ENT Flashcards

1
Q

Borders of the anterior triangle in the neck

A

Superiorly – inferior border of the mandible (jawbone).
Laterally – anterior border of the sternocleidomastoid.
Medially – sagittal line down the midline of the neck.

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2
Q

The contents and boundaries of the anterior triangle in the neck

A

Carotid triangle

  • Sup – post belly digastric; Lat - med SCM; infer - sup belly omohyoid
  • common carotid artery (bifurcates; + carotid sinus - BaroR), internal jugular vein, hypoglossal and vagus nerves.

Submental triangle

  • Inf – hyoid bone; Med– midline of neck; La - ant belly digastric
  • submental lymph nodes

Submandibular Triangle
- Sup – body of mandible; Ant – ant belly digastric;
Post – post belly digastric
- submandibular gland (salivary), and lymph nodes.

Muscular Triangle

  • Sup– hyoid bone; Med – midline of the neck; Supero-lat – sup belly omohyoid; Infero-lat – inf portion SCM
  • Infrahyoid muscles, the pharynx, and the thyroid, parathyroid glands.
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3
Q

Borders of the posterior triangle and its contents

A

Ant – post SCM; Post– ant trapezius; Inf– middle 1/3 clavicle.
Split by omohyoid into - occipital triangle (larger + sup) and subclavian triangle (contains distal subclavian A)

  • Muscles –> omohyoid;
  • external jugular vein –> empties into the subclavian vein
  • distal part of the subclavian artery
  • accessory nerve (CN XI) and cervical plexus (+phrenic N)
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4
Q

Where is the thyroid gland located and its blood supply

A
  • anterior neck (post to muscles)
  • C5 and T1 vertebrae; inferior to thyroid cartilage
  • divided into 2 lobes connected by an isthmus
  • butterfly shape.
  • superior and inferior thyroid A; drainage via super, middle and infer thyroid veins –> int jug vein
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5
Q

Pathophysiology and presentation of thryoglossal cysts

A

As the thyroid gland descends during development, it moves through a duct called the thyroglossal duct. This duct normally fuses and regresses in the adult.

However, in 50% of individuals, the distal portion of the duct continues as a pyramidal lobe – effectively an extra piece of thyroid tissue. (No clinical consequences).

Other portions of the duct may persist as thyroglossal cysts. These present with a mass in the midline of neck, and can be excised surgically.
Sx - non-tender fluctuant swelling in the midline. Move up when pt extends tongue.

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6
Q

Where are the parathyroid glands located and how many are there

A

Posterior aspect of the lateral lobes of the thyroid gland. They are flattened and oval in shape, situated external to the gland itself, but within its sheath.
Most people have 4 - 2 from sup parathyroid (from 4th pharyngeal pouch); 2 from inf parathyroid ( from 3rd pharyngeal pouch)
- supplied by inf thyroid A

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7
Q

What are the paranasal sinuses and where do they empty

A

Extensions of the respiratory part of the nasal cavity - air filled space lined by ciliated pseudostratified resp epi.
4 pairs:
named according to the bone in which they are located; - - maxillary - middle meatus
- frontal- middle meatus
- sphenoid - roof or nasal cavity
- ethmoid - post= sup meatus; middle/ant = middle meatus

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8
Q

Where is epistaxis most likely to develop from

A

Kiesselbach/ Littles area.

kiesselbachs plexus:

  • -> anterior ethmoidal artery
  • -> sphenopalatine artery (max A branch)
  • -> greater palatine artery (max A branch)
  • -> superior labial artery (branch facial A)
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9
Q

Types of audiometry

A

Quantify loss and determine its nature.

Pure tone audiometry (PTA)
- tones at different frequencies and strengths
-Pt. indicates when sound appears and disappears
-Mastoid vibrator –>bone conduction threshold.
- Threshold at different frequencies are plotted to
give an audiogram.
> age-related hearing loss and sensoineural - lose high freq
> otosclerosis + menieres- lose all freq

Tympanometry

  • Measures stiffness of ear drum
  • Evaluates middle ear function
  • Flat tympanogram: mid ear fluid or perforation
  • Shifted tympanogram: +/- mid ear pressure

Evoked response audiometry

  • Auditory stimulus –> measurement of elicited brain response by surface electrode.
  • Used for neonatal screening (if otoacoustic emission testing negative)
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10
Q

