Nutrition Planning and Intervention Flashcards
Nutrition Intervention
purposely planned actions designed with the intent of changing a bheavior, risk factor, or condition
- based on nutiriton dx and provides the bases upon which outcomes are measured and evaluated
four categories:
1) food delivery
2) education
3) counseling
4) coordination of care
Primary prevention programs
reduced exposure to a promoter of disease
ex. early screening for DM
Secondary prevention programs
recruiting those w/ elevated risk factors into tx program; reduce impact of condition
ex. risk reduction
FTC / NCAHF
Federal Trade Commision
National Council Against Health Fraud
fights against health care fraud
Tertiary prevention programs
as disease progress, intervention to reduce severity, manage complications
ex. rehab
Ulcer
eroded mucosal lesion typically caused by H. pylori
Diet:
- as tolerated
- avoid late night snacks
- omit gastric irritants (e.g. pepper, chili, caffeine, alcohol)
Hiatal hernia
protrusion of a portion of the stomach
Diet:
- small, bland feedings
- avoid late night snacks
- omit gastric irritants
Dumping syndrome
food moves quickly from stomach to SI
*rapid digestion and absorption of CHO stimulates overproduction of insulin resulting in rapid drop in BGs (reactive hypoglycemia)
Billroth I
attaches remaining stomach post-gastrectomy to duodenum
Billroth 2
attaches remaining stomach post-gastrectomy to jejunum
*no duodenum, no enzymes to stimulate pancreatic secretion
*affects Ca (absorbed in duodenum) & Fe absorption (requires HCl)
Complete gastrectomy
Removal of stomach
*results in Fe, B12 (lack of intrinsic factor + backterial overgrowth), folate (needs B12 for transport + low serum Fe which is a cofactor), Ca, Vit D, B1, and copper deficiencies
Diet:
- frequent small, dry feedings, fluids before/after meals
- 50-60% complex CHO, protein, mod fat
Gastroparesis
Delayed gastric emptying / slow movement from stomach to SI
Rx: prokinetics to increase stomach contractility (e.g. erythromycin, metoclopramide)
Diet:
- small, frequent meals
- avoid high fiber and high fat
- avoid gastric irritants
Tropical Sprue
chronic GI diesease caused by bac, vir, and para infection that affects absorption
SS: diarrhea, malnutrition, B12 and folate deficiency
Diet:
- B12 and folate supplement
- high cal, high pro
Celiac disease
immune reaction to gluten, a type of prolamin
SS: malabsoprtion, macrocytic anemia, weight loss, steatorrhea, Fe deficiency anemia
Diet:
- gluten-free/gliadin-free diet (no wheat, rye, oats, buckwheat, bran, graham, malt, mulgur, couscous, durum, orzo, thickening agents)
Constipation
hard to pass
Diet: high fluid, high fiber, exercise
Diverticulosisr
presence of mucosal sacs protruding on intestinal wall
diet: high fiber diet
Diverticulitis
when mucosal sacts on intestine wall become inflamed
diet: clear liquid, gradual return to fiber, elemental formula
Benefits of soluble fiber
1) decrease serum cholesterol by binding bile acids
2) delay gastric emptying by absorbing water and form soft gel to slow passage and glucose and cholesterol absoprtion
AI of fiber
M: 38g/day
F: 25g/day
Gastritis
Inflammation of stomach
Diet:
-clear liquids advance as tolerated
- avoid gastric irritants
Inflammatory bowel disease (IBD)
1) Crohn’s: affects the terminal ileum leading to B12 and Fe deficiency
2) Ulcerative colitis (UC): ulcerative disease of colon leading to bloody diarrhea, electrolyte (Na, K) losses
Diet:
- maintain fluid and electrolytes
- limit fat only if steattorhea (if so, also assess Ca, Mg, Zn since these minerals are lost in fatty stools)
- protein at each meal
- chewable MVit
Irritable Bowel Syndrome (IBS)
chronic abdominal discomfort, altered intestinal motility, bloating
Diet: goal is for adequate nutrient intake
- avoid gas-forming foods
- use food diary to track intake and symptoms
- low FODMAP to eliminate sources of discomfort
Lactose intolerance
absence of lactase enzyme
*with lactose present:
- water is drawn to intestine to dilute load resulting to distention, cramps, diarrhea
- bacteria ferment lactose & release CO2
can test using:
1) breath hydrogen test
2) lactoce intolerance test
