Shock Flashcards

1
Q

What are the chances of dying from shock?

How do you prevent it?

A

10% to 100% chance

prevent= prophylactic treatment

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2
Q

Significance of shock

It is a life threatening condition–

A

affects all body systems
organs struggle to survive
often develops rapidly (depending on the cause)

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3
Q

what is shock caused by

A

nonspecific “assault” to the body
infectious or noninfectious
– hence all patients are at risk for shock

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4
Q

what is the best treatment of shock

A

prevention

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5
Q

what can early recognition do

A

save lives

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6
Q

Shock

the condition itself:

A

a state of hypoperfusion

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7
Q

a state of hypoperfusion =>

A

O2 debt: decreased O2 supply

Acidosis: poor removal of metabolites and toxins at the cellular level

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8
Q

body’s response to shock

A

stimulation of ANS sympathetic pathway

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9
Q

Body’s response: stimulation of ANS sympathetic pathway

A

hypermetabolism=> increase demand

  • worsening o2 debt => anerobic metabolism => lactic acidosis
  • peripheral insulin resistance, later systemic and ketoacidosis

inflammatory response=> SIRS, hypercoagulability

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10
Q

SIRS

systemic inflammatory response syndrome =>

A
response to nonspecific assult=> 
Ischemia
Inflammation
Trauma
Infection
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11
Q

criteria for SIRS (2 or more)

A
Temp >100.4 or <96.4
HR >90
RR >20
PaCO2 < 32 (35-45 mmHg)
WBC >12000, <4000, >10% band
Lactate level >2 mmol/L
SBP < 90 after fluid bolus test
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12
Q

Components of perfusion

A

Circulating Volume:
Fluid volume status
Preload

Pump: 
contractility
valve function
HR
pre/after-load
rhythm

Vascular System:
Vascular integrity
tone
resistance

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13
Q

Types of shock

A

Hypovolemic Shock

Cardiogenic Shock

Obstructive Shock

Circulatory Shock

  • Septic
  • Neurogenic
  • Anaphylactic
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14
Q

Hemodynamic MonitoringHow to monitor when all you have at the bedside is BP!

A

MAP = (SBP+ 2DBP) / 3
MAP must be > 65 to provide adequate perfusion

PP: measure of stroke volume and vascular resistance
PP= SBP – DBP (30-40)
Narrow (↓)PP = early poor perfusion (vasoconstriction = higher DBP)
Wide (↑) PP = late poor perfusion (vasodilation = lower DBP)

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15
Q

Timely recognition shock is difficult!

A

No definitive S&S

Each patient presents differently

Early clinical manifestations may go unrecognized

  • can be very apparent (massive external hemorrhage)
  • can be silent (compensatory phase of septic shock)
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16
Q

Stages of Shock

A

Stage I: “Compensatory stage”
compensatory responses stabilize the perfusion, preventing further deterioration

Stage II: “Progressive stage”
clear signs of hypoperfusion and organ dysfunction

Stage III: “Irreversible stage” (refractory)
Massive cellular destruction results inevitably in death