What is the normal range of hearing and the grades of hearing loss

A
  • 0-20dB

mild - 20-40
mod - 41-70
severe - 71-95
profound - >95

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11
Q

Causes of hearing loss

A
Conductive - problem with transmission of soundwaves from external ear through middle ear --> WIDENING
Wax/FB
Infection (otitis media +/- effusion)
Drum perf (trauma/infection)
Extra - otosclerosis
Neoplasia / no vent of middle ear
Injury (barotrauma)
Granulomatous (wegeners/sarcoid)

Sensoineural - problem in cochlear, cochlear N or brainstem –> DIVINITY
Development (alport/TORCH/perinatal anoxia) and degen (presby)
Infection - VZV, measles, mumps, influenza, meningitis, HZV
Vasc - int aud A ischaemia (+vertigo), stroke
Inflam (vasculitis/sarcoid)
Neoplasia (CPA tumour, acoustic neuroma)
Injury (noise, head trauma)/ MS/ low b12
Toxins (gentamycin, furosemide, aspirin, vancomycin)
lYmph (menieres, perilymphatic fistula (rupture round window))

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12
Q

Presentation, causes and differential of otitis externa

A

Presentation
􏰀 Purulent discharge; 􏰀 Itch/ ear fullness 􏰀 Pain and tragal tenderness; 􏰀 localised red +swelling; warm 􏰀 +/- blood; 􏰀 conductive hearing loss
- narrowing of the canal + accumulation of debris, leads to further entrapment of pathogens and propagating the infective process.

Discharge - White-yellow – bacterial; Thick white grey with visible hyphae or spores – fungal;

Causes –> Any interruption in wax formation

  • repeated water exposure
  • trauma to the canal (e.g. cotton buds)
  • blockage (e.g. debris)

Organisms –> Mainly pseudomonas; S. Epidermidis; S.aureus

Differential - Clear grey – otitis media +/- blood - perf. Relief of pain.

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13
Q

Mx and complications of otitis media

A
  • prevention - debris removed by microsuction; avoid swimming; mx eczema
  • aural toileting
  • topical antibiotics (depending on local protocol)
  • simple analgesia
  • Steroid drops (if canal inflam)

Complication - Malignant otitis externa –> extension –> skull osteomyelitis
-90% of pts. are diabetic (or other immune compromise)
Presentation
- Severe otalgia which is worse @ night +/- headache
- Copious otorrhoea
- Granulation tissue in the canal
- can involve CN VII
Ix - urgent CT scan
Rx - Surgical debridement; IV abx

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14
Q

What is Bullous Myringitis

A

Painful haemorrhagic blisters on deep meatal skin and TM.

Assoc. ̄c influenza infection

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15
Q

presentation of TMJ dysfunction

A
- Symptoms
􏰀 Earache (referred pain from auriculotemporal N.)
􏰀 Facial pain
􏰀 Joint clicking/popping
􏰀 Teeth-grinding (bruxism)
􏰀 Stress (assoc.  ̄c depression)

Signs
􏰀 Joint tenderness exacerbated by lateral movements of an open jaw.

Investigation –> MRI
Management –> NSAIDs; Stabilising orthodontic occlusal prostheses

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16
Q

Presentation and mx of Ramsay-Hunt Syndrome

A

Herpes Zoster oticus - unilateral facial palsy caused by reactivation of VZV from the geniculate nucleus on CNVII

Clinical Features

  • moderate to severe ear pain
  • facial palsy within few days (more severe than bells) + ipsilateral vertigo, hyperacusis, and tinnitus.

O/E - Vesicles will be visible during this latter period, covering the concha, anterior ⅔ tongue, +/- soft palate

Mx - prednisolone and acyclovir ASAP
~ 75% of cases will resolve
Complications - chronic tinnitus + vestibular dysfunction.

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17
Q

Presentation, RF and common organisms causing acute otitis media

A
Presentation
􏰀 Usually children post viral URTI
􏰀 Rapid onset ear pain, tugging @ ear.
􏰀 Irritability, anorexia, vomiting
􏰀 Purulent discharge if drum perforates
\+/- malaise, fever, and coryzal symptoms
o/e
􏰀 Bulging, red TM; 􏰀 Fever
- make sure test function of facial N

Common organisms - RSV, H. influenzae, S. pneumoniae, Moraxella catarrhalis, and S.pyogenes,

RF - Age (peak age 6-24 months); Parenteral / passive smoking; Previous URTI; Presence of enlarged adenoids; Bottle feeding or dummy use (breast feeding is protective); GORD and ­BMI (in adults)

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18
Q

Ix, Mx and complications of acute otitis media

A

Ix - cultures if pyrexial, swab discharge if present

Mx -majority resolve < 24 hours, nearly all within 3 days.