Diet: lactose-free, Ca and riboflavin supplments recommended
Lactose intolerance test
take oral dose of lactose after fast
BG = <25 mg/dl above fasting = intolerant
BG = >25 mg/dl above fasting = tolerant
Diarrhea in infants & children
Acute Tx: immediate rehydration and replace electrolytes
Chronic Tx: 40% calories as fat, balance with limited fluids; dilute fruit juices
Diarrhea in adults
Watery stool
Tx:
- replace fluids and electrolytes
- decrease gastric motility by avoiding clear liquids, caffeine, alcohol, fiber, and foods high in simple sugars
- thicken consistency of stool
- repopulate GI tract with normal flora using prebiotics (fructooligosaccharides like onion, garlic, asparagus) and probiotics (e.g. fermented foods)
Steatorrhea
high fat in stool as a result of malabsorption
Diet: high protein, high complex CHO, fat as tolerated, MCT
Short bowel syndrome (SBS)
consequences associated with significant resections of the small intestine (especially if ileum)
lead to malabsorption, malnutrition, fluid and electrolyte imbalances, weight loss
Jejunal resection
ileum can adapt and take over fuctions
diet after resection:
- normal balance of CHO, PRO
- vit and min supplements
Ileal resection
- will affect absorption of B12, intrinsic factor, and bile salts
- will impact absorption of fluid since that’s where a majority of fluid is absorbed (diet: increased water needs)
- if cannot recycle bile salts: lead to malabsorption of fat-soluble vitamins & non-emulsified fat will combine with Ca, Zn, Mg to create soaps which will interfere with absorption of these minerals
diet after resection:
- limit fat
- use MCT since it doesn’t need bile salts
- supplement fat-soluble vitamins
- supplement Ca, Mg, Zn
Liver function tests
indicate enzyme levels
in liver disease, enzyme levels (ALP, AST, ALT) are elevated indicating tissue damage
ALP (alkaline phosphatase)
upper level: 120
increased = liver disease, bone disease
AST (aspartate amino transferase)
upper level: 35
increased = hepatatis
ALT (alanine aminotransferase)
upper level: 36
increased = liver disease
Acute viral hepatitis
leads to inflammation, necrosis (death of tissue d/t lack of O2), and *anorexia
Hep A: fecal to oral (*most directly connected to food)
Hep B: blood to blood contact
diet:
- increase fluids
50-55% CHO to replenish glycogen
- high protein (1-1.2 g/kg) to prevent fatty liver
- mod to liberal fat
- small, frequent feedings
Cirrhosis
scarred/damaged liver tissue replaced by connective tissue that divides liver, rerouting veins and capillaries
blood flow: portal vein -> liver -> vena cava
when blood cannot leave liver –x–> vena cava = *ascites [fluid build-up in abdomen]
when blood cannot enter (portal vein –x–> liver) = *portal hypertension
resulting to varices [enlarged veins]
diet:
- high kcal
- adequate to high pro
- mod to low fat
- if ascites: low sodium
- if varices: low fiber
Alcoholic liver disease
liver injury d/t alcohol’s effect on metabolism (alcohol -> acetaldehyde + excess hydrogen, which replaces fat as fuel)
associated w/ malnutrition
diet:
- supplement thiamin (B1) and folic acid
- increased need for B vitamins and Mg
Hepatic failure / End-stage liver disease
liver cannot convert ammonia into urea causing build-up
diet:
- if not comatose: mod to high PRO
- high kcal, 30-35% fat
Steatosis / Non-alcoholic fatty liver disease (NAFLD)
excess fat build up in liver unrelated to EtOH
can be managed with lifestyle changes: slow wt loss, healthful eating, physical activity
Cholecystitis
inflammation of gall bladder
diet:
- low fat
- if cholecystectomy (removal): limit fat intake annd slowly increase fiber
Pancreatitis
inflammation of pancreas
common characteristic: premature activation of enzymes within pancreas leading to autodigestion (enzyme destroying own tissue)
diet/tx if acute:
- put pancreas at rest, progress to easily digested foods with low fat
- elemental EN
diet/tx if chronic:
- give pancreatic enzymes orally (PERT) to minimize fat malabsorption
- to avoid pain during eating: avoid large meals with fatty foods
Cystic fibrosis
disease of exocrine glands resulting to secretion of thick mucus (could lead to CPD)
