2017 disease classification Flashcards
1
Q
- Define gingival health
A
- absence of BoP
- absence of erthema and edema
- absence of patient symptoms
- absence of attachment and bone loss
- Physiological bone levels range from 1.0 to 3.0mm apical to the CEJ
- For an intact periodontium and a reduced and stable periodontium, gingival health is defined as <10% bleeding sites and no pocket depth exceeding 3mm
2
Q
- Describe plaque-induced gingivitis (localised/generalised gingivitis)
A
- Associated with dental biofilm alone
- mediated by systemic or local risk factors
- drug influenced gingival enlargement
3
Q
what BPE scores might plaque induced gingivitis get
A
2s
4
Q
what modifying factors can be included with plaque induced gingivits diagnosis
A
- puberty
- pregnancy epulis
- poor restorative margins
- drug influenced gingival enlargement
5
Q
- What are non-plaque induced gingival diseases
A
- genetic/developmental e.g hereditary gingival fibromatosis
- specific infections e.g. herpetic gingival stomatitis, candida albicans
- inflammatory/immune conditions e.g lichen planus (more common, biopsy), benign mucous membrane pemphigoid, vitamin C deficiency
6
Q
- What comes under necrotising periodontal disease
A
- Necrotising gingivits (NG)
- Necrotising periodontitis (NP)
- Necrotising Stomatitis (NS)
7
Q
Describe necrotising gingivitis (NG)
A
- necrosis and ulcer in the interdental papilla
- gingival bleeding
- pain
- pseudomembrane formation
- halitosis
- extraoral - regional lymphadenopathy / fever
- in children, pain and halitosis less frequent, whereas fever, lymphadenopathy, and sialorrhea were more frequent
8
Q
Describe necrotising periodontitis
A
- in addition to signs and symptoms of NG
- periodontal attachment and bone destruction
- frequent extraoral signs
- in severely immune-compromised patients, bone sequestrum may occur
9
Q
Describe necrotising stomatitis (NS)
A
- bone denudation extended through the alveolar mucosa
- larger areas of osteitis and bone sequestrum
10
Q
- What counts as periodontitis as manifestation of systemic disease
A
- classification based on the primary systemic disease
- mainly rare diseases that affect the course of periodontitis resulting in the early presentation of severe periodontitis
- papillon lefevre syndrome
- leucocyte adhesion deficiency
- hypophosphatasia
- down’s syndrome
- Ehlers-danlos
11
Q
- What are systemic diseases of conditions affecting the periodontal tissues
A
- mainly rare contitions affecting the periodontal supporting tissues independently of dental plaque biofilm-induced inflammation i.e. condition itself causing breakdown
- group of conditions which result in the breakdown of periodontal tissues and some of which may mimic the clinical presentation of periodontitis
- squamous cell carcinoma
- langerhans cell histiocytosis
12
Q
- For periodontal abscess what do you do to diagnose
A
- work out what is causing it
- most have exisitng disease
- present with pus draining through pocket or sinus
13
Q
- Periodontal endodonic lesions. What do you do
A
- need to work out if there is root damage or not
- then need to work out what the root damage is
- if no root damage, then need to work out why…
14
Q
- Mucogingival deformities and conditions. What to do
A
Decide on the type of recession you have
- Recession type 1 (RT1): gingival recession with no loss or inter-proximal aspects of the tooth
- Recession type 2 (RT2): gingival recession associated with loss of interproximal attachment. The amount of interproximal attachment loss (measured from the interproximal CDJ to the depth of the interproximal sulcus/pocket) is less than or equal to the buccal attachment loss (measured from the buccal CEJ to the apical end of the buccal sulcus/pocket).
- Recession type 3 (RT3): gingival recession associated with loss of interproximal attachment. The amount of interproximal attachment loss (measured from the interproximal CEJ to the apical end of the sulcus/pocket) is greater than the bucca attachment loss (measured from the buccal CEJ to the apical end of the buccal sulcus/pocket).
15
Q
A