  • simple analgesics
  • if systemically unwell - Amoxicillin

Complications
Mastoiditits; Meningitis; Facial nerve paresis; Intracranial abscess; Sigmoid sinus thrombosis; Chronic Otitis Media; OME; perforation TM; sepsis; IE; septic arthritis

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19
Q

Presentation, ix and rx of otitis media with effusion

A
Presentation
􏰀 Inattention at school
􏰀 Poor speech development
􏰀 Hearing impairment/ aural fullness
Hx previous acute otitis media
o/e
􏰀 Retracted dull TM; 􏰀 Fluid level
Ix
􏰀 Audiometry: flat tympanogram 
Rx
􏰀 Usually resolves spontaneously,
􏰀 Consider grommets if persistent hearing loss
SE: infections and tympanosclerosis
Same complications as acute otitis media
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20
Q

Presentation of chronic suppurative otitis media

A

Painless discharge and hearing loss (conductive)
- chronic discharging ear >6 weeks
o/e –> TM perforation +/- discharge

Rx
- Aural toilet; Abx / Steroid ear drops
+/- tympanoplasty if ear drum doesn’t repair itself

Complications
- Cholesteatoma; labyrinths

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21
Q

presentation and mx of mastoiditis

A

Middle-ear inflam - destruction of mastoid air cells and abscess formation.
Sx - Fever; Mastoid tenderness/ swelling; Protruding auricle
Rx- IV Abx; CT head if no improvement >24 hr
? Myringotomy ± mastoidectomy

Risk meningitis

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22
Q

presentation and mx of Cholesteatoma

A

Locally destructive expansion of stratified squamous epithelium within the middle ear.
- congenital/ acquired secondary to chronic OM

Presentation
􏰀 Foul smelling white discharge
􏰀 Headache, pain
􏰀 CN Involvement –> Vertigo; Deafness (conductive) ; Facial paralysis
o/e –> Appears pearly white - surrounding inflammation

Complications
􏰀 Deafness (ossicle destruction); 􏰀 Meningitis; 􏰀 Cerebral abscess

Mx
Surgery to remove (open/closed)

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23
Q

Define and name causes of tinnitus

A
Sensation of sound w/o external sound stimulation Causes
- Specific
􏰂 Meniere’s (vertigo+deafness))
􏰂 Acoustic neuroma (unilateral +vertigo+ deafness)
􏰂 Otosclerosis (?FH)
􏰂 Noise-induced
􏰂 Head injury
􏰂 Hearing loss: e.g. presbyacusis 
  • General
    􏰂 high BP
    􏰂 anaemia
- Drugs
􏰂 Aspirin
􏰂 Aminoglycosides 
􏰂 Loop diuretics
􏰂 EtOH
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24
Q

Mx of tinnitus

A

Treat any underlying causes
􏰀 Psych support: tinnitus retraining therapy
􏰀 Hypnotics @ night may help

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25
Q

Presentation and mx of otosclerosis

A

AD condition characterised by fixation of stapes at the oval window. F>M=2:1
Presentation
􏰀 Begins in early adult life
􏰀 Bilateral conductive deafness + tinnitus
􏰀 HL improved in noisy places: Willis’ paracousis
􏰀 Worsened by pregnancy/ menstruation/ menopause
􏰀 difficulty hearing low, deep sounds and whispers
􏰀 ?dizziness

Ix - PTA shows dip (Caharts notch) @ 2kHz
Rx - Hearing aid or stapes implant

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26
Q

Causes of vertigo

A

IMBALANCE
Infection/injury e.g. labyrinthitis/ head injury
Meniere’s
BPV
Aminoglycosides/Fudrosemide/ metronidazole
Lypmph - peri-fistula
Arterial - migraine; TIA; stroke
Nerve - acoustic neuroma /vestibular schwannoma
Central lesion - demyelination (MS), tumour, infarct
Epilepsy - complex partial

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27
Q

Presentation and mx of menieres disease

A

Dilatation of endolymph spaces of membranous labyrinth (endolymphatic oedema)

Presentation
􏰀 Attacks of recurrent vertigo (~20mins) occur in clusters and last up to 12h
􏰀 Progressive SNHL 􏰀 +/- N&V 􏰀 Tinnitus 􏰀 Aural fullness

Audiometry –> low-freq SNHL which fluctuates

Rx
- Medical –> cyclizine, betahistine
Surgical –> Gentamicin instillation via grommets OR Saccus decompression
Inform DVLA

28
Q

Presentation and mx of Vestibular neuronitis (Labyrinitis)

A

neuronitis - Inflammation of the vestibular nerve - lasts for days.