*affects transport of chloride across the cell membrane
diet:
- PERT
- high kcal, PRO, unrestricted fat (to meet energy needs, liberal salt
- supplement Zn & water-soluble forms of Vit A & E
Hypertension
High blood pressure
*can be managed by diuretics, salt restriction, DASH diet, Mediterranean diet
Stages of hypertension
Normal: <120/80 mm/Hg
Elevated: 120-129 / <80
Stage 1: sys 130-139 OR dia 80-89
Stage 2: sys >140 OR dia >90
Notes:
- systolic: contraction, greatest pressure
- diasotlic: relaxation, least pressure
Atherosclerosis
progressive narrowing of arteries
Coronary artery disease (CAD)
hard, narrow arteries from plaque build-up
Ischemia
deficiency of blood d/t obstruction
Arteriosclerosis
loss of elasticity of blood vessel walls
Myocardial infarction
reduced coronary flow to myocardium d/t blood clot
Dyslipidemia
Abnormal levels of blood lipids, includes high TGs and low HDL
*small, dense LDL-C associated with increased risk of CVD
Metabolic syndrome
3 or more of the following risk factors are linked to insulin resistance which can increase risk for coronary issues
1) hypertension (>130 and/or >85)
2) hyperlipidemia (elevated TG >150 mg/dl)
3) hyperglycemia (fasting >100 mg/dl)
4) high waist circumference
5) low HDL
Cholesterol Levels
can assess risk of CVD
Total cholesterol: <200 mg/dl is desirable
LDL-C: <100 optimal
HDL-C: <40 (M) and <50 (F) is low, >60 is high
Heart healthy diet
- Limit sat fat, cholesterol, sodium; no trans fat
- promote whole grains, FV, unsaturated fats
- include 20-30g fiber per day (5-10g soluble fiber)
Heart failure
leads to decreased output and fluid being held
diet:
- low Na (e.g. DASH)
- high PRO
- kcal needs (RMR x physical activity factor): 22 kcal/kg if nourished, more if not
sedentary: 1-1.4
low active: 1.4-1.6
active: 1.6-1.9
very active: 1.9-2.5
-possibly thiamin; folate, Mg, mvit w/ B12
Cardiac cachexia
complication of heart failure resulting to unintended weight loss
diet:
- low sat fat, cholesterol, trans fat
- limit sodium
- high calorie
Vasopressin / anti-diuretic hormone (ADH)
released from hypothalamus and elevates BP and increases reabsorption of water in kidneys
Renin
released by kidneys to control BP & increase Na absorption
Erythropoietin (EPO)
produced by kidney and stimulates bone marrow to produce RBC
Renal lab tests
High creatinine and BUN & BUN/creatinine ratio = impaired kidney fx
ratio of >20:1: may be reversible
ration of <10:1: may need dialysis
Renal solute load: measures nitrogen and electrolytes
Kidney stones / calcium oxalate stones
most common renal disorder
diet:
- adequate Ca intake
- low oxalate diet (foods high: dark leafy greens, chocolate, nuts, beets, tea)
Alkaline ash / acidic ash diet
minerals not oxidized can leave residue in urine
*to prevent acidic stones: create alkaline ash
- increase cations by adding FV, brown sugar
*to prevent alkaline stones: create acidic ash
- increase anions by adding animal protein
Acute kidney injury
sudden shutdown of kidney w/ previously adequate capacity
possibly oliguria (low urine output) and azotemia (increased urea in blood)
*energy expenditure increases as kidney fx declines
diet:
- low Na, K, phos
- replace fluid output
Nephrosis
defect in glomerulus permiting escape of large amounts of PRO
symptoms: albuminuria, edema, malnutrition, hyperlipideia
diet:
- restrict fat intake (<30% of kcal)
- modest sodium restriction
Chronic kidney disease
kidney damage affecting filtration of blood
diet:
-DASH, Mediterranean, high FV
-adequate energy
-restrict sodium
*if stage 3-5: restrict protein
-fluid unrestricted unless stage 5
Dialysis
long term goal is to prevent malnutrition
hemodialysis: 1-1.2g protein/kg SBW
peritoneal dialysis: 1-1.2g protein/kg SBW or adj BW
Diabetes Mellitus
Type 1: insulin deficient & dependent on exogenous insulin
- strategies: consistent CHO if fixed daily dose of insulin, reduce insulin for planned exercise
Type 2: insulin-resistant
- strategies: healthy eating and PA
*risk factors:
- acanthosis nigricans from insulin-resistance
- GAD antibodies (glutamic acid decarboxylase)
goal:
*maintain normal blood glucose
- maintain serum lipids & BP