Schizophrenia- Psychological Explanations For SZ Flashcards

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1
Q

Family dysfunction -

A

Abnormal processes within a family such as poor family communication, cold parenting and high levels of expressed emotion. These may be risk factors for both the development and maintenance of schizophrenia.

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2
Q

Cognitive explanations -

A

Explanations that focus on mental processes such as thinking, language and attention.

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3
Q

Dysfunctional thought processing -

A

A general term meaning information processing that is not functioning normally and produces undesirable consequences.

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4
Q

Psychological Explanations for Schizophrenia

A

There are a range of psychological explanations for schizophrenia, focusing on both the family environment and abnormal cognitive processes.

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5
Q

What do psychological explanations for schizophrenia focus on?

A

They focus on the family environment’s role in vulnerability to schizophrenia and abnormal cognition in sufferers.

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6
Q

Family Dysfunction in Schizophrenia

A

Family dysfunction links schizophrenia to childhood and adult experiences of living in a dysfunctional family environment.

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7
Q

How does family dysfunction relate to schizophrenia?

A

It suggests that experiences in a dysfunctional family increase vulnerability to schizophrenia.

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8
Q

Schizophrenogenic Mother

A

Frieda Fromm-Reichmann (1948) proposed that a “schizophrenogenic mother” is cold, rejecting, and controlling, creating a family climate of tension and secrecy. This climate leads to distrust, which later develops into paranoid delusions and ultimately schizophrenia.

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9
Q

What is the schizophrenogenic mother, according to Fromm-Reichmann?

A

A mother who is cold, rejecting, and controlling, fostering an atmosphere of distrust that may lead to schizophrenia.

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10
Q

Double-Bind Theory

A

Gregory Bateson et al. (1972) proposed that family communication style, particularly “double-bind” situations, contributes to schizophrenia. In these situations, a child receives mixed messages and cannot comment on the unfairness, leading to confusion and anxiety. This experience is linked to disorganized thinking and paranoid delusions.

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11
Q

What is double-bind theory?

A

The idea that mixed messages in family communication create confusion and fear, potentially leading to disorganized thinking and paranoia.

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12
Q

Expressed Emotion (EE) and Schizophrenia

A

Expressed Emotion (EE) is the level of negative emotion directed toward a patient by their carers. It includes elements like verbal criticism, hostility, and over-involvement. High EE levels increase stress for the patient, making relapse more likely, and may trigger schizophrenia onset in those genetically predisposed.

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13
Q

What is Expressed Emotion (EE), and how does it affect schizophrenia?

A

EE is the level of negative emotion from carers, which can increase relapse risk and potentially trigger schizophrenia in vulnerable individuals.

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14
Q

Cognitive Explanations for Schizophrenia

A

Cognitive explanations focus on the role of mental processes and abnormal information processing in schizophrenia.

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15
Q

What do cognitive explanations for schizophrenia focus on?

A

They focus on abnormal thought processing as a basis for understanding schizophrenia symptoms.

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16
Q

Disrupted Thought Processing in Schizophrenia

A

Schizophrenia is characterized by disruptions in normal thought processing. Reduced activity in the ventral striatum correlates with negative symptoms, while reduced processing in the temporal and cingulate gyri is associated with hallucinations.

17
Q

How does disrupted thought processing manifest in schizophrenia?

A

It includes lower-than-normal processing in specific brain regions, leading to symptoms like hallucinations and negative symptoms.

18
Q

Frith et al. (1992) on Dysfunctional Thought Processing

A

Christopher Frith et al. (1992) identified two types of dysfunctional thought processing in schizophrenia: metarepresentation and central control.

19
Q

What two types of dysfunctional thought processing did Frith et al. identify in schizophrenia?

A

Metarepresentation and central control.

20
Q

Metarepresentation Dysfunction in Schizophrenia

A

Metarepresentation is the cognitive ability to reflect on one’s own thoughts and behavior and to interpret others’ actions. Dysfunction here could explain hallucinations and delusions like thought insertion.

21
Q

What role does metarepresentation play in schizophrenia?