Presentation
􏰀 Follows febrile illness (e.g. URTI)
􏰀 Sudden vomiting
􏰀 Severe vertigo exacerbated by head movement

Mx

  • cyclizine
  • if persists –> vestibular rehab via Cawthorne-Cooksey exercises.
29
Q

Presentation and mx of BPPV

A

Displacement of otoliths in semicircular canals

Presentation
􏰀 Sudden rotational vertigo for <30s
􏰂 Provoked by head turning
􏰀 Nystagmus

Causes
􏰀 Idiopathic 􏰀 Head injury 􏰀 Otosclerosis 􏰀 Post-viral

Dx –> Hallpike manoeuvre - upbeat-torsional nystagmus

Rx
􏰀 Self-limiting; advise not to drive when dizzy; reduce head movements
􏰀 Epley manoeuvre to redistribute particles
􏰀 Betahistine: histamine analogue

30
Q

What is an acoustic neuroma and the presentation +mx

A

also called vestibular schwannoma - Benign, slow-growing tumour of schwann cells surrounding vestibulocochlear N
􏰀 Acts as SOL–> Cerebellopontine angle syndrome (80% of CPA tumours)
􏰀 Assoc. ̄c NF2 (bilateral)

Presentation
􏰀 Slow onset, unilat SNHL, tinnitus ± vertigo 
􏰀 Headache (Raised ICP)/ seizures
􏰀 CN palsies: 5,7 and 8
􏰀 Cerebellar signs
Ix
􏰀 MRI of cerebellopontine angle
􏰀 PTA
Differentials
􏰀 Meningioma
􏰀 Cerebellar astrocytoma 
􏰀 Mets
Rx
- monitor - 
􏰀 Gamma knife
􏰀 Surgery (risk of hearing loss)
31
Q

What is Presbyacussis

A

Age-related hearing loss

Presentation
􏰀 >65yrs 􏰀 Bilateral 􏰀 Slow onset 􏰀 ± tinnitus
Ix: Loss of higher frequencies
Rx: hearing aid - external/ internal (cochlear implant)

32
Q

Causes of Tympanic membrane perforation

A
  • OM
  • FB
  • Barotrauma
  • Trauma (e.g. ear buds)

90% resolve <4weeks

33
Q

When would you refer tinnitus as an emergency

A
If they have features of:
Sudden onset pulsatile tinnitus
Significant neurology
Severe vertigo
Secondary to head trauma
Unexplained sudden hearing loss
34
Q

Causes of hearing loss in children

A

Congenital Causes
Conductive
􏰀 Anomalies of pinna, external auditory canal, TM or ossicles.
􏰀 Congenital cholesteatoma

SNHL
􏰂 Waardenburgs: SNHL, heterochromia +
telecanthus
􏰂 Alport’s: SNHL + haematuria
􏰀 Infections: TORCH
􏰀 Ototoxic drugs
Perinatal
􏰀 Anoxia
􏰀 Cerebral palsy
􏰀 Kernicterus
􏰀 Infection: meningitis

Acquired Causes
􏰀 OM/OME
􏰀 Infection: meningitis, measles
􏰀 Head injury

35
Q

Presentation and mx of pinna haematoma

A

Caused –> Blunt trauma –>subperichondrial haematoma.
–> ischaemic necrosis of cartilage and subsequent fibrosis to “cauliflower ears”.

Mx: aspiration + firm packing to auricle contour.

