cardiac conduction system Flashcards

1
Q

SAN function

A

generates normal sinus rhythm

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2
Q

SAN rhythm

A

60-100 bpm

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3
Q

atrial cells rhythm

A

<60 bpm

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4
Q

AVN function (2)

A

builds in delay, acts as back-up pacemaker if SAN fails

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5
Q

AVN rhythm

A

40-60 bpm

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6
Q

His-Purkinje system components (3)

A

common bundle, left and right bundle branches, Purkinje fibres

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7
Q

Purkinje fibres rhythm

A

20-40 bpm

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8
Q

SAN dysfunction complications

A

bradycardia, tachycardia, tachycardia-bradycardia, sinus arrest, sinus node exit block

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9
Q

sinus node exit block

A

electrical activity fails to leave SAN so p-wave is hidden

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10
Q

escape rhythm

A

rhythm generated by other part of heart if SAN fails

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11
Q

junctional rhythm

A

escape rhythm generated by AVN

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12
Q

automatic re-entry

A

impulses repeatedly loop through fixed anatomical circuits e.g. scar tissue from previous MI or pathways in WPW syndrome

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13
Q

functional re-entry

A

multiple small re-entry circuits changing in size and location due to dynamic and transient electrical instability in myocardium with differing conduction velocities and refractory periods

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14
Q

atrial fibrillation

A

uncoordinated atrial contractions due to re-entry circuits

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15
Q

atrial flutter

A

increased coordinated atrial contractions

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16
Q

why are rapid atrial rates harmful? (4)

A

increased calcium load toxicity, AP shape changes, atrial fibrosis, atrial remodelling

17
Q

macro-re-entrant tachycardia

A

leaking of current from tricuspid valve annulus

18
Q

AVN first degree block (3)

A

least severe, slowed or delayed conduction, does not require treatment

19
Q

AVN second degree block

A

intermittent conduction

20
Q

AVN second degree block Mobitz T1 (2)

A

slowing between beats, elongation and eventually QRS depression

21
Q

AVN second degree block Mobitz T2

A

no slowing between beats, sometimes signals reach ventricles

22
Q

AVN third degree block

A

complete failure, wide QRS complexes

23
Q

AVN heart blocks causes

A

pathway fibrosis, CAD, decreased perfusion, inflammation, autoimmunity, hyperkalemia

24
Q

bundle branch blocks

A

ventricles contract asynchronously due to infarct or inflammation, often requires pacing

25
Q

left bundle branch block

A

right ventricle contracts before left ventricle, common in HF patients

26
Q

right bundle branch block

A

left ventricle contracts before right ventricle