A

Dysfunction in metarepresentation may cause individuals to misattribute thoughts and actions, leading to symptoms like hallucinations.

22
Q

Central Control Dysfunction in Schizophrenia

A

Central control is the cognitive ability to suppress automatic thoughts. Dysfunction here could lead to disorganized speech, as the patient struggles to suppress automatic associations that derail thoughts and spoken sentences.

23
Q

What happens when central control is dysfunctional in schizophrenia?

A

Patients experience disorganized speech, as they are unable to suppress automatic responses and associations.

24
Q

Evaluation: Support for Family Dysfunction as a Risk Factor

Read et al. (2005)

A

Study Name: Read et al. (2005)

Findings: Reviewed 46 studies and found that 69% of adult women and 59% of men with a diagnosis of schizophrenia had a history of physical or sexual abuse in childhood.

Conclusion: This study suggests a link between childhood abuse and later development of schizophrenia, supporting family dysfunction as a risk factor.

25
Q

Evaluation: Support for Family Dysfunction as a Risk Factor

Berry et al. (2008)

A

Study Name: Berry et al. (2008)

Findings: Found that adults with insecure attachments to their primary caregivers are more likely to develop schizophrenia.

Conclusion: This adds to the body of evidence that family dysfunction, specifically insecure attachment, may increase vulnerability to schizophrenia.

26
Q

Limitations of Evidence for Family Dysfunction

A

Issue: Most evidence is based on retrospective accounts, meaning patients recall childhood experiences after developing schizophrenia.
Problem: Schizophrenia may distort patients’ memories of their childhood, creating a validity issue.

Alternative Evidence: Only a small number of prospective studies (e.g., Tienari et al.) exist that follow children over time to observe if childhood experiences predict schizophrenia, and results from these studies are inconsistent.

27
Q

Evaluation: Weak Evidence for Family-Based Explanations

A

Critique: There is limited evidence to support the specific theories of the schizophrenogenic mother or double-bind communication.

Reason: These theories are largely based on clinical observation and the assessment of patient mothers for ‘crazy-making characteristics,’ a subjective and outdated method (Harrington 2012).

28
Q

Parent-Blaming Issue:

A

Problem: Family-based theories have historically led to blaming parents, adding trauma for parents already dealing with their child’s condition.

Historical Shift: The move from hospital to community care in the 1980s, which often placed responsibility on parents, may have led to a decline in popularity of the schizophrenogenic mother and double-bind theories, as parents rejected these ideas.

29
Q

Evaluation: Strong Evidence for Dysfunctional Information Processing

Stirling et al. (2006)

A

Study Name: Stirling et al. (2006)

Method: Compared 30 schizophrenia patients to 18 non-patient controls on cognitive tasks, including the Stroop Test (which measures central control by having participants name the ink color of color words while suppressing the impulse to read the words).

Findings: Patients with schizophrenia took more than twice as long as controls to name the ink colors, supporting Frith’s theory of central control dysfunction.

30
Q

Criticism of Cognitive Explanations:

A

Issue: Cognitive explanations only clarify the proximal causes of schizophrenia (i.e., they explain current symptoms) and not the distal causes (i.e., the origins of the condition).

Conclusion: Although cognitive theories reveal links between symptoms and faulty cognition, they don’t explain the root cause of schizophrenia.

31
Q

Evidence for biological factors is not adequately considered

A

In their pure forms at least, psychological explanations (particularly family dysfunction) for schizophrenia can be hard to reconcile with the biological explanations we looked at on the previous spread. It could be that both biological and psychological factors can separately produce the same symptoms, which raises the question of whether both outcomes are really schizophrenia.

Alternatively, we can view this in terms of the diathesis-stress model where the diathesis may be biological (as discussed on the previous spread) or psychological (as discussed on this spread).

32
Q

Direction of causality

A

We have a mass of information concerning abnormal cognitions as well as a mass of information about abnormal biology in schizophrenia. However, it remains unclear what causes what, including whether cognitive tactors are a cause or are a result of the neural correlates and abnormal neurotransmitter levels seen in schizophrenia.