36
Q

Presentation and mx of Exostoses

A

Bony hypertrophy due to cold exposure e.g. from swimming / surfing –> smooth, symmetrical narrowing of external canal

Symptoms
􏰀 Asympto unless narrowing occludes
􏰁 conductive deafness.
Rx: conservative or surgical widening

37
Q

Mx of ear wax

A

Cerumen Auris
- Secreted in outer 3rd of canal to prevent maceration
- Wax accumulation can –> conductive deafness.
Mx
􏰂 Suction under direct vision - microscope
􏰂Syringing after 1wk softening with olive oil

38
Q

Mx of epistaxis

A

Examine - anterior (littles) or posterior bleed
Pressure - over nose with compression device or fingers and head tilted down
Insert gauze - soaked in xylometazoline +lignocaine (Ant)
Silver nitrate to cauterise- locally (Ant)
Then pack
Admit and pack 48h (ant + post) if posterior
Interventional radiology- embolization (post)
Surgical consultation - ENT consult for severe or high risk bleeding

39
Q

Causes of epistaxis

A
  • nose picking
  • #
  • URTI
  • Pyogenic granuloma (overgrowth littles)
  • HHT
  • Coagulopathy - warfarin, NSAIDs, Haemophilia, low plt, vWD, high alcohol
  • neoplasm
40
Q

Mx after epistaxis

A
􏰀 Don’t pick nose
􏰀 Sit upright, out of the sun
􏰀 Avoid bending, lifting or straining 
􏰀 Sneeze through mouth
􏰀 No hot food or drink
􏰀 Avoid EtOH and tobacco
41
Q

Presentation, pathophysiology and mx of allergic sinusitis

A
  • sneezing, pruitis, rhinorrhoea
    +/- nasal polps
    Seasonal - pollen; persistent - dust mites/pets; occupational - wood dust; latex etc

IgE -mediated inflammation of nasal mucosa; causing large release of histamine from mast cells within mucosa

Mx

  • allergen avoidance + regularly wash bedding on high heat; dont go outside when pollen count high
    1) anti-hist; beclometasone nasal spray
    2) intranasal steroids
    3) immunotherapy
42
Q

Cause, sx and mx of sinusitis

A

Majority are bacterial infection (pneumoccocas, haemophilus, moraxella) secondary to viral
Acute 7-30d; chronic >90d

Sx
􏰀 Pain
- Maxillary (cheek/teeth)
- Ethmoidal (between eyes) 
- increase on bending / straining
􏰀 Discharge: from nose--> post-nasal drip and foul taste
􏰀 Nasal obstruction / congestion
􏰀 Anosmia or cacosmia (bad smell w/o external source)
􏰀 Systemic symptoms: e.g. fever

Mx
- acute –> bed rest, decongestants, analgesia
+/- nasal douching and topical steroids
- chronic - stop smoking, fluticasone nasal spray
? functional endoscopic sinus surgery

43
Q

Complications of sinusistis

A

rare

  • meningitis
  • orbital cellulitis
  • osteomyelitis
  • cavernous sinus thrombosis
44
Q

Pathophysiology and presentation of nasal polyps

A

Lesions developing from nasal mucosa, linked with asthma; CF; Allergic / non-allergic rhinitis. Often M >40 y/o
Sites
􏰀 Middle turbinates 􏰀 Middle meatus 􏰀 Ethmoids

Symptoms
􏰀 Watery, anterior rhinorrhoea
􏰀 Purulent post-nasal drip
􏰀 Nasal obstruction
􏰀 Sinusitis
􏰀 Headaches
􏰀 Snoring

Signs - Mobile, pale, insensitive

Single Unilateral Polyp- May be sign of rare but sinister pathology: 􏰂 Nasopharyngeal SCC 􏰂 Glioma 􏰂 Lymphoma
􏰂 Neuroblastoma 􏰂 Sarcoma
–> Do CT and get histology

Nasal Polyps in Children
􏰀 Rare <10yrs old; consider neoplasms and CF

Mx - Stop smoking, weight loss
􏰂 Betamathasone drops for 2/7
􏰂 Short course of oral steroids
􏰀 Endoscopic Polypectomy

45
Q

What is the commonest congenital abnormality of the nose

A
  • choanal atreia –> bony septum between nose and pharynx
  • if bilateral –> present as airway obstruction with cyclical cyanosis and crying relieves resp distress in neonates (as obligate nasal breathers)
46
Q

Mx of fractures nose

A
  • exclude septal haematoma (can lead to septal necrosis and nasal collapse –> swelling and nasal obstruction. Mx - evacuate and pack)
  • exclude basal skull #

re-examine after 1 weeks (swelling reduced)

reduction under GA and post-op splinting <2weeks

47
Q

Causes of a hoarse voice

A
  • infection e.g. acute laryngitis
  • inflammation
  • laryngeal ca
  • vocal cord nodule
  • hypothyriod, acromegaly, goitre
  • damage to nerve (surgery/disease)
  • trauma
  • oesophageal/ lung cancer
  • AA
  • neuropathic/ DM
  • cervical lymphadenopathy
  • stroke
  • MG (fatigue)
48
Q

