the wound site

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6
Q

What occurs during the proliferation stage of wound healing?

A

the wound fills in with new tissue and a new capillary bed are put in place

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7
Q

what type of collagen is initially laid down in the wound?

A

type III, more elastic, minimal tensile strength

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8
Q

what type of collagen is normal skin?

A

type I, more tensile strength

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9
Q

what is the role of myofibrilblasts?

A

pull the edges of the wound together

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10
Q

what occurs during the remodeling phase of wound healing?

A

type III collagen is replaced by type I collagen

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11
Q

what color is eschar?

A

black or brown tissue

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12
Q

what color is slough?

A

white, gray, yellow, or tan tissue

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13
Q

what color is granulation?

A

light pink to beefy red

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14
Q

what color is epithelialization?

A

whitish, pale pink to red

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15
Q

what color is tendon?

A

yellow to white

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16
Q

what is a big difference between sharps debridement and mechanical debridement?

A

sharp is selective and mechanical is non selective

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17
Q

describe wet to dry dressing

A

put dressing on for 8 hours, pull off, hope tissue comes with it. Poor option and not effective on dry eschar

18
Q

describe irrigation

A

spray wound with syringe

19
Q

describe enzymatic debridement

A

usually used in foreign countries, selective, takes 3-30 days, DON’T USE on clean wounds

20
Q

describe autolytic debridement

A

selective, basically a mix of macrophages that eat necrotic tissue

21
Q

for autolytic debridement what type of dressing would you use with dry eschar?

A

film

22
Q

for autolytic debridement what type of dressing would you use with necrotic dry wounds?

A

hydrogels

23
Q

for autolytic debridement what type of dressing would you use with necrosis?

A

hydrocolloids

24
Q

when should you not use autolytic debridement?

A

with a patient who has an active infection!!!!!

25
Q

what is a bright point of hydrogels and hydrocolloids?

A

they do autolytic debridement once they are placed on.

26
Q

what is the biggest difference between hydrogels and hydrocolloids?

A

hydrogels won’t adhere to wounds and contributes a lot more water

27
Q

what dressing can’t be used on a patient with a glycol sensitivity?

A

hydrogels

28
Q

if you are concerned about hypergranulation which dressing should not be used and why?

A

hydrocolloids because they cause hyperoxygenation which will accelerate granulation

29
Q

Who should regranex be used on and who should it not be used on?

A

use on LE diabetic ulcers, DON’T use on patients with known neoplasm

30
Q

what is extra cellular matrix replacement?

A

really expensive stuff that enhances growth factor

31
Q

why should antiseptics only be used in the early phases of healing?

A

they kill bacteria but also kill granulation cells so they blunt healing

32
Q

what is a good use for chlorhexidine?

A

hand sanitizer, not good for much else as it can cause burns

33
Q

what is quaternary ammonium compounds good against?

A

bacterial gram + and - organisms

34
Q

what is iodine good for?

A

fungis, viruses, bacteria, and spores. Can damage healing of incisions though

35
Q

cadexomer iodine may improve healing in _________

A

venous wounds

36
Q

silver release dressings may improve healing in _______

A

chronic wounds

37
Q

UV light may _______

A

reduce bacterial load including MRSA

38
Q

what are the benefits of US during the inflammation stage?

A

It accelerates the inflammation phase, can restart inflammation phase in chronic wounds

39
Q

what are the benefits of US during proliferation?

A

stimulates angiogenesis, accelerates wound contraction

40
Q

what are the benefits of US during remodeling?

A

can only help if started in inflammatory phase, increases wound strength, increases elasticity

41
Q

what are the benefits of UV?

A

initiates inflammatory response, has a bactericidal effect

MAP III final (1 decks)

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