Shock Flashcards
What are the chances of dying from shock?
How do you prevent it?
10% to 100% chance
prevent= prophylactic treatment
Significance of shock
It is a life threatening condition–
affects all body systems
organs struggle to survive
often develops rapidly (depending on the cause)
what is shock caused by
nonspecific “assault” to the body
infectious or noninfectious
– hence all patients are at risk for shock
what is the best treatment of shock
prevention
what can early recognition do
save lives
Shock
the condition itself:
a state of hypoperfusion
a state of hypoperfusion =>
O2 debt: decreased O2 supply
Acidosis: poor removal of metabolites and toxins at the cellular level
body’s response to shock
stimulation of ANS sympathetic pathway
Body’s response: stimulation of ANS sympathetic pathway
hypermetabolism=> increase demand
- worsening o2 debt => anerobic metabolism => lactic acidosis
- peripheral insulin resistance, later systemic and ketoacidosis
inflammatory response=> SIRS, hypercoagulability
SIRS
systemic inflammatory response syndrome =>
response to nonspecific assult=> Ischemia Inflammation Trauma Infection
criteria for SIRS (2 or more)
Temp >100.4 or <96.4 HR >90 RR >20 PaCO2 < 32 (35-45 mmHg) WBC >12000, <4000, >10% band Lactate level >2 mmol/L SBP < 90 after fluid bolus test
Components of perfusion
Circulating Volume:
Fluid volume status
Preload
Pump: contractility valve function HR pre/after-load rhythm
Vascular System:
Vascular integrity
tone
resistance
Types of shock
Hypovolemic Shock
Cardiogenic Shock
Obstructive Shock
Circulatory Shock
- Septic
- Neurogenic
- Anaphylactic
Hemodynamic MonitoringHow to monitor when all you have at the bedside is BP!
MAP = (SBP+ 2DBP) / 3
MAP must be > 65 to provide adequate perfusion
PP: measure of stroke volume and vascular resistance
PP= SBP – DBP (30-40)
Narrow (↓)PP = early poor perfusion (vasoconstriction = higher DBP)
Wide (↑) PP = late poor perfusion (vasodilation = lower DBP)
Timely recognition shock is difficult!
No definitive S&S
Each patient presents differently
Early clinical manifestations may go unrecognized
- can be very apparent (massive external hemorrhage)
- can be silent (compensatory phase of septic shock)