Presentation and criteria for tonsilitis

A
Sore throat, inflamed tonsils and oropharynx
CENTOR Criteria
1) Age <14
2) Tonsillar exudates
3) Tender anterior cervical adenopathy
4) No cough
5) Fever 

Organisms - EBV; GAS: Pyogenes; Staphs; Moraxella

Mx
Analgesia: Ibuprofen / Paracetamol ± gargle
Centor >4 = Abx - PenV 10 days

49
Q

Complications of strep throat + their presentation and the mx

A

Peritonsillar Abscess (Quinsy) - Typically adults
Symptoms - Trismus; Odonophagia: unable to swallow saliva/drooling; Halitosis
Signs - Tonsillitis; Unilateral tonsillar enlargement; Contralateral uvula displacement; Cervical lymphadenopathy
Rx –> Admit and IV Abx
􏰂 Incision and drain under LA or tonsillectomy under GA

Retropharyngeal Abscess - children
- Unwell child - stiff, extended neck +refuses to eat/ drink + sx of quinsy; Fails to improve on IV Abx
􏰂 Unilateral swelling of tonsil and neck (one sided due to median raphe)
Ix- Lat. neck x-rays show soft tissue swelling
- CT from skull-base to diaphragm (hypodense lesion)
Rx –> IV Abx; Incision and drainage

Lemierre’s Syndrome
􏰀 IJV thrombophlebitis –> septic embolization most commonly affecting the lungs.
Organism: Fusobacterium necrophorum
Rx –> IV Abx: pen G, clinda, metro

Scarlet Fever
􏰀 “Sandpaper”-like rash on chest, axillae or behind ears 12-48h after pharyngotonsillitis.
􏰀 Circumoral pallor 􏰀 Strawberry tongue
Rx –> Start Pen V/G and notify HPA.

Rheumatic Fever
􏰀 Carditis 􏰀 Arthritis 􏰀 Subcutaneous nodules 􏰀 Erythema marginatum 􏰀 Sydenham’s chorea

Post-streptococcal Glomerulonephritis
􏰀 Malaise and smoky urine 1-2wks after a pharyngitis

50
Q

Indications for tonsillectomy

A

Recurrent tonsillitis if all the below criteria are met:
􏰂 Caused by tonsillitis
􏰂 5+ episodes/yr
􏰂 Symptoms for >1yr
􏰂 Episodes are disabling and prevent normal
functioning

Airway obstruction: e.g. OSA in children

Quinsy

Suspicion of Ca: unilateral enlargement or ulceration

51
Q

Presentation and mx of epiglottitis

A

No cough, sore throat, fever, dyspnoea, voice changes, dysphagia, tender anterior neck +/- cellulitis, hoarseness, pharyngitis; drooling

Most commonly caused by s.pneumonia

Mx - take to ITU, dont examine (risk resp arrest)

  • call ENT + anaesthetist
  • NEB Adr and IV dex and IV abx
  • cultures
  • pain relief
52
Q

Presentation and mx of Laryngitis

A

Usually viral and self-limiting
- 2O bacterial infection may develop

Symptoms: pain, hoarseness and fever +/- globus pharygeus
o/e redness and swelling of the vocal cords

Rx: Supportive - rest voice, no smoking/alcohol/ hydration
-Pen V if fever >48h

53
Q

Presentation and mx of Ludwigs angina

A

Infection of the space between the floor of the mouth and mylohyoid, most commonly associated with dental infection (group A strep, s,aureus, bacteriodes)
Clinical Features
 Swelling of the floor of the mouth  Painful mouth
 Protruding tongue  Airway compromise  Drooling

Investigations
 CT neck
 OPG

Management

  1. Secure airway if any concerns
  2. IV antibiotics
  3. Surgery to drain any collection
54
Q

What is laryngomalacia

A

Immature and floppy aryepiglottic folds and glottis –>laryngeal collapse on inspiration

Presentation
Stridor: commonest cause in children - w/i first wks of life.
Noticeable @ certain times 
􏰂 Lying on back,
􏰂 Feeding
􏰂 Excited/upset
Problems can occur - concurrent laryngeal infections
or feeding.
55
Q

