Goats Flashcards

1
Q

what type of breeders are goats?

A

seasonally polyoestrous (sept-feb)

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2
Q

what is a bill rag

A

to ID if female on heat

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3
Q

who can disbud goat kids

A

vets only - cant tolerate local an, so GA! (v thin skull!)

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4
Q

what poisonous plants cause v+ in goats

A

rhododendron

pieris

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5
Q

what is the most common cause of enterotoxaemia

A

c. perfringens type d (pulpy kidney in sheep)

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6
Q

what are the signs of c. perfringens enterotoxaema

A

d++ w blood
rapid death
acute or peracute
PME - liver dark and haemorrhagic

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7
Q

what can trigger a c perfringens enterotoxaema

A
stress
illness
kidding
acidosis (gorging)
changes in housing/diet
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8
Q

how can you tx and prev enterotoxaemia from c perfringens

A

IVFT and oral FT
carprofen
vac - q6mths. lambivac

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9
Q

what are the signs of Listeriosis infection

A
encephalitis
septicaemia
pytalism (cant swallow)
droopy eyelid
droopy ear
lateral recumbancy
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10
Q

how do you tx listeria

A

abx - pot sulphonamides
NSAIDs
IVFT - often acidotic due to loss of buffer saliva
nursing care

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11
Q

what is CAE and what does it stand for?

A

caprine arthritis encephalitis,

lentivirus

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12
Q

what is the significance of the blood Ab test

A

if its +ve, its +ve for life

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13
Q

desc the signs of CAE

A

slow
not always sign - BIOS vital
arthritis - +- carpal pain
mastitis

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14
Q

how does CAE spread

A

colostrum

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15
Q

how do you tx CAE

A

you don’t - you cull

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16
Q

what are the notable signs of johnes

A
nothing outstanding, unlike cows:
poor yield
wasting
anaemia
d+ (end stage)
17
Q

is there a tx for johnes

A

gudair vaccine

18
Q

what does johnes appear like at PME in goats and sheep (not cattle!)

A

velvety mucosa

big mesenteric LN (calcified likely)

19
Q

what faecal stain IDs johens

A

ZN

20
Q

what is the path that causes CLA

A

corynebacterium pseudo-tb

CLA = caseous lymphadenitits

21
Q

is CLA anything to worry about

A

YEs - severe disease

rapid spread

22
Q

do milking goats get teat dipped?

A

not usually - clinical mastitis not an issue! sub clin is common however

23
Q

why isn’t the SCC rule applied to goat milk?

A

goats are apocrine milkers, cows are merocrine. apocrine means that cells MUST break away for milk to be ‘let downn’

24
Q

gooats get hypocal and ketosis just like ocws, but why is it uncommon to get mypoMg

A

they don’t JUST eat grass - other sources of Mg

25
Q

name the 4 main causes of goat respiratory disease

A
  • pasteurellosis (mannheimia haemolytica)
  • mycoplasma
  • lung worm (dictylocaulus filiaria & meulleria capillaria)
  • TB
26
Q

how do you protect against pasteurellosis

A

vaccinate

use sheep vacc, or just use the 4-in-1 clostridium vacc too

27
Q

because of so few licensed drugs, what is the cascade rule for milk withhold

A

7d

28
Q

what is pygmy goat syndrome

A

heritable
1ry or 2ry keratinisation defect
ears, eyes, nose

29
Q

orf is….

A

zoonotic

30
Q

why is PGE always a ddx for anything GI

A

goats never develop immunity to parasites

31
Q

how do you tx round worm infections

A

double dose

get accurate kg (fat is abdominal..)

32
Q

why are coccidia so bad

A

50% of goat spp are path

so its worth getting them ‘speciated’ soo you know

33
Q

whats the name for false pregnancy

A

hydrometra

34
Q

how do you treat hydrometra

A

2 doses of PGs, q12hr