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D2 Fall - Pathology Exam 1 > 8/27: Sequelae of Pulpal Disease > Flashcards

8/27: Sequelae of Pulpal Disease Flashcards

1
Q
  1. This is inflammation of the pulpal tissue
A

a. Pulpitis

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2
Q
  1. In what ways can you have inflammation of the pulp?
A

a. Limited blood supply
b. No collateral support
c. Destructive
d. Expansile process

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3
Q
  1. What expanisle processes can occur in inflammation of the pulp?
A

a. Blood vessel
b. Leakage of fluid into surrounding tissue
c. Migration of cells

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4
Q
  1. This looks like a pulp polyp
A

a. Chronic hyperplastic pulpitis

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5
Q
  1. How can you see periapical lesions?
A

a. Presence of open or closed pulpitis
b. Virulence of involved microorganisms
c. Extent of sclerosis of dentinal tubules
d. Competency of host immune response

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6
Q
  1. How can you tell apart granuloma, abscess, cyst?
A

a. Granuloma = lining outside the epithelium
b. Abscess = neutrophils
c. Cyst = epithelium lining and open spaces

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7
Q
  1. Periapical granulmoas are found where?
A

a. Apex of non-vital tooth

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8
Q
  1. Is misnomer a true granulomatous inflammation?
A

No

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9
Q
  1. How can a periapical granulomas develop?
A

a. Can come from periapical pathosis or initial periapical abscess

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10
Q
  1. This is a pathologic cavity located in soft tissue or bone lined by epithelium
A

a. Cyst

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11
Q
  1. Cyst must be lined by what?
A

a. Epithelial lining

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12
Q
  1. This is a asymptomatic, slow-growing lesion associated with the root apex of a non-viral tooth
A

a. Apical periodontal cyst

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13
Q
  1. Is external root resorption possible in apical periodontal cyst?
A

Yes

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14
Q
  1. This accumulation of acute inflammatory cells at the apex of a non-vital tooth
A

a. Periapical abscess

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15
Q
  1. Can an acute periapical pathosis exacerbation of chronic periapical lesion lead to a periapical abscess?
A

Yes

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16
Q
  1. Are periapical abscess generally symptomatic or asymptomatic?
A

a. Symptomatic

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17
Q
  1. How can a periapical abscess be asymptomatic?
A

a. Lack of accumulation of purulent material due to a chronic path of drainage

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18
Q
  1. What are the drainage pathways of acute periapical infections?
A

a. Surface of the gingiva (parulis)
b. Palate (palatal abscess)
c. Maxillary sinus
d. Soft tissue space (cellulitis)
e. Floor of mouth (ludwig angina)

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19
Q
  1. This is a gum boil
A

a. Parulis

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20
Q
  1. A parulis is what inflammation?
A

a. Acute periapical inflammation

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21
Q
  1. This is the intraoral opening of a sinus tract
A

a. Parulis

22
Q
  1. A localized collection of pus that has accumulated in a tissue cavity, producing fluctuance
A

a. absess

23
Q
  1. This tract goes from within to the exterior
A

a. Sinus tract

24
Q
  1. This tract connects two anatomic cavities
A

a. Fistula tract

25
Q
  1. What are examples of fistula tracts?
A

a. Oroantral, oronasal
b. Tracheo-esophageal

26
Q
  1. Is cellulitis able to establish drainage into the oral cavity or onto the surface of the skin?
A

No

27
Q
  1. This is an aggressive, rapidly spreading cellulitis involving multiple anatomic spaces, submental, sublingual, submandibular
A

a. Ludwigs angina

28
Q
  1. This produces massive swelling of neck that may extend close to clavicles and cause airway obstruction
A

a. Ludwigs angina

29
Q
  1. Describe what occurs in a cavernous sinus thrombosis
A

a. Valveless venous system may allow retrograde spread of infection from middle third of the face → formation of a blood clot withing the cavernous sinus

30
Q
  1. A cavernous sinus thrombosis may be associated with the spread of infection from
A

a. Maxillary teeth

31
Q
  1. The cavernous sinus uses what structures and Cranial nerves?
A

a. Internal carotid artery
b. CN:3,4,6
c. Perpendicular plate of the ethmoid bone

32
Q
  1. This is a bacterial infection of bone
A

a. Osteomyelitis

33
Q
  1. Can you be predisposed to Osteomyelitis?
A

Yes

34
Q
  1. What occurs in acute osteomyelitis?
A

a. Spread through medullary spaces
b. Minimal tissue reaction

35
Q
  1. What occurs in chronic osteomyelitis?
A

a. Prominent tissue reaction
i. Granulation tissue
ii. Fibrosis

36
Q
  1. This is a fragment of necrotic bone separated from the adjacent vital bone that usually undergoes spontaneous exfoliation
A

a. Sequestrum

37
Q
  1. This is a non-vital bone, encased by a vital bone
A

a. Involucrum

38
Q
  1. Proliferative periostitis is a form of what?
A

a. Chronic osteomyelitis

39
Q
  1. This is osteomyelitis with proliferative periostitis
A

a. Garre syndrome

40
Q
  1. What occurs in a Proliferative periostitis reaction?
A

a. Layers of reactive bone are formed, producing cortical expansion

41
Q
  1. Where is proliferative periostitis most frequently seen?
A

a. Children, young adults in the mandibular molar or premolar area

42
Q
  1. This is also known as focal chronic sclerosing osteomyelitis
A

a. Condensing osteitis

43
Q
  1. What is condensing osteitis?
A

a. Localized are of bone sclerosis associated with the apices of teeth with pulpal disease

44
Q
  1. Actinomycosis is also known as
A

a. Ray fungus

45
Q
  1. Is actinomycosis fungal or bacteria?
A

a. Bacterial

46
Q
  1. What are the actinomyces species in actinomycosis?
A

a. Actinomyces israelii
b. Actinomyces viscosus

47
Q
  1. What are the filamentous bacteria associated with actinomycosis?
A

a. Branching
b. Gram positive
c. Anaerobic

48
Q
  1. What are the ranks of most prevalent actinomycosis?
A

a. Cervico-facial
b. Abdomino-pelvic
c. Pulmonary

49
Q
  1. In cervico-facial actinomycosis, what is the suppuration with?
A

a. Sulfur granules

50
Q
  1. Cervico-facial actinomycosis is also known as
A

a. Lumpy jaw disease

D2 Fall - Pathology Exam 1 (8 decks)

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