Functions of the larynx

A

􏰀 Phonation
􏰀 Positive thoracic pressure: inc. auto-PEEP
􏰀 Respiration
􏰀 Prevention of aspiration

56
Q

Causes of neck swellings

A

Reactive

  • bacteria (beta haem strep; s.aurues, TB) –> unilat
  • viral (EBV -glandular fever, CMV) –> bilat
  • parasitic - head lice
  • non-infective - sarcoidosis, CT disease

benign

  • lipoma
  • benign ca (fibroma, chrondroma, neuroma, thyroid)
  • blocked salivary gland

Malignant
- leukaemia, lymphoma, mets, thyroid

Congenital

  • thyroglossal cyst
  • branchial cyst
57
Q

Ix of neck swellings

A
  • bloods +/- film
  • viral serology
  • throat swab +culture
  • imaging
    > USS +/- FNA (superficial)
    > CXR/ CT/ MRI +/- biopsy - mass extension and additional LN involvement
58
Q

Red flags for neck swellings

A
  • fever
  • night sweats
  • weight loss
  • HSM
  • supraclavicular LN
  • > 2cm LN
  • LN hard
  • SOB
59
Q

Presentation and mx of laryngeal cancer

A

Majority SCC

Presentation
􏰀 Male smoker 􏰀 Progressive hoarseness 􏰁 stridor
􏰀 Dys-/odono-phagia 􏰀 Wt. loss +/- neck lump/cough

Ix –>Laryngoscopy + biopsy (inc. nodes); MRI staging

Based on stage –> RT; Laryngectomy
After laryngectomuy –> permanent tracheostomy
(Speech valve and Electrolarynx) + Oseophageal speech (swallowed air)
Regular f/up for recurrence

60
Q

Presentation, causes and mx of Subglottic Stenosis

A

Subglottis is narrowest part of respiratory tract in children.
Symptoms: stridor, FTT
Causes - Prolonged intubation; Congenital abnormalities
Rx
􏰂 Mild: conservative
􏰂 Severe: Tracheostomy or partial tracheal
resection

61
Q

Causes of thyroid nodules

A

Non – neoplastic nodules
 Single nodule – colloid, cystic
 Multinodular goitre

Benign
o Adenoma – Mainly follicular

Malignant
o Papillary adenocarcinoma – 70% - younger pt/ hx irradiation of the neck.
o Follicular carcinoma – 20% - mets to bones and lungs.
o Medullary carcinoma – 5% - C-cells, seen in MEN
o Anaplastic carcinoma - ~5% - older patients, poor prognosis

62
Q

Red flags and Ix for thyroid nodules

A

Red flags

  • FH
  • High radiation exposure
  • painless rapidly enlarging mass
  • insidious pain lasting several weeks

Ix
(i) Thyroid function tests (calcitonin high in medullary)
(ii) Ultrasound guided fine needle aspiration
+/- CT/MRI

63
Q

Mx of thyroid cancer

A
  • Thyroidectomy (SE recurrent laryngeal nerve damage and hypoparathyroid) - lifelong levothyroxine
  • if anaplastic - chemo/RT
64
Q

Types, presentation and mx of salivary gland cancers

A

80% of salivary gland neoplasms occur in parotid gland.

  • malignant mucoepidermoid carcinoma
  • benign - pleumorphic adenoma (80%)

submandibular gland - 50% malignant.
sublingual gland - 80% malignant

Presentation

  • slowly enlarging painless mass
  • fullness in gland region

Red flags - facial N palsy; mucosal ulceration, painless enlarging mass (hard and craggy) , increasing pain

Ix - USS + FNA +/- CT/MRI
Mx - Chemo/ radio; parotidectomy

65
Q

Presentation of sialadenitis

A

Sialadenitis - infection of salivary glands
- Bacterial –> staphylococcal
typically seen in dehydrated or immunocompromised individuals.
- Viral - 1. Paramyxovirus – Mumps 2. Coxsackievirus
3. Echovirus 4. HIV
Chronic sialadenitis is rare, and sometimes seen in TB, sarcoidosis, HIV, and syphilis.

66
Q

Presentation of Sialolithiasis

A
  • Stones in the salivary duct cause obstruction and subsequently lead to pain and swelling which is worse during meals. Stones are 9 times more common in the submandibular gland than the parotid.

Ix –> Ultrasound or sialogram
Management
Conservative - most settle with analgesia and hydration
Or –> Radiological or surgical removal

Complications
 Sialadenitis
 